H.H. Ropers

779 total citations
11 papers, 562 citations indexed

About

H.H. Ropers is a scholar working on Genetics, Molecular Biology and Plant Science. According to data from OpenAlex, H.H. Ropers has authored 11 papers receiving a total of 562 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Genetics, 7 papers in Molecular Biology and 4 papers in Plant Science. Recurrent topics in H.H. Ropers's work include Genomic variations and chromosomal abnormalities (5 papers), Chromosomal and Genetic Variations (4 papers) and Genetics and Neurodevelopmental Disorders (3 papers). H.H. Ropers is often cited by papers focused on Genomic variations and chromosomal abnormalities (5 papers), Chromosomal and Genetic Variations (4 papers) and Genetics and Neurodevelopmental Disorders (3 papers). H.H. Ropers collaborates with scholars based in Germany, Netherlands and Poland. H.H. Ropers's co-authors include Matthew L. Warman, Richard Mayne, George E. Tiller, Han G. Brunner, Sylvia E. C. van Beersum, Bjørn R. Olsen, Miikka Vikkula, N.I. Zhidkova, Mary B. Goldring and Kathryn S.E. Cheah and has published in prestigious journals such as Cell, Genome Research and Kidney International.

In The Last Decade

H.H. Ropers

11 papers receiving 544 citations

Peers

H.H. Ropers
H.H. Ropers
Citations per year, relative to H.H. Ropers H.H. Ropers (= 1×) peers Agnès Fichard

Countries citing papers authored by H.H. Ropers

Since Specialization
Citations

This map shows the geographic impact of H.H. Ropers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H.H. Ropers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H.H. Ropers more than expected).

Fields of papers citing papers by H.H. Ropers

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H.H. Ropers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H.H. Ropers. The network helps show where H.H. Ropers may publish in the future.

Co-authorship network of co-authors of H.H. Ropers

This figure shows the co-authorship network connecting the top 25 collaborators of H.H. Ropers. A scholar is included among the top collaborators of H.H. Ropers based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H.H. Ropers. H.H. Ropers is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Tzschach, Andreas, Wei Chen, Fikret Erdogan, et al.. (2007). Characterization of interstitial Xp duplications in two families by tiling path array CGH. American Journal of Medical Genetics Part A. 146A(2). 197–203. 29 indexed citations
2.
Cingöz, Sultan, Anne‐Marie Bisgaard, Iben Bache, et al.. (2006). 4q35 deletion and 10p15 duplication associated with immunodeficiency. American Journal of Medical Genetics Part A. 140A(20). 2231–2235. 9 indexed citations
3.
Erdogan, Fikret, Wei Chen, Maria Kirchhoff, et al.. (2006). Impact of low copy repeats on the generation of balanced and unbalanced chromosomal aberrations in mental retardation. Cytogenetic and Genome Research. 115(3-4). 247–253. 58 indexed citations
4.
Tzschach, Andreas, Corinna Menzel, Vera M. Kalscheuer, et al.. (2006). Molecular cytogenetic analysis of a de novo interstitial deletion of 5q23.3q31.2 and its phenotypic consequences. American Journal of Medical Genetics Part A. 140A(5). 496–502. 10 indexed citations
5.
Müller, Dominik N., Eva Klopocki, Lars Neumann, et al.. (2006). A complex phenotype with cystic renal disease. Kidney International. 70(9). 1656–1660. 12 indexed citations
6.
Brunner, Bodo, et al.. (1999). Genomic Structure and Comparative Analysis of Nine Fugu Genes: Conservation of Synteny with Human Chromosome Xp22.2–p22.1. Genome Research. 9(5). 437–448. 40 indexed citations
7.
Hamel, Ben C.J., Arie P.T. Smits, Bellinda van den Helm, et al.. (1999). Four families (MRX43, MRX44, MRX45, MRX52) with nonspecific X-linked mental retardation: Clinical and psychometric data and results of linkage analysis. American Journal of Medical Genetics. 85(3). 290–304. 25 indexed citations
8.
Hamel, Ben C.J., Pieter Wesseling, W.O. Renier, et al.. (1999). A new X linked neurodegenerative syndrome with mental retardation, blindness, convulsions, spasticity, mild hypomyelination, and early death maps to the pericentromeric region. Journal of Medical Genetics. 36(2). 140–143. 5 indexed citations
9.
Vikkula, Miikka, Vincent Chi Hang Lui, N.I. Zhidkova, et al.. (1995). Autosomal dominant and recessive osteochondrodysplasias associated with the COL11A2 locus. Cell. 80(3). 431–437. 292 indexed citations
10.
Kremer, Hannie, Patrick L.J.M. Zeeuwen, W.H. Irwin McLean, et al.. (1994). Ichthyosis Bullosa of Siemens Is Caused by Mutations in the Keratin 2e Gene. Journal of Investigative Dermatology. 103(3). 286–289. 81 indexed citations
11.
Berger, Wilhelm, A Meindl, T.J.R. van de Pol, et al.. (1992). Berger, W. et al. Isolation of a candidate gene for Norrie disease by positional cloning. Nature Genet. 1, 199-203. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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