Hermann Bauer

1.2k total citations
18 papers, 812 citations indexed

About

Hermann Bauer is a scholar working on Molecular Biology, Genetics and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Hermann Bauer has authored 18 papers receiving a total of 812 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Genetics and 4 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Hermann Bauer's work include Developmental Biology and Gene Regulation (6 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers) and Reproductive Biology and Fertility (4 papers). Hermann Bauer is often cited by papers focused on Developmental Biology and Gene Regulation (6 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers) and Reproductive Biology and Fertility (4 papers). Hermann Bauer collaborates with scholars based in Germany, United States and France. Hermann Bauer's co-authors include Matthias Hammerschmidt, Bernhard G. Herrmann, Marc Hild, Robert Geisler, Zsolt Lele, Gerd-Jörg Rauch, William S. Talbot, Yoshiyuki Imai, Tewis Bouwmeester and Alexander F. Schier and has published in prestigious journals such as Nature Communications, Nature Genetics and Genes & Development.

In The Last Decade

Hermann Bauer

17 papers receiving 794 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hermann Bauer Germany 13 638 215 178 56 52 18 812
Minori Shinya Japan 14 782 1.2× 253 1.2× 240 1.3× 49 0.9× 39 0.8× 26 1.0k
Zhen‐Yu She China 14 421 0.7× 218 1.0× 243 1.4× 59 1.1× 63 1.2× 33 668
Gordon Polevoy Canada 10 923 1.4× 134 0.6× 445 2.5× 48 0.9× 45 0.9× 13 1.2k
Keiji Itoh United States 19 1.5k 2.4× 333 1.5× 404 2.3× 32 0.6× 50 1.0× 39 1.7k
Chiyo Takagi Japan 17 991 1.6× 264 1.2× 147 0.8× 114 2.0× 99 1.9× 27 1.3k
Sakura Saburi Japan 9 654 1.0× 216 1.0× 337 1.9× 37 0.7× 32 0.6× 16 815
Harma Feitsma Netherlands 11 578 0.9× 152 0.7× 209 1.2× 38 0.7× 17 0.3× 19 810
Hiroki Nagai Japan 16 413 0.6× 183 0.9× 132 0.7× 29 0.5× 73 1.4× 31 624
Ufuk Günesdogan United Kingdom 12 793 1.2× 191 0.9× 114 0.6× 88 1.6× 100 1.9× 18 976
Norma T. Takaesu United States 15 1.1k 1.6× 367 1.7× 116 0.7× 118 2.1× 77 1.5× 29 1.3k

Countries citing papers authored by Hermann Bauer

Since Specialization
Citations

This map shows the geographic impact of Hermann Bauer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hermann Bauer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hermann Bauer more than expected).

Fields of papers citing papers by Hermann Bauer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hermann Bauer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hermann Bauer. The network helps show where Hermann Bauer may publish in the future.

Co-authorship network of co-authors of Hermann Bauer

This figure shows the co-authorship network connecting the top 25 collaborators of Hermann Bauer. A scholar is included among the top collaborators of Hermann Bauer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hermann Bauer. Hermann Bauer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Zhang, Lucy, Björn Brändl, Hermann Bauer, et al.. (2025). Acquisition of ampliconic sequences marks a selfish mouse t-haplotype. Nature Communications. 17(1). 21–21.
2.
Chang, Yu‐Chen, Chih‐Wei Chen, Reika Tei, et al.. (2023). NME3 binds to phosphatidic acid and mediates PLD6-induced mitochondrial tethering. The Journal of Cell Biology. 222(10). 15 indexed citations
3.
Malcher, Agnieszka, Hermann Bauer, Tomasz Stokowy, et al.. (2023). ESX1 gene as a potential candidate responsible for male infertility in nonobstructive azoospermia. Scientific Reports. 13(1). 16563–16563. 4 indexed citations
4.
Lipkowitz, Bettina, et al.. (2019). Two isoforms of the RAC-specific guanine nucleotide exchange factor TIAM2 act oppositely on transmission ratio distortion by the mouse t-haplotype. PLoS Genetics. 15(2). e1007964–e1007964. 19 indexed citations
5.
Liu, Jinhua, et al.. (2017). Patterning and gastrulation defects caused by thetw18lethal are due to loss ofPpp2r1a. Biology Open. 6(6). 752–764. 16 indexed citations
6.
Liao, Juan, Humberto Jijon, Gautam Goel, et al.. (2014). An Image-Based Genetic Assay Identifies Genes in T1D Susceptibility Loci Controlling Cellular Antiviral Immunity in Mouse. PLoS ONE. 9(9). e108777–e108777. 4 indexed citations
7.
Bauer, Hermann, et al.. (2012). The Nucleoside Diphosphate Kinase Gene Nme3 Acts as Quantitative Trait Locus Promoting Non-Mendelian Inheritance. PLoS Genetics. 8(3). e1002567–e1002567. 33 indexed citations
8.
Herrmann, Bernhard G. & Hermann Bauer. (2012). The mouse t-haplotype: a selfish chromosome-genetics molecular mechanism and evollution. MPG.PuRe (Max Planck Society). 297–314. 9 indexed citations
9.
Bauer, Hermann, et al.. (2009). Retention of gene products in syncytial spermatids promotes non-Mendelian inheritance as revealed by the t complex responder. Genes & Development. 23(23). 2705–2710. 36 indexed citations
11.
Bauer, Hermann, et al.. (2005). The t complex–encoded GTPase-activating protein Tagap1 acts as a transmission ratio distorter in mice. Nature Genetics. 37(9). 969–973. 64 indexed citations
12.
Garcia, Dana, et al.. (2005). Identification of keratins and analysis of their expression in carp and goldfish: comparison with the zebrafish and trout keratin catalog. Cell and Tissue Research. 322(2). 245–256. 22 indexed citations
13.
Kramer, Carina, Thomas Mayr, Matthias Nowak, et al.. (2002). Maternally Supplied Smad5 Is Required for Ventral Specification in Zebrafish Embryos Prior to Zygotic Bmp Signaling. Developmental Biology. 250(2). 263–279. 62 indexed citations
14.
Bauer, Hermann, Zsolt Lele, Gerd-Jörg Rauch, Robert Geisler, & Matthias Hammerschmidt. (2001). The type I serine/threonine kinase receptor Alk8/Lost-a-fin is required for Bmp2b/7 signal transduction during dorsoventral patterning of the zebrafish embryo. Development. 128(6). 849–858. 137 indexed citations
15.
Dick, Alexander, et al.. (2000). Cloning and characterization of zebrafish smad2, smad3 and smad4. Gene. 246(1-2). 69–80. 57 indexed citations
16.
Hild, Marc, Alexander Dick, Hermann Bauer, et al.. (2000). The Roles of BMPs, BMP Antagonists, and the BMP Signaling Transducers Smad1 and Smad5 During Dorsoventral Patterning of the Zebrafish Embryo. PubMed. 81–106. 9 indexed citations
17.
Dick, Alexander, Marc Hild, Hermann Bauer, et al.. (2000). Essential role of Bmp7 (snailhouse) and its prodomain in dorsoventral patterning of the zebrafish embryo. Development. 127(2). 343–354. 181 indexed citations
18.
Bauer, Hermann, Marc Hild, Scott E. Stachel, et al.. (1998). Follistatin and Noggin Are Excluded from the Zebrafish Organizer. Developmental Biology. 204(2). 488–507. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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