Henri Bernardi

6.4k total citations
31 papers, 1.8k citations indexed

About

Henri Bernardi is a scholar working on Molecular Biology, Physiology and Pathology and Forensic Medicine. According to data from OpenAlex, Henri Bernardi has authored 31 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 8 papers in Physiology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in Henri Bernardi's work include Muscle Physiology and Disorders (18 papers), Ion channel regulation and function (7 papers) and Adipose Tissue and Metabolism (6 papers). Henri Bernardi is often cited by papers focused on Muscle Physiology and Disorders (18 papers), Ion channel regulation and function (7 papers) and Adipose Tissue and Metabolism (6 papers). Henri Bernardi collaborates with scholars based in France, Morocco and Switzerland. Henri Bernardi's co-authors include Anthony M. J. Sanchez, Robin Candau, Michel Lazdunski, Michel Fosset, Audrey Raibon, Barbara Vernus, Christiane Mourre, Alfredo Csibi, Gérard Cabello and Yehezkel Ben‐Ari and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Physiology and Biochemistry.

In The Last Decade

Henri Bernardi

30 papers receiving 1.8k citations

Peers

Henri Bernardi
Emanuele Loro United States
Lionel Tintignac Switzerland
Michael Sinnreich Switzerland
Jianxun Yi United States
Emanuele Loro United States
Henri Bernardi
Citations per year, relative to Henri Bernardi Henri Bernardi (= 1×) peers Emanuele Loro

Countries citing papers authored by Henri Bernardi

Since Specialization
Citations

This map shows the geographic impact of Henri Bernardi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Henri Bernardi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Henri Bernardi more than expected).

Fields of papers citing papers by Henri Bernardi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Henri Bernardi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Henri Bernardi. The network helps show where Henri Bernardi may publish in the future.

Co-authorship network of co-authors of Henri Bernardi

This figure shows the co-authorship network connecting the top 25 collaborators of Henri Bernardi. A scholar is included among the top collaborators of Henri Bernardi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Henri Bernardi. Henri Bernardi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bertrand‐Gaday, Christelle, et al.. (2025). Acute effects of heat intervention and hybrid exercise on protein synthesis, ribosome biogenesis and autophagy. Journal of Thermal Biology. 131. 104169–104169.
2.
Zampeli, Frantzeska, Anthony M. J. Sanchez, Marco Cardinale, et al.. (2025). Muscle regeneration is improved by hot water immersion but unchanged by cold following a simulated musculoskeletal injury in humans. The Journal of Physiology. 603(23). 7603–7625. 4 indexed citations
3.
Borrani, Fabio, et al.. (2024). DNA methylation changes during a sprint interval exercise performed under normobaric hypoxia or with blood flow restriction: A pilot study in men. Physiological Reports. 12(11). e16044–e16044. 2 indexed citations
4.
Racinais, Sébastien, et al.. (2024). Effects of thermal interventions on skeletal muscle adaptations and regeneration: perspectives on epigenetics: a narrative review. European Journal of Applied Physiology. 125(2). 277–301. 5 indexed citations
5.
Docquier, Aurélie, Audrey Raibon, Christelle Bertrand‐Gaday, et al.. (2019). eIF3f depletion impedes mouse embryonic development, reduces adult skeletal muscle mass and amplifies muscle loss during disuse. The Journal of Physiology. 597(12). 3107–3131. 16 indexed citations
6.
Sanchez, Anthony M. J., Robin Candau, & Henri Bernardi. (2018). AMP-activated protein kinase stabilizes FOXO3 in primary myotubes. Biochemical and Biophysical Research Communications. 499(3). 493–498. 22 indexed citations
7.
Pagano, Allan F., Guillaume Py, Henri Bernardi, Robin Candau, & Anthony M. J. Sanchez. (2014). Autophagy and Protein Turnover Signaling in Slow-Twitch Muscle during Exercise. Medicine & Science in Sports & Exercise. 46(7). 1314–1325. 67 indexed citations
8.
Sanchez, Anthony M. J., Henri Bernardi, Guillaume Py, & Robin Candau. (2014). Autophagy is essential to support skeletal muscle plasticity in response to endurance exercise. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 307(8). R956–R969. 106 indexed citations
9.
Sanchez, Anthony M. J., Alfredo Csibi, Audrey Raibon, et al.. (2013). eIF3f: A central regulator of the antagonism atrophy/hypertrophy in skeletal muscle. The International Journal of Biochemistry & Cell Biology. 45(10). 2158–2162. 41 indexed citations
10.
Sanchez, Anthony M. J., Alfredo Csibi, Audrey Raibon, et al.. (2011). AMPK promotes skeletal muscle autophagy through activation of forkhead FoxO3a and interaction with Ulk1. Journal of Cellular Biochemistry. 113(2). 695–710. 257 indexed citations
11.
Descamps, Simon, Francis Bacou, Henri Bernardi, et al.. (2008). Inhibition of myoblast differentiation by Sfrp1 and Sfrp2. Cell and Tissue Research. 332(2). 299–306. 44 indexed citations
12.
Navarro, Magali, et al.. (2008). The atypical α2β2 IGF receptor expressed in inducible c2.7 myoblasts is derived from post-translational modifications of the mouse IGF-I receptor. Growth Hormone & IGF Research. 18(5). 412–423. 3 indexed citations
13.
Vernus, Barbara, Michel Jacques M.J. Duclos, Henri Bernardi, et al.. (2003). Inhibition of autocrine secretion of myostatin enhances terminal differentiation in human rhabdomyosarcoma cells. Oncogene. 22(51). 8221–8232. 26 indexed citations
14.
Bernardi, Henri, et al.. (2003). Mechanisms involved in the inhibition of myoblast proliferation and differentiation by myostatin. Experimental Cell Research. 286(2). 263–275. 267 indexed citations
16.
Talesa, Vincenzo Nicola, et al.. (1995). Characterization of a null mutation in ace‐1, the gene encoding class A acetylcholinesterase in the nematode Caenorhabditis elegans. FEBS Letters. 357(3). 265–268. 13 indexed citations
17.
Lazdunski, Michel, et al.. (1994). ATP-Sensitive K<sup>+</sup> Channels. Kidney & Blood Pressure Research. 17(3-4). 118–120. 15 indexed citations
18.
Bernardi, Henri, Michel Fosset, & Michel Lazdunski. (1992). ATP/ADP binding sites are present in the sulfonylurea binding protein associated with brain ATP-sensitive potassium channels. Biochemistry. 31(27). 6328–6332. 29 indexed citations
19.
Weille, Jan R. de, et al.. (1989). Pharmacology and regulation of ATP-sensitive K+ channels. Pflügers Archiv - European Journal of Physiology. 414(S1). S80–S87. 46 indexed citations
20.
Mourre, Christiane, Yehezkel Ben‐Ari, Henri Bernardi, Michel Fosset, & Michel Lazdunski. (1989). Antidiabetic sulfonylureas: localization of binding sites in the brain and effects on the hyperpolarization induced by anoxia in hippocampal slices. Brain Research. 486(1). 159–164. 211 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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