Hans Wils

2.7k total citations
15 papers, 852 citations indexed

About

Hans Wils is a scholar working on Neurology, Genetics and Molecular Biology. According to data from OpenAlex, Hans Wils has authored 15 papers receiving a total of 852 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Neurology, 6 papers in Genetics and 3 papers in Molecular Biology. Recurrent topics in Hans Wils's work include Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (6 papers) and Alzheimer's disease research and treatments (3 papers). Hans Wils is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (8 papers), Neurogenetic and Muscular Disorders Research (6 papers) and Alzheimer's disease research and treatments (3 papers). Hans Wils collaborates with scholars based in Belgium, Germany and United Kingdom. Hans Wils's co-authors include Christine Van Broeckhoven, Samir Kumar‐Singh, Gernot Kleinberger, Geert Joris, Ivy Cuijt, Jonathan Janssens, Sandra Pereson, Chantal Ceuterick‐de Groote, Debby Van Dam and Bianca Van Broeck and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neurochemistry and Journal of Hepatology.

In The Last Decade

Hans Wils

15 papers receiving 842 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hans Wils Belgium	9	672	337	325	240	153	15	852
Ivy Cuijt Belgium	8	831 1.2×	440 1.3×	417 1.3×	270 1.1×	173 1.1×	12	1.0k
Edward Pokrishevsky Canada	9	671 1.0×	316 0.9×	570 1.8×	191 0.8×	206 1.3×	12	983
Clark R. Restrepo United States	7	493 0.7×	251 0.7×	367 1.1×	159 0.7×	229 1.5×	10	760
SungWon Song United States	7	568 0.8×	352 1.0×	495 1.5×	146 0.6×	230 1.5×	8	1.0k
Wendy Scheveneels Belgium	11	591 0.9×	358 1.1×	456 1.4×	131 0.5×	136 0.9×	13	915
Martin H. Schludi Germany	7	770 1.1×	419 1.2×	498 1.5×	185 0.8×	107 0.7×	7	972
Reika Wate Japan	13	531 0.8×	263 0.8×	319 1.0×	95 0.4×	109 0.7×	27	707
Lillian M. Daughrity United States	9	740 1.1×	432 1.3×	564 1.7×	223 0.9×	129 0.8×	12	1.1k
Sharon Carmona United States	4	652 1.0×	422 1.3×	512 1.6×	153 0.6×	169 1.1×	4	977
Paul W.J. van Vught Netherlands	14	732 1.1×	439 1.3×	395 1.2×	140 0.6×	254 1.7×	22	1.0k

Countries citing papers authored by Hans Wils

Since Specialization

Citations

This map shows the geographic impact of Hans Wils's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hans Wils with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hans Wils more than expected).

Fields of papers citing papers by Hans Wils

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hans Wils. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hans Wils. The network helps show where Hans Wils may publish in the future.

Co-authorship network of co-authors of Hans Wils

This figure shows the co-authorship network connecting the top 25 collaborators of Hans Wils. A scholar is included among the top collaborators of Hans Wils based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hans Wils. Hans Wils is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Venken, Koen, Frederik Stevenaert, Yann Abraham, et al.. (2024). Shared lung and joint T cell repertoire in early rheumatoid arthritis driven by cigarette smoking. Annals of the Rheumatic Diseases. 84(3). 409–420. 1 indexed citations

Thys, Kim, Marjolein Crabbe, Hans Wils, et al.. (2024). WED-379 Intrahepatic changes in immunologic and virologic markers during siRNA JNJ-73763989 (JNJ-3989) based treatment of chronic hepatitis B (CHB) patients: imaging mass cytometry (IMC) analyses from the INSIGHT study. Journal of Hepatology. 80. S813–S813. 1 indexed citations

Matetovici, Irina, Suresh Poovathingal, Jeroen Aerts, et al.. (2022). Comparative analysis of antibody- and lipid-based multiplexing methods for single-cell RNA-seq. Genome biology. 23(1). 55–55. 24 indexed citations

Thomson, Paul, Xiaoli Meng, Jan Snoeys, et al.. (2020). Drug‐specific T‐cell responses in patients with liver injury following treatment with the BACE inhibitor atabecestat. Allergy. 76(6). 1825–1835. 15 indexed citations

Wils, Hans, et al.. (2018). Methods to Investigate the Molecular Basis of Progranulin Actions on Brain and Behavior In Vivo Using Knockout Mice. Methods in molecular biology. 1806. 233–253. 1 indexed citations

Gielis, Els M., Kristien J. Ledeganck, Hans Wils, et al.. (2016). SO002QUANTIFICATION OF PLASMA DONOR-DERIVED CELL-FREE DNA TO MONITOR KIDNEY TRANSPLANT HEALTH: PRELIMINARY RESULTS OF A SINGLE TUBE MULTIPLEX PCR ASSAY. Nephrology Dialysis Transplantation. 31(suppl_1). i1–i1. 4 indexed citations

Janssens, Jonathan, Hans Wils, Gernot Kleinberger, et al.. (2013). Overexpression of ALS-Associated p.M337V Human TDP-43 in Mice Worsens Disease Features Compared to Wild-type Human TDP-43 Mice. Molecular Neurobiology. 48(1). 22–35. 79 indexed citations

Wils, Hans, Gernot Kleinberger, Sandra Pereson, et al.. (2012). Cellular ageing, increased mortality and FTLD‐TDP‐associated neuropathology in progranulin knockout mice. The Journal of Pathology. 228(1). 67–76. 94 indexed citations

Janssens, Jonathan, Gernot Kleinberger, Hans Wils, & Christine Van Broeckhoven. (2011). The role of mutant TAR DNA-binding protein 43 in amyotrophic lateral sclerosis and frontotemporal lobar degeneration. Biochemical Society Transactions. 39(4). 954–959. 13 indexed citations

10.

Wils, Hans, Gernot Kleinberger, Jonathan Janssens, et al.. (2010). TDP-43 transgenic mice develop spastic paralysis and neuronal inclusions characteristic of ALS and frontotemporal lobar degeneration. Proceedings of the National Academy of Sciences. 107(8). 3858–3863. 445 indexed citations

11.

Kleinberger, Gernot, Hans Wils, Peter Ponsaerts, et al.. (2010). Increased caspase activation and decreased TDP‐43 solubility in progranulin knockout cortical cultures. Journal of Neurochemistry. 115(3). 735–747. 52 indexed citations

12.

Pereson, Sandra, Hans Wils, Gernot Kleinberger, et al.. (2009). Progranulin expression correlates with dense‐core amyloid plaque burden in Alzheimer disease mouse models. The Journal of Pathology. 219(2). 173–181. 70 indexed citations

13.

Kumar‐Singh, Samir, Hans Wils, Gernot Kleinberger, et al.. (2009). S4‐04‐02: Overexpression of Wild‐type TDP‐43 Leads to Motor Neuron Degeneration and Spastic Quadriplegia in Germline Transgenic Mice. Alzheimer s & Dementia. 5(4S_Part_5). 1 indexed citations

14.

Broeck, Bianca Van, Daniel Pirici, Debby Van Dam, et al.. (2006). Intraneuronal amyloid β and reduced brain volume in a novel APP T714I mouse model for Alzheimer's disease. Neurobiology of Aging. 29(2). 241–252. 50 indexed citations

15.

Broeck, Bianca Van, Daniel Pirici, Debby Van Dam, et al.. (2006). P1–045: Intraneuronal amyloid β and reduced brain volume in a novel APP ‘Austrian’ mouse model for Alzheimer's disease. Alzheimer s & Dementia. 2(3S_Part_4). 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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