Anna Vainshtein

2.2k total citations
29 papers, 1.7k citations indexed

About

Anna Vainshtein is a scholar working on Physiology, Molecular Biology and Epidemiology. According to data from OpenAlex, Anna Vainshtein has authored 29 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 14 papers in Molecular Biology and 12 papers in Epidemiology. Recurrent topics in Anna Vainshtein's work include Adipose Tissue and Metabolism (14 papers), Autophagy in Disease and Therapy (12 papers) and Mitochondrial Function and Pathology (8 papers). Anna Vainshtein is often cited by papers focused on Adipose Tissue and Metabolism (14 papers), Autophagy in Disease and Therapy (12 papers) and Mitochondrial Function and Pathology (8 papers). Anna Vainshtein collaborates with scholars based in Canada, United States and Italy. Anna Vainshtein's co-authors include David A. Hood, Marco Sandri, Liam D. Tryon, Marion Pauly, Michael F. N. O′Leary, Andrea Armani, Elior Peles, Paolo Grumati, Silvia Carnio and Galia Maik-Rachline and has published in prestigious journals such as Nature Communications, Neuron and Journal of Neuroscience.

In The Last Decade

Anna Vainshtein

28 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anna Vainshtein Canada 20 1.0k 805 558 303 186 29 1.7k
Andreas Schild Switzerland 11 1.2k 1.2× 602 0.7× 596 1.1× 339 1.1× 204 1.1× 13 1.7k
Luisa Coletto Italy 12 1.0k 1.0× 527 0.7× 571 1.0× 295 1.0× 189 1.0× 12 1.5k
Henri Bernardi France 18 1.4k 1.3× 544 0.7× 330 0.6× 237 0.8× 318 1.7× 31 1.8k
Joshua C. Drake United States 19 880 0.9× 579 0.7× 359 0.6× 254 0.8× 56 0.3× 41 1.4k
Jessica Segalés Spain 15 1.4k 1.3× 582 0.7× 341 0.6× 259 0.9× 95 0.5× 16 1.7k
Étienne Mouisel France 24 1.2k 1.2× 820 1.0× 272 0.5× 383 1.3× 247 1.3× 36 2.1k
Bernat Baeza-Raja United States 13 1.1k 1.0× 564 0.7× 167 0.3× 258 0.9× 162 0.9× 15 1.8k
Perrine Castets Switzerland 15 735 0.7× 398 0.5× 240 0.4× 224 0.7× 203 1.1× 21 1.1k
Leslie M. Baehr United States 23 1.9k 1.8× 978 1.2× 227 0.4× 725 2.4× 294 1.6× 33 2.5k
Yuho Kim United States 12 579 0.6× 433 0.5× 174 0.3× 138 0.5× 61 0.3× 21 905

Countries citing papers authored by Anna Vainshtein

Since Specialization
Citations

This map shows the geographic impact of Anna Vainshtein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Vainshtein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Vainshtein more than expected).

Fields of papers citing papers by Anna Vainshtein

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna Vainshtein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Vainshtein. The network helps show where Anna Vainshtein may publish in the future.

Co-authorship network of co-authors of Anna Vainshtein

This figure shows the co-authorship network connecting the top 25 collaborators of Anna Vainshtein. A scholar is included among the top collaborators of Anna Vainshtein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna Vainshtein. Anna Vainshtein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Vainshtein, Anna, et al.. (2025). Autophagy, ER-phagy and ER Dynamics During Cell Differentiation. Journal of Molecular Biology. 437(18). 169151–169151. 1 indexed citations
2.
Vainshtein, Anna, et al.. (2024). Age-dependent regulation of axoglial interactions and behavior by oligodendrocyte AnkyrinG. Nature Communications. 15(1). 10865–10865.
3.
Ogawa, Yuki, et al.. (2024). TRIM46 Is Required for Microtubule Fasciculation In Vivo But Not Axon Specification or Axon Initial Segment Formation. Journal of Neuroscience. 44(42). e0976242024–e0976242024. 4 indexed citations
4.
Vainshtein, Anna, Arthur J. Cheng, Jonathan M. Memme, et al.. (2022). Scientific meeting report: International Biochemistry of Exercise 2022. Journal of Applied Physiology. 133(6). 1381–1393. 1 indexed citations
5.
Steinberg, Daniel, Anna Vainshtein, Yael Eshed‐Eisenbach, et al.. (2021). Neuronal deletion of Wwox, associated with WOREE syndrome, causes epilepsy and myelin defects. Brain. 144(10). 3061–3077. 21 indexed citations
6.
Chang, Kae-Jiun, Ira Agrawal, Anna Vainshtein, et al.. (2021). TDP-43 maximizes nerve conduction velocity by repressing a cryptic exon for paranodal junction assembly in Schwann cells. eLife. 10. 15 indexed citations
7.
Eshed‐Eisenbach, Yael, Jérôme Devaux, Anna Vainshtein, et al.. (2020). Precise Spatiotemporal Control of Nodal Na+ Channel Clustering by Bone Morphogenetic Protein-1/Tolloid-like Proteinases. Neuron. 106(5). 806–815.e6. 10 indexed citations
8.
Vainshtein, Anna & Paolo Grumati. (2020). Selective Autophagy by Close Encounters of the Ubiquitin Kind. Cells. 9(11). 2349–2349. 32 indexed citations
9.
Pastore, Nunzia, Anna Vainshtein, Niculin J. Herz, et al.. (2019). Nutrient‐sensitive transcription factors TFEB and TFE 3 couple autophagy and metabolism to the peripheral clock. The EMBO Journal. 38(12). 59 indexed citations
10.
Pastore, Nunzia, Anna Vainshtein, Tiemo J. Klisch, et al.. (2017). TFE 3 regulates whole‐body energy metabolism in cooperation with TFEB. EMBO Molecular Medicine. 9(5). 605–621. 107 indexed citations
11.
Klisch, Tiemo J., Anna Vainshtein, Akash J. Patel, & Huda Y. Zoghbi. (2017). Jak2-mediated phosphorylation of Atoh1 is critical for medulloblastoma growth. eLife. 6. 15 indexed citations
12.
Yang, Hyun‐Jeong, Anna Vainshtein, Galia Maik-Rachline, & Elior Peles. (2016). G protein-coupled receptor 37 is a negative regulator of oligodendrocyte differentiation and myelination. Nature Communications. 7(1). 10884–10884. 114 indexed citations
13.
Hood, David A., Liam D. Tryon, Anna Vainshtein, et al.. (2015). Exercise and the Regulation of Mitochondrial Turnover. Progress in molecular biology and translational science. 135. 99–127. 33 indexed citations
14.
Vainshtein, Anna, Eric M. Desjardins, Andrea Armani, Marco Sandri, & David A. Hood. (2015). PGC-1α modulates denervation-induced mitophagy in skeletal muscle. Skeletal Muscle. 5(1). 9–9. 146 indexed citations
15.
Vainshtein, Anna, Liam D. Tryon, Marion Pauly, & David A. Hood. (2015). Acute Exercise‐Induced Mitophagy is Mediated in Part by PGC‐1α. The FASEB Journal. 29(S1). 1 indexed citations
17.
Tryon, Liam D., et al.. (2014). Recent advances in mitochondrial turnover during chronic muscle disuse. Integrative Medicine Research. 3(4). 161–171. 28 indexed citations
18.
Vainshtein, Anna, Paolo Grumati, Marco Sandri, & Paolo Bonaldo. (2013). Skeletal muscle, autophagy, and physical activity: the ménage à trois of metabolic regulation in health and disease. Journal of Molecular Medicine. 92(2). 127–137. 68 indexed citations
19.
Hood, David A., Giulia Uguccioni, Anna Vainshtein, & Donna M. D’Souza. (2011). Mechanisms of Exercise‐Induced Mitochondrial Biogenesis in Skeletal Muscle: Implications for Health and Disease. Comprehensive physiology. 1(3). 1119–1134. 75 indexed citations
20.
Hood, David A., Giulia Uguccioni, Anna Vainshtein, & Donna M. D’Souza. (2011). Mechanisms of Exercise‐Induced Mitochondrial Biogenesis in Skeletal Muscle: Implications for Health and Disease. Comprehensive physiology. 1(3). 1119–1134. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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