Hideki Sakahira
- Molecular Biology top 1%
- Cell death mechanisms and regulation 15
- RNA Interference and Gene Delivery 4
- DNA Repair Mechanisms 3
- Mitochondrial Function and Pathology 3
- RNA and protein synthesis mechanisms 3
- Muscle Physiology and Disorders 2
- Immunology top 2%
- Aging top 5%
- Cancer Research top 5%
- NF-κB Signaling Pathways 3
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- Trauma Management and Diagnosis 2
- Co-authors
- Shigekazu NagataMasato EnariAkihiro IwamatsuHideki YokoyamaKatsuya OkawaF. Ulrich HartlPéter BreuerManajit Hayer‐Hartl
- Cited by
- Molecular BiologyImmunologyAging
- Journals
- Journal of Biological Chemistry (5 papers)Nature (2 papers)Proceedings of the National Academy of Sciences (2 papers)
- Partner nations
- JapanGermanyUnited States
In The Last Decade
Hideki Sakahira
19 papers receiving 5.6k citations
Hit Papers
Peers
Comparison fields: 5 of 120
- Molecular Biology 4.6k
- Immunology 1.1k
- Aging 71
- Cancer Research 524
- Cellular and Molecular Neuroscience 584
Countries citing papers authored by Hideki Sakahira
This map shows the geographic impact of Hideki Sakahira's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideki Sakahira with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideki Sakahira more than expected).
Fields of papers citing papers by Hideki Sakahira
This network shows the impact of papers produced by Hideki Sakahira. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideki Sakahira. The network helps show where Hideki Sakahira may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Hideki Sakahira, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 0 | |
| 2 | 2022 | 0 | |
| 3 | 2004 | 335 | |
| 4 | 2002 | 51 | |
| 5 | 2001 | 33 | |
| 6 | 2000 | 55 | |
| 7 | 2000 | 40 | |
| 8 | 2000 | 31 | |
| 9 | 2000 | 53 | |
| 10 | 2000 | 48 | |
| 11 | 2000 | 157 | |
| 12 | 1999 | 136 | |
| 13 | 1999 | 29 | |
| 14 | 1999 | 104 | |
| 15 | 1999 | 86 | |
| 16 | A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICADbreakdown → | 1998 | 2670 |
| 17 | Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosisbreakdown → | 1998 | 1398 |
| 18 | 1998 | 158 | |
| 19 | 1998 | 106 | |
| 20 | 1996 | 12 |
About Hideki Sakahira
Hideki Sakahira is a scholar working on Molecular Biology, Virology and Emergency Medicine, having authored 21 papers that have together received 5.7k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (15 papers), RNA Interference and Gene Delivery (4 papers), DNA Repair Mechanisms (3 papers), Mitochondrial Function and Pathology (3 papers), RNA and protein synthesis mechanisms (3 papers), NF-κB Signaling Pathways (3 papers), Muscle Physiology and Disorders (2 papers) and Trauma Management and Diagnosis (2 papers). The work is most often cited by research in Molecular Biology (4.6k citations), Immunology (1.1k citations) and Aging (71 citations). Hideki Sakahira has collaborated with scholars based in Japan, Germany and United States. Frequent co-authors include Shigekazu Nagata, Masato Enari, Akihiro Iwamatsu, Hideki Yokoyama, Katsuya Okawa, F. Ulrich Hartl, Péter Breuer, Manajit Hayer‐Hartl, Yoshiyuki Ohsawa and Yasuo Uchiyama. Their work appears in journals such as Journal of Biological Chemistry, Nature, Proceedings of the National Academy of Sciences, The Journal of Cell Biology and Current Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.