Henry Hennings

8.3k total citations · 1 hit paper
118 papers, 7.1k citations indexed

About

Henry Hennings is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Henry Hennings has authored 118 papers receiving a total of 7.1k indexed citations (citations by other indexed papers that have themselves been cited), including 63 papers in Molecular Biology, 30 papers in Cell Biology and 25 papers in Cancer Research. Recurrent topics in Henry Hennings's work include Skin and Cellular Biology Research (22 papers), Carcinogens and Genotoxicity Assessment (19 papers) and Polyamine Metabolism and Applications (11 papers). Henry Hennings is often cited by papers focused on Skin and Cellular Biology Research (22 papers), Carcinogens and Genotoxicity Assessment (19 papers) and Polyamine Metabolism and Applications (11 papers). Henry Hennings collaborates with scholars based in United States, Norway and India. Henry Hennings's co-authors include Stuart H. Yuspa, Karen A. Holbrook, Delores Michael, Peter M. Steinert, Christina Cheng, Ulrike Lichti, K A Holbrook, Theresa Ben, K. Elgjo and R. K. Boutwell and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Henry Hennings

115 papers receiving 6.6k citations

Hit Papers

Calcium regulation of growth and differentiation of mouse... 1980 2026 1995 2010 1980 500 1000 1.5k

Peers

Henry Hennings
Estela E. Medrano United States
Ulrike Lichti United States
Goberdhan P. Dimri United States
Mitchell F. Denning United States
Claudio J. Conti United States
Ivica Rubelj Croatia
Ambra Pozzi United States
Estela E. Medrano United States
Henry Hennings
Citations per year, relative to Henry Hennings Henry Hennings (= 1×) peers Estela E. Medrano

Countries citing papers authored by Henry Hennings

Since Specialization
Citations

This map shows the geographic impact of Henry Hennings's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Henry Hennings with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Henry Hennings more than expected).

Fields of papers citing papers by Henry Hennings

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Henry Hennings. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Henry Hennings. The network helps show where Henry Hennings may publish in the future.

Co-authorship network of co-authors of Henry Hennings

This figure shows the co-authorship network connecting the top 25 collaborators of Henry Hennings. A scholar is included among the top collaborators of Henry Hennings based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Henry Hennings. Henry Hennings is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cataisson, Christophe, et al.. (2008). Inducible Cutaneous Inflammation Reveals a Protumorigenic Role for Keratinocyte CXCR2 in Skin Carcinogenesis. Cancer Research. 69(1). 319–328. 57 indexed citations
2.
Glick, Adam B., Andrew Ryscavage, Rolando Pérez‐Lorenzo, et al.. (2007). The high‐risk benign tumor: Evidence from the two‐stage skin cancer model and relevance for human cancer. Molecular Carcinogenesis. 46(8). 605–610. 17 indexed citations
3.
Darwiche, Nadine, Andrew Ryscavage, Rolando Pérez‐Lorenzo, et al.. (2007). Expression profile of skin papillomas with high cancer risk displays a unique genetic signature that clusters with squamous cell carcinomas and predicts risk for malignant conversion. Oncogene. 26(48). 6885–6895. 37 indexed citations
4.
Abdel‐Fattah, Rana, et al.. (2005). Methylation of the O6‐methylguanine‐DNA methyltransferase promoter suppresses expression in mouse skin tumors and varies with the tumor induction protocol. International Journal of Cancer. 118(3). 527–531. 6 indexed citations
5.
Strickland, James E., Andrzej A. Dlugosz, Henry Hennings, & S H Yuspa. (1993). Inhibition of tumor formation from grafted murine papilloma cells by treatment of grafts with staurosporine, an inducer of squamous differentiation. Carcinogenesis. 14(2). 205–209. 21 indexed citations
6.
Hennings, Henry, et al.. (1993). FVB/N mice: an inbred strain sensitive to the chemical induction of squamous cell carcinomas in the skin. Carcinogenesis. 14(11). 2353–2358. 137 indexed citations
7.
Hennings, Henry, Adam B. Glick, David A. Greenhalgh, et al.. (1993). Critical Aspects of Initiation, Promotion, and Progression in Multistage Epidermal Carcinogenesis. Experimental Biology and Medicine. 202(1). 1–8. 97 indexed citations
8.
Hennings, Henry, David T. Lowry, Victor Robinson, et al.. (1992). Activity of diverse tumor promoters in a keratinocyte co-culture model of initiated epidermis. Carcinogenesis. 13(11). 2145–2151. 28 indexed citations
9.
Yuspa, Stuart H., Anne Kilkenny, Dennis R. Roop, et al.. (1991). Alterations in epidermal biochemistry as a consequence of stage-specific genetic changes in skin carcinogenesis.. Environmental Health Perspectives. 93. 3–10. 15 indexed citations
10.
12.
Hennings, Henry, et al.. (1990). Enhanced Malignant Conversion of Benign Mouse Skin Tumors by Cisplatin. JNCI Journal of the National Cancer Institute. 82(10). 836–840. 23 indexed citations
13.
Yuspa, Stuart H., Henry Hennings, Dennis R. Roop, J.H. Strickland, & David A. Greenhalgh. (1990). The malignant conversion step of mouse skin carcinogenesis.. Environmental Health Perspectives. 88. 193–195. 8 indexed citations
14.
Hennings, Henry. (1989). Malignant conversion: the first stage in progression from benign to malignant tumors.. PubMed. 298. 95–100. 3 indexed citations
15.
Yuspa, Stuart H., Henry Hennings, Robert W. Tucker, et al.. (1988). Signal Transduction for Proliferation and Differentiation in Keratinocytes. Annals of the New York Academy of Sciences. 548(1). 191–196. 93 indexed citations
16.
Hennings, Henry, Edwin F. Spangler, Deborah E. Devor, et al.. (1986). Malignant conversion and metastasis of mouse skin tumors: a comparison of SENCAR and CD-1 mice.. Environmental Health Perspectives. 68. 69–74. 21 indexed citations
17.
Ward, Jerrold M., Sabine Rehm, Deborah E. Devor, Henry Hennings, & Martin L. Wenk. (1986). Differential carcinogenic effects of intraperitoneal initiation with 7,12-dimethylbenz(a)anthracene or urethane and topical promotion with 12-O-tetradecanoylphorbol-13-acetate in skin and internal tissues of female SENCAR and BALB/c mice.. Environmental Health Perspectives. 68. 61–68. 16 indexed citations
18.
Hennings, Henry, et al.. (1982). Susceptibility determinants for mouse epidermal carcinogenesis.. PubMed. 259–68. 4 indexed citations
19.
Sh, Yuspa, Henry Hennings, Molly Kulesz‐Martin, & Ulrike Lichti. (1982). The study of tumor promotion in a cell culture model for mouse skin--a tissue that exhibits multistage carcinogenesis in vivo.. PubMed. 7. 217–30. 18 indexed citations
20.
Kulesz‐Martin, Molly, Barbara Koehler, Henry Hennings, & S H Yuspa. (1980). Quantitative assay for carcinogen altered differentiation in mouse epidermal cells. Carcinogenesis. 1(12). 995–1006. 72 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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