Hideko Matsumoto

531 total citations
10 papers, 438 citations indexed

About

Hideko Matsumoto is a scholar working on Molecular Biology, Cell Biology and Surgery. According to data from OpenAlex, Hideko Matsumoto has authored 10 papers receiving a total of 438 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Cell Biology and 2 papers in Surgery. Recurrent topics in Hideko Matsumoto's work include Metabolism, Diabetes, and Cancer (3 papers), Adenosine and Purinergic Signaling (2 papers) and Peptidase Inhibition and Analysis (2 papers). Hideko Matsumoto is often cited by papers focused on Metabolism, Diabetes, and Cancer (3 papers), Adenosine and Purinergic Signaling (2 papers) and Peptidase Inhibition and Analysis (2 papers). Hideko Matsumoto collaborates with scholars based in Japan, United States and Germany. Hideko Matsumoto's co-authors include Samuel W. Cushman, Joshua Zimmerberg, Vadim A. Frolov, Vladimir A. Lizunov, Akira Hattori, Masafumi Tsujimoto, Nobuo Tsuruoka, Tomohiro Rogi, Shigehiko Mizutani and Masabumi Nagashima and has published in prestigious journals such as The Journal of Cell Biology, Biochemical and Biophysical Research Communications and Journal of Medicinal Chemistry.

In The Last Decade

Hideko Matsumoto

10 papers receiving 429 citations

Peers

Hideko Matsumoto
Kimberly L. Dodge United States
P S Dannies United States
Alex Legg United Kingdom
Derek McCulloch Australia
Jeremy J. G. Brown United States
F.W. Kluxen Germany
Kimberly L. Dodge United States
Hideko Matsumoto
Citations per year, relative to Hideko Matsumoto Hideko Matsumoto (= 1×) peers Kimberly L. Dodge

Countries citing papers authored by Hideko Matsumoto

Since Specialization
Citations

This map shows the geographic impact of Hideko Matsumoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideko Matsumoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideko Matsumoto more than expected).

Fields of papers citing papers by Hideko Matsumoto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideko Matsumoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideko Matsumoto. The network helps show where Hideko Matsumoto may publish in the future.

Co-authorship network of co-authors of Hideko Matsumoto

This figure shows the co-authorship network connecting the top 25 collaborators of Hideko Matsumoto. A scholar is included among the top collaborators of Hideko Matsumoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideko Matsumoto. Hideko Matsumoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Matsumoto, Hideko & Masabumi Nagashima. (2017). Shift in the function of netrin-1 from axon outgrowth to axon branching in developing cerebral cortical neurons. BMC Neuroscience. 18(1). 74–74. 8 indexed citations
2.
Matsumoto, Hideko & Masabumi Nagashima. (2010). Netrin-1 elevates the level and induces cluster formation of its receptor DCC at the surface of cortical axon shafts in an exocytosis-dependent manner. Neuroscience Research. 67(2). 99–107. 17 indexed citations
3.
Matsumoto, Hideko, et al.. (2006). Identification and characterization of p49/STRAP as a novel GLUT4-binding protein. Biochemical and Biophysical Research Communications. 344(4). 1179–1185. 6 indexed citations
4.
Lizunov, Vladimir A., Hideko Matsumoto, Joshua Zimmerberg, Samuel W. Cushman, & Vadim A. Frolov. (2005). Insulin stimulates the halting, tethering, and fusion of mobile GLUT4 vesicles in rat adipose cells. The Journal of Cell Biology. 169(3). 481–489. 150 indexed citations
5.
Chen, Xiaoli, Hideko Matsumoto, Cynthia S. Hinck, et al.. (2005). Demonstration of differential quantitative requirements for NSF among multiple vesicle fusion pathways of GLUT4 using a dominant-negative ATPase-deficient NSF. Biochemical and Biophysical Research Communications. 333(1). 28–34. 3 indexed citations
6.
Masuda, Shinako, Akira Hattori, Hideko Matsumoto, et al.. (2003). Involvement of the V2 receptor in vasopressin‐stimulated translocation of placental leucine aminopeptidase/oxytocinase in renal cells. European Journal of Biochemistry. 270(9). 1988–1994. 26 indexed citations
7.
Matsumoto, Hideko, Tetsuro Nagasaka, Akira Hattori, et al.. (2001). Expression of placental leucine aminopeptidase/oxytocinase in neuronal cells and its action on neuronal peptides. European Journal of Biochemistry. 268(11). 3259–3266. 92 indexed citations
8.
Matsumoto, Hideko, Tomohiro Rogi, Kyoko Yamashiro, et al.. (2000). Characterization of a recombinant soluble form of human placental leucine aminopeptidase/oxytocinase expressed in Chinese hamster ovary cells. European Journal of Biochemistry. 267(1). 46–52. 97 indexed citations
9.
10.
Nishigaki, Sadao, et al.. (1969). Synthetic antibacterials. I. Nitrofurylvinyl-s-triazine derivatives. Journal of Medicinal Chemistry. 12(1). 39–42. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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