Hannah M. Knochelmann
- Immunology top 5%
- Immunotherapy and Immune Responses 14
- Immune Cell Function and Interaction 9
- T-cell and B-cell Immunology 4
- Oncology top 5%
- CAR-T cell therapy research 17
- Cancer Immunotherapy and Biomarkers 17
- Peptidase Inhibition and Analysis 3
- Lung Cancer Research Studies 2
- Otorhinolaryngology top 5%
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- Cancer Research and Treatments 2
- Co-authors
- Chrystal M. PaulosConnor J. DwyerStefanie R. BaileyDirk M. ElstonMegan M. WyattAubrey S. SmithDavid M. NeskeyShikhar Mehrotra
- Partner nations
- United StatesIndiaGeorgia
In The Last Decade
Hannah M. Knochelmann
26 papers receiving 1.1k citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Immunology 542
- Oncology 649
- Otorhinolaryngology 82
- Cancer Research 113
- Biological Psychiatry 11
Countries citing papers authored by Hannah M. Knochelmann
This map shows the geographic impact of Hannah M. Knochelmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hannah M. Knochelmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hannah M. Knochelmann more than expected).
Fields of papers citing papers by Hannah M. Knochelmann
This network shows the impact of papers produced by Hannah M. Knochelmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hannah M. Knochelmann. The network helps show where Hannah M. Knochelmann may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Hannah M. Knochelmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2024 | 3 | |
| 3 | 2023 | 16 | |
| 4 | 2023 | 10 | |
| 5 | 2022 | 12 | |
| 6 | 2022 | 3 | |
| 7 | 2022 | 2 | |
| 8 | 2021 | 5 | |
| 9 | 2021 | 31 | |
| 10 | 2021 | 84 | |
| 11 | 2020 | 26 | |
| 12 | 2020 | 31 | |
| 13 | Identification of human CD4+ T cell populations with distinct antitumor activity | 2020 | 1 |
| 14 | 2020 | 48 | |
| 15 | 2020 | 0 | |
| 16 | 2020 | 29 | |
| 17 | 2019 | 58 | |
| 18 | 2019 | 72 | |
| 19 | 2018 | 150 | |
| 20 | When worlds collide: Th17 and Treg cells in cancer and autoimmunitybreakdown → | 2018 | 407 |
About Hannah M. Knochelmann
Hannah M. Knochelmann is a scholar working on Oncology, Immunology and Biotechnology, having authored 28 papers that have together received 1.1k indexed citations. Recurring topics across this work include CAR-T cell therapy research (17 papers), Cancer Immunotherapy and Biomarkers (17 papers), Immunotherapy and Immune Responses (14 papers), Immune Cell Function and Interaction (9 papers), T-cell and B-cell Immunology (4 papers), Peptidase Inhibition and Analysis (3 papers), Cancer Research and Treatments (2 papers) and Lung Cancer Research Studies (2 papers). The work is most often cited by research in Immunology (542 citations), Oncology (649 citations) and Otorhinolaryngology (82 citations). Hannah M. Knochelmann has collaborated with scholars based in United States, India and Georgia. Frequent co-authors include Chrystal M. Paulos, Connor J. Dwyer, Stefanie R. Bailey, Dirk M. Elston, Megan M. Wyatt, Aubrey S. Smith, David M. Neskey, Shikhar Mehrotra, Terry A. Day and Guillermo O. Rangel Rivera.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.