Hequan Li

1.3k total citations · 1 hit paper
16 papers, 1.0k citations indexed

About

Hequan Li is a scholar working on Physiology, Immunology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Hequan Li has authored 16 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Physiology, 8 papers in Immunology and 5 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Hequan Li's work include Asthma and respiratory diseases (10 papers), IL-33, ST2, and ILC Pathways (4 papers) and Immune Response and Inflammation (3 papers). Hequan Li is often cited by papers focused on Asthma and respiratory diseases (10 papers), IL-33, ST2, and ILC Pathways (4 papers) and Immune Response and Inflammation (3 papers). Hequan Li collaborates with scholars based in China, Croatia and France. Hequan Li's co-authors include Yanmei Han, Minggang Zhang, Xuetao Cao, Jianying Zhou, Dajing Xia, Qīng Wáng, Guohua Lu, Dajing Xia, Yinan Yao and Wanjun Yu and has published in prestigious journals such as The Journal of Immunology, American Journal of Respiratory and Critical Care Medicine and Frontiers in Immunology.

In The Last Decade

Hequan Li

16 papers receiving 998 citations

Hit Papers

Cancer-Expanded Myeloid-Derived Suppressor Cells Induce A... 2009 2026 2014 2020 2009 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Cancer-Expanded Myeloid-Derived Suppressor Cells Induce Anergy of NK Cells through Membrane-Bound TGF-β1
    2009 639 citations Hequan Li, Yanmei Han et al. The Journal of Immunology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hequan Li China 12 705 305 183 183 136 16 1.0k
Erica M. Hanson United States 7 849 1.2× 344 1.1× 222 1.2× 119 0.7× 52 0.4× 8 1.1k
Hirokazu Kurata Japan 12 731 1.0× 230 0.8× 315 1.7× 180 1.0× 39 0.3× 12 1.1k
Anneke H. van Houwelingen Netherlands 8 267 0.4× 121 0.4× 161 0.9× 231 1.3× 244 1.8× 12 771
Ann E. Kelly-Welch United States 9 383 0.5× 104 0.3× 185 1.0× 180 1.0× 55 0.4× 10 672
Samuel L. Collins United States 16 398 0.6× 153 0.5× 331 1.8× 112 0.6× 358 2.6× 34 1.1k
Kristina Lejon Sweden 14 764 1.1× 151 0.5× 207 1.1× 49 0.3× 28 0.2× 35 1.2k
Charlotte G. Smith United Kingdom 8 259 0.4× 220 0.7× 147 0.8× 156 0.9× 189 1.4× 14 682
Tomoko Tsuchiya Japan 17 216 0.3× 162 0.5× 471 2.6× 186 1.0× 55 0.4× 34 882
Kelly A. Remedios United States 8 442 0.6× 124 0.4× 234 1.3× 72 0.4× 38 0.3× 8 790
Patricia Rafferty United States 10 252 0.4× 201 0.7× 370 2.0× 79 0.4× 46 0.3× 13 642

Countries citing papers authored by Hequan Li

Since Specialization
Citations

This map shows the geographic impact of Hequan Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hequan Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hequan Li more than expected).

Fields of papers citing papers by Hequan Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hequan Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hequan Li. The network helps show where Hequan Li may publish in the future.

Co-authorship network of co-authors of Hequan Li

This figure shows the co-authorship network connecting the top 25 collaborators of Hequan Li. A scholar is included among the top collaborators of Hequan Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hequan Li. Hequan Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Li, Hequan, et al.. (2023). How does cigarette smoking affect airway remodeling inasthmatics?. Tobacco Induced Diseases. 21(January). 1–14. 12 indexed citations
2.
Li, Hequan, et al.. (2023). T-helper cells and their cytokines in pathogenesis and treatment of asthma. Frontiers in Immunology. 14. 1149203–1149203. 54 indexed citations
3.
Huang, Bo, et al.. (2018). Ibrutinib Exacerbates Bleomycin-Induced Pulmonary Fibrosis via Promoting Inflammation. Inflammation. 41(3). 904–913. 12 indexed citations
4.
Wáng, Qīng, Hequan Li, Yinan Yao, et al.. (2016). HB-EGF–Promoted Airway Smooth Muscle Cells and Their Progenitor Migration Contribute to Airway Smooth Muscle Remodeling in Asthmatic Mouse. The Journal of Immunology. 196(5). 2361–2367. 23 indexed citations
5.
Lu, Shan, Hequan Li, Xuan Gao, et al.. (2015). IL-17A, But Not IL-17F, Is Indispensable for Airway Vascular Remodeling Induced by Exaggerated Th17 Cell Responses in Prolonged Ovalbumin-Challenged Mice. The Journal of Immunology. 194(8). 3557–3566. 23 indexed citations
6.
Zhang, Pei, Yihong Shen, Hequan Li, et al.. (2015). Mannose prevents acute lung injury through mannose receptor pathway and contributes to regulate PPARγ and TGF-β1 level.. PubMed. 8(6). 6214–24. 13 indexed citations
7.
8.
Yang, Guangdie, Hequan Li, Yinan Yao, et al.. (2014). Treg/Th17 imbalance in malignant pleural effusion partially predicts poor prognosis. Oncology Reports. 33(1). 478–484. 23 indexed citations
9.
Yao, Yinan, Shan Lu, Hequan Li, Guohua Lu, & Jianying Zhou. (2012). Low Doses of Exogenous Interferon-γ Attenuated Airway Inflammation Through Enhancing Fas/FasL-Induced CD4 + T Cell Apoptosis in a Mouse Asthma Model. Journal of Interferon & Cytokine Research. 32(11). 534–541. 5 indexed citations
10.
Zhang, Cheng–Hai, Yinchuan Li, Wei Zhao, et al.. (2012). The Transmembrane Protein 16A Ca2+-activated Cl− Channel in Airway Smooth Muscle Contributes to Airway Hyperresponsiveness. American Journal of Respiratory and Critical Care Medicine. 187(4). 374–381. 63 indexed citations
11.
Wang, Huaying, et al.. (2012). Imbalance of Th17/Treg cells in mice with chronic cigarette smoke exposure. International Immunopharmacology. 14(4). 504–512. 59 indexed citations
12.
Wang, Qing, Hequan Li, Zhiyuan Zhang, Yinan Yao, & Jianying Zhou. (2010). Prolonged ovalbumin challenge facilitates Th17 polarization in sensitized mice. Inflammation Research. 59(7). 561–569. 5 indexed citations
13.
Wáng, Qīng, et al.. (2010). The Overexpression of Heparin-Binding Epidermal Growth Factor Is Responsible for Th17-Induced Airway Remodeling in an Experimental Asthma Model. The Journal of Immunology. 185(2). 834–841. 65 indexed citations
14.
Li, Hequan, et al.. (2009). Cancer-Expanded Myeloid-Derived Suppressor Cells Induce Anergy of NK Cells through Membrane-Bound TGF-β1. The Journal of Immunology. 182(1). 240–249. 639 indexed citations breakdown →
15.
16.
Zhang, Lihong, et al.. (2001). Effects of extract F of red-rooted Salvia on mucosal lesions of gastric corpus and antrum induced by hemorrhagic shock-reperfusion in rats. World Journal of Gastroenterology. 7(4). 672–672. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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