Hana Vlášková

739 total citations
27 papers, 483 citations indexed

About

Hana Vlášková is a scholar working on Molecular Biology, Physiology and Epidemiology. According to data from OpenAlex, Hana Vlášková has authored 27 papers receiving a total of 483 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 11 papers in Physiology and 7 papers in Epidemiology. Recurrent topics in Hana Vlášková's work include Lysosomal Storage Disorders Research (10 papers), Cardiomyopathy and Myosin Studies (6 papers) and Metabolism and Genetic Disorders (5 papers). Hana Vlášková is often cited by papers focused on Lysosomal Storage Disorders Research (10 papers), Cardiomyopathy and Myosin Studies (6 papers) and Metabolism and Genetic Disorders (5 papers). Hana Vlášková collaborates with scholars based in Czechia, United Kingdom and Slovakia. Hana Vlášková's co-authors include Lenka Dvořáková, Helena Poupětová, M. Elleder, Martin Hřebı́ček, Helena Hůlková, Helena Jahnová, Sarenur Gökben, Meral Topçu, Julie Turnbull and Sara Mole and has published in prestigious journals such as Brain, Gene and Nutrients.

In The Last Decade

Hana Vlášková

26 papers receiving 473 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hana Vlášková Czechia 13 272 179 119 96 95 27 483
Roberta Taurisano Italy 14 257 0.9× 174 1.0× 107 0.9× 72 0.8× 45 0.5× 28 501
Mónica Cozar Spain 12 179 0.7× 113 0.6× 51 0.4× 39 0.4× 80 0.8× 24 369
Carmela Zizzo Italy 14 384 1.4× 137 0.8× 118 1.0× 140 1.5× 117 1.2× 34 490
Christine Broissand France 7 175 0.6× 138 0.8× 66 0.6× 82 0.9× 51 0.5× 10 416
Susana Teijeira Spain 16 192 0.7× 305 1.7× 322 2.7× 71 0.7× 85 0.9× 38 689
Stephanie DeArmey United States 14 467 1.7× 99 0.6× 319 2.7× 128 1.3× 31 0.3× 19 573
Alexander Broomfield United Kingdom 14 358 1.3× 205 1.1× 137 1.2× 124 1.3× 54 0.6× 54 613
Dawn Peck United States 13 320 1.2× 329 1.8× 85 0.7× 79 0.8× 33 0.3× 32 607
Johanna M. P. van den Hout Netherlands 12 634 2.3× 144 0.8× 420 3.5× 150 1.6× 61 0.6× 31 729
Miriam Rigoldi Italy 16 286 1.1× 119 0.7× 228 1.9× 114 1.2× 42 0.4× 29 544

Countries citing papers authored by Hana Vlášková

Since Specialization
Citations

This map shows the geographic impact of Hana Vlášková's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hana Vlášková with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hana Vlášková more than expected).

Fields of papers citing papers by Hana Vlášková

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hana Vlášková. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hana Vlášková. The network helps show where Hana Vlášková may publish in the future.

Co-authorship network of co-authors of Hana Vlášková

This figure shows the co-authorship network connecting the top 25 collaborators of Hana Vlášková. A scholar is included among the top collaborators of Hana Vlášková based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hana Vlášková. Hana Vlášková is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chrastina, Petr, Lenka Dvořáková, Hana Vlášková, et al.. (2021). X-linked adrenoleukodystrophy: phenotype-genotype correlation in hemizygous males and heterozygous females with ABCD1 mutations.. PubMed. 42(5). 359–367. 4 indexed citations
2.
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Kuchař, Ladislav, Ondřej Lukšan, Befekadu Asfaw, et al.. (2020). Transcript, protein, metabolite and cellular studies in skin fibroblasts demonstrate variable pathogenic impacts of NPC1 mutations. Orphanet Journal of Rare Diseases. 15(1). 85–85. 4 indexed citations
4.
Jahnová, Helena, Helena Poupětová, Hana Vlášková, et al.. (2019). Amyotrophy, cerebellar impairment and psychiatric disease are the main symptoms in a cohort of 14 Czech patients with the late-onset form of Tay–Sachs disease. Journal of Neurology. 266(8). 1953–1959. 16 indexed citations
5.
Kousal, Bohdan, Petr Dušek, Lenka Piherová, et al.. (2019). Alu‐mediated Xq24 deletion encompassing CUL4B, LAMP2, ATP1B4, TMEM255A, and ZBTB33 genes causes Danon disease in a female patient. American Journal of Medical Genetics Part A. 182(1). 219–223. 8 indexed citations
6.
Pika, Tomáš, et al.. (2015). Familiar Amyloid Polyneuropathy –  a Case Report. Česká a slovenská neurologie a neurochirurgie. 78/111(6). 710–714.
7.
Burda, Patricie, Alexandra Schäfer, Terttu Suormala, et al.. (2015). Insights into Severe 5,10-Methylenetetrahydrofolate Reductase Deficiency: Molecular Genetic and Enzymatic Characterization of 76 Patients. Human Mutation. 36(6). 611–621. 52 indexed citations
8.
Jahnová, Helena, Lenka Dvořáková, Hana Vlášková, et al.. (2014). Observational, retrospective study of a large cohort of patients with Niemann-Pick disease type C in the Czech Republic: a surprisingly stable diagnostic rate spanning almost 40 years. Orphanet Journal of Rare Diseases. 9(1). 140–140. 45 indexed citations
9.
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Hůlková, Helena, J. Hanuš, Miroslav Souček, et al.. (2013). Systemic AL amyloidosis with unusual cutaneous presentation unmasked by carotenoderma. Amyloid. 21(1). 57–61. 1 indexed citations
11.
Peisker, Tomáš, et al.. (2013). Clinical spectrum in CADASIL family with a new mutation. Biomedical Papers. 157(4). 379–382. 6 indexed citations
12.
Paleček, Tomáš, Helena Poupětová, Hana Vlášková, et al.. (2013). Prevalence of Fabry disease in male patients with unexplained left ventricular hypertrophy in primary cardiology practice: prospective Fabry cardiomyopathy screening study (FACSS). Journal of Inherited Metabolic Disease. 37(3). 455–460. 43 indexed citations
13.
Pelák, Ondřej, Tomáš Kalina, Hana Vlášková, et al.. (2013). Mosaic tissue distribution of the tandem duplication of LAMP2 exons 4 and 5 demonstrates the limits of Danon disease cellular and molecular diagnostics. Journal of Inherited Metabolic Disease. 37(1). 117–124. 13 indexed citations
14.
Vlášková, Hana, Tomáš Kalina, Miloš Kubánek, et al.. (2012). Danon disease: A focus on processing of the novel LAMP2 mutation and comments on the beneficial use of peripheral white blood cells in the diagnosis of LAMP2 deficiency. Gene. 498(2). 183–195. 22 indexed citations
15.
Lukšan, Ondřej, et al.. (2010). Disruption of OTC promoter-enhancer interaction in a patient with symptoms of ornithine carbamoyltransferase deficiency. Human Mutation. 31(4). E1294–E1303. 12 indexed citations
16.
Żarowski, Marcin, et al.. (2010). Treatment of cataplexy in Niemann–Pick disease type C with the use of miglustat. European Journal of Paediatric Neurology. 15(1). 84–87. 15 indexed citations
17.
Kubánek, Miloš, M. Elleder, Jakub Sikora, et al.. (2010). Danon disease - a disorder of autophagy as a cause of hypertrophic cardiomyopathy. Cor et Vasa. 52(11-12). 706–712. 1 indexed citations
18.
Beesley, Clare, Linda Berná, Hana Vlášková, et al.. (2009). Mucopolysaccharidosis type I in 21 Czech and Slovak patients: Mutation analysis suggests a functional importance of C‐terminus of the IDUA protein. American Journal of Medical Genetics Part A. 149A(5). 965–974. 29 indexed citations
19.
Kousi, Maria, Eija Siintola, Lenka Dvořáková, et al.. (2009). Mutations in CLN7/MFSD8 are a common cause of variant late-infantile neuronal ceroid lipofuscinosis. Brain. 132(3). 810–819. 94 indexed citations
20.
Elleder, M., Lenka Dvořáková, Hana Vlášková, et al.. (2008). Atypical CLN2 with later onset and prolonged course: a neuropathologic study showing different sensitivity of neuronal subpopulations to TPP1 deficiency. Acta Neuropathologica. 116(1). 119–124. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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