Hannah J. Uckelmann
- Molecular Biology
- Hematology top 2%
- Cancer Research top 5%
- Immunology
- Genetics top 10%
- Co-authors
- Evgenij FiškinThomas LongerichPeter SchirmacherRobert GeffersScott A. ArmstrongTony GutschnerKai BreuhahnBritta Skawran
- Topics
- Acute Myeloid Leukemia Research (7 papers)Hematopoietic Stem Cell Transplantation (4 papers)Protein Degradation and Inhibitors (4 papers)
- Cited by
- HematologyCancer ResearchImmunology
- Partner nations
- GermanyUnited StatesSwitzerland
In The Last Decade
Hannah J. Uckelmann
15 papers receiving 982 citations
Hit Papers
Peers
Comparison fields: 5 of 64
- Molecular Biology 656
- Hematology 455
- Cancer Research 322
- Immunology 208
- Genetics 88
Countries citing papers authored by Hannah J. Uckelmann
This map shows the geographic impact of Hannah J. Uckelmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hannah J. Uckelmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hannah J. Uckelmann more than expected).
Fields of papers citing papers by Hannah J. Uckelmann
This network shows the impact of papers produced by Hannah J. Uckelmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hannah J. Uckelmann. The network helps show where Hannah J. Uckelmann may publish in the future.
Co-authorship network of co-authors of Hannah J. Uckelmann
This figure shows the co-authorship network connecting the top 25 collaborators of Hannah J. Uckelmann. A scholar is included among the top collaborators of Hannah J. Uckelmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hannah J. Uckelmann. Hannah J. Uckelmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 15 | |
| 3 | 82 | |
| 4 | 152 | |
| 5 | 16 | |
| 6 | 11 | |
| 7 | 1 | |
| 8 | 31 | |
| 9 | Inflammation-Induced Emergency Megakaryopoiesis Driven by Hematopoietic Stem Cell-like Megakaryocyte Progenitorsbreakdown → | 292 |
| 10 | 1 | |
| 11 | 152 | |
| 12 | 24 | |
| 13 | 203 | |
| 14 | 2 | |
| 15 | BBF RFC 41: Units for Promoter Measurement in Mammalian Cells | 1 |
About Hannah J. Uckelmann
Hannah J. Uckelmann is a scholar working on Hematology, Genetics and Physiology, having authored 15 papers that have together received 986 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (7 papers), Hematopoietic Stem Cell Transplantation (4 papers) and Protein Degradation and Inhibitors (4 papers). The work is most often cited by research in Hematology (455 citations), Cancer Research (322 citations) and Immunology (208 citations). Hannah J. Uckelmann has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Evgenij Fiškin, Thomas Longerich, Peter Schirmacher, Robert Geffers, Scott A. Armstrong, Tony Gutschner, Kai Breuhahn, Britta Skawran, Monika Hämmerle and Sven Diederichs. Their work appears in journals such as Science, The Journal of Experimental Medicine and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.