Hans-Peter Seelig

1.3k citations
10 papers · 1.1k indexed · 1 hit paper · h-index 6

Hans-Peter Seelig

9 papers receiving 1.1k citations

Hit Papers

The Dermatomyositis-Specific Autoantigen Mi2 Is a Compone...6831998202620072016200400600

Peers

Hans-Peter Seelig
Comparison fields: 5 of 71
  • Pathology and Forensic Medicine 216
  • Molecular Biology 759
  • Aging 18
  • Neurology 130
  • Rheumatology 113
Replace Toru Wakioka with:
Toru Wakioka Japan
Michelle Hussain United Kingdom
Kanta Miyoshi Japan
Hetty N. Wong United States
Joseph F. Maher United States
Bieke Broux Belgium
Yukihiko Aikawa Japan
Gillian Borland United Kingdom
Yixue Gu China
David Liberg Sweden
Hans-Peter Seelig relative to Toru Wakioka Japan Toru Wakioka's profile →
Citations per field
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Toru Wakioka · 1×
Citations per year

Countries citing papers authored by Hans-Peter Seelig

Since Specialization
Citations

This map shows the geographic impact of Hans-Peter Seelig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hans-Peter Seelig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hans-Peter Seelig more than expected).

Fields of papers citing papers by Hans-Peter Seelig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hans-Peter Seelig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hans-Peter Seelig. The network helps show where Hans-Peter Seelig may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Hans-Peter Seelig, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Hans-Peter Seelig Line = papers co-authored together Hans-Peter Seelig links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 20220
2 20201
3 20162
4 201631
5 20131
6 2007165
7 2000145
8
The Dermatomyositis-Specific Autoantigen Mi2 Is a Component of a Complex Containing Histone Deacetylase and Nucleosome Remodeling Activitiesbreakdown →
1998683
9 197862
10 19698

About Hans-Peter Seelig

Hans-Peter Seelig is a scholar working on Genetics, Pathology and Forensic Medicine and Immunology, having authored 10 papers that have together received 1.1k indexed citations. Recurring topics across this work include Autoimmune Neurological Disorders and Treatments (1 paper), Glycogen Storage Diseases and Myoclonus (1 paper), Coagulation, Bradykinin, Polyphosphates, and Angioedema (1 paper), Diabetes and associated disorders (1 paper), Myasthenia Gravis and Thymoma (1 paper), Peripheral Neuropathies and Disorders (1 paper), Chronic Lymphocytic Leukemia Research (1 paper) and Acute Lymphoblastic Leukemia research (1 paper). The work is most often cited by research in Pathology and Forensic Medicine (216 citations), Molecular Biology (759 citations) and Aging (18 citations). Hans-Peter Seelig has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Danny Reinberg, William S. Lane, Yi Zhang, Gary LeRoy, Reinhard Hohlfeld, Frauke Zipp, Friedemann Paul, Angela Vincent, Heribert Appelhans and Edward Littleton. Their work appears in journals such as Cell, Molecular and Cellular Biology and PLoS Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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