Hassan Yousefi
- Molecular Biology top 5%
- Cancer Research top 1%
- Immunology top 10%
- Oncology top 10%
- Pulmonary and Respiratory Medicine
- Co-authors
- Suresh K. AlahariLadan MashouriFatemeh MolaeiAli Mohammad AhadiAmir Reza ArefMajid MomenyMohammad Esmaeil AkbariNahid Eskandari
- Topics
- Cancer-related molecular mechanisms research (10 papers)MicroRNA in disease regulation (6 papers)Epigenetics and DNA Methylation (6 papers)
- Partner nations
- United StatesIranFinland
In The Last Decade
Hassan Yousefi
46 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 106
- Molecular Biology 1.8k
- Cancer Research 1.1k
- Immunology 368
- Oncology 299
- Pulmonary and Respiratory Medicine 174
Countries citing papers authored by Hassan Yousefi
This map shows the geographic impact of Hassan Yousefi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hassan Yousefi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hassan Yousefi more than expected).
Fields of papers citing papers by Hassan Yousefi
This network shows the impact of papers produced by Hassan Yousefi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hassan Yousefi. The network helps show where Hassan Yousefi may publish in the future.
Co-authorship network of co-authors of Hassan Yousefi
This figure shows the co-authorship network connecting the top 25 collaborators of Hassan Yousefi. A scholar is included among the top collaborators of Hassan Yousefi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hassan Yousefi. Hassan Yousefi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 6 | |
| 4 | 8 | |
| 5 | 10 | |
| 6 | 31 | |
| 7 | 81 | |
| 8 | 6 | |
| 9 | 14 | |
| 10 | 92 | |
| 11 | 36 | |
| 12 | 32 | |
| 13 | 29 | |
| 14 | 6 | |
| 15 | 32 | |
| 16 | 12 | |
| 17 | 3 | |
| 18 | Exosomes: composition, biogenesis, and mechanisms in cancer metastasis and drug resistancebreakdown → | 1143 |
| 19 | 30 | |
| 20 | Natural Anti-Leishmaniasis Compounds in Traditional Iranian Medicine | 4 |
About Hassan Yousefi
Hassan Yousefi is a scholar working on Cancer Research, Architecture and Oncology, having authored 46 papers that have together received 2.3k indexed citations. Recurring topics across this work include Cancer-related molecular mechanisms research (10 papers), MicroRNA in disease regulation (6 papers) and Epigenetics and DNA Methylation (6 papers). The work is most often cited by research in Cancer Research (1.1k citations), Molecular Biology (1.8k citations) and Immunology (368 citations). Hassan Yousefi has collaborated with scholars based in United States, Iran and Finland. Frequent co-authors include Suresh K. Alahari, Ladan Mashouri, Fatemeh Molaei, Ali Mohammad Ahadi, Amir Reza Aref, Majid Momeny, Amir Reza Aref, Mohammad Esmaeil Akbari, Nahid Eskandari and Amirreza Bitaraf. Their work appears in journals such as Oncogene, Scientific Reports and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.