Hemal H. Mehta

3.5k citations
40 papers · 2.4k indexed · 1 hit paper · h-index 23

Hemal H. Mehta

36 papers receiving 2.4k citations

Hit Papers

The Mitochondrial-Derived Peptide MOTS-c Promotes Metabol...5112015202620182022100200300400500

Peers

Hemal H. Mehta
Comparison fields: 5 of 104
  • Rheumatology 1.0k
  • Aging 76
  • Physiology 809
  • Immunology 550
  • Molecular Biology 1.3k
Replace Laura J. Cobb with:
Laura J. Cobb United States
Ying Hong United Kingdom
Jakob Voelkl Germany
Osamu Yasuda Japan
Ju Hee Lee South Korea
Elena Ambrogini United States
Florence Gizard France
Tamar Aprahamian United States
Yazmín Macotela Mexico
Hemal H. Mehta relative to Laura J. Cobb United States Laura J. Cobb's profile →
Citations per field
00.5×3.3×
Laura J. Cobb · 1×
Citations per year

Countries citing papers authored by Hemal H. Mehta

Since Specialization
Citations

This map shows the geographic impact of Hemal H. Mehta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hemal H. Mehta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hemal H. Mehta more than expected).

Fields of papers citing papers by Hemal H. Mehta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hemal H. Mehta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hemal H. Mehta. The network helps show where Hemal H. Mehta may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Hemal H. Mehta, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hemal H. Mehta Line = papers co-authored together Hemal H. Mehta links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 20250
3 20247
4 20225
5 202145
6 202126
7 202163
8 20201
9 202056
10 201980
11 201934
12 201872
13 2018108
14 2016208
15
The Mitochondrial-Derived Peptide MOTS-c Promotes Metabolic Homeostasis and Reduces Obesity and Insulin Resistancebreakdown →
2015511
16 2012151
17 201178
18 2007226
19 2006118
20 200413

About Hemal H. Mehta

Hemal H. Mehta is a scholar working on Rheumatology, Physiology and Aging, having authored 40 papers that have together received 2.4k indexed citations. Recurring topics across this work include GDF15 and Related Biomarkers (19 papers), Nutrition and Health in Aging (14 papers), Growth Hormone and Insulin-like Growth Factors (5 papers), Mitochondrial Function and Pathology (3 papers), Lipid metabolism and disorders (3 papers), Metabolism, Diabetes, and Cancer (3 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Apelin-related biomedical research (3 papers). The work is most often cited by research in Rheumatology (1.0k citations), Aging (76 citations) and Physiology (809 citations). Hemal H. Mehta has collaborated with scholars based in United States, Japan and Australia. Frequent co-authors include Pinchas Cohen, Junxiang Wan, Kelvin Yen, Su‐Jeong Kim, Changhan Lee, Mariana J. Kaplan, W. Joseph McCune, Laura J. Cobb, Rafael de Cabo and Tamer Sallam. Their work appears in journals such as Journal of Clinical Oncology, SHILAP Revista de lepidopterología and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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