H. Wick
Impact in
- Clinical Biochemistry top 2%
- Metabolism and Genetic Disorders
- Cell Biology top 5%
- Biotin and Related Studies
Papers in ⓘ
-
- Metabolism and Genetic Disorders 13
- Cell Biology 16
- Biotin and Related Studies 14
- Cellular transport and secretion 4
- Co-authors
- E. R. Baumgartner (15 shared papers)Terttu Suormala (10 shared papers)C. Bachmann (7 shared papers)R. Baumgartner (6 shared papers)Joachim Seelig (1 shared paper)Katrin Schweizer (2 shared papers)Nicolau Beckmann (1 shared paper)D Clerc (1 shared paper)
- Journals
- Pediatric Research (5 papers)European Journal of Pediatrics (5 papers)Clinica Chimica Acta (4 papers)Journal of Inherited Metabolic Disease (3 papers)The Lancet (2 papers)
- Partner nations
- SwitzerlandUnited StatesGermany
In The Last Decade
H. Wick
40 papers receiving 639 citations
Peers
Comparison fields: 5 of 73
- Clinical Biochemistry 250
- Cell Biology 295
- Rheumatology 114
- Biochemistry 49
- Pathology and Forensic Medicine 102
Countries citing papers authored by H. Wick
This map shows the geographic impact of H. Wick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Wick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Wick more than expected).
Fields of papers citing papers by H. Wick
This network shows the impact of papers produced by H. Wick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Wick. The network helps show where H. Wick may publish in the future.
Co-authors
The 25 scholars most cited alongside H. Wick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 43 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 1985 | 78 | |
| 2 | 1989 | 63 | |
| 3 | 1990 | 41 | |
| 4 | 1997 | 32 | |
| 5 | Congenital defect in intracellular cobalamin metabolism resulting in homocysteinuria and methylmalonic aciduria. I. Case report and histopathology. | 1979 | 30 |
| 6 | 1986 | 29 | |
| 7 | 1985 | 28 | |
| 8 | 1973 | 27 | |
| 9 | 1977 | 27 | |
| 10 | 1998 | 26 | |
| 11 | 1989 | 24 | |
| 12 | 1985 | 23 | |
| 13 | Congenital defect in intracellular cobalamin metabolism resulting in homocystinuria and methylmalonic aciduria. II. Biochemical investigations. | 1979 | 22 |
| 14 | 1988 | 22 | |
| 15 | 1988 | 21 | |
| 16 | 1985 | 20 | |
| 17 | 1984 | 15 | |
| 18 | 1978 | 15 | |
| 19 | 1980 | 14 | |
| 20 | 1970 | 14 |
About H. Wick
H. Wick is a scholar working on Clinical Biochemistry, Cell Biology, Rheumatology, Biochemistry and Organic Chemistry, having authored 43 papers that have together received 676 indexed citations. Recurring topics across this work include Biotin and Related Studies (14 papers), Metabolism and Genetic Disorders (13 papers), Click Chemistry and Applications (8 papers), Folate and B Vitamins Research (6 papers), Diet and metabolism studies (5 papers), Vitamin D Research Studies (4 papers), Mitochondrial Function and Pathology (4 papers) and Cellular transport and secretion (4 papers). The work is most often cited by research in Clinical Biochemistry (250 citations), Cell Biology (295 citations), Rheumatology (114 citations), Biochemistry (49 citations) and Pathology and Forensic Medicine (102 citations). H. Wick has collaborated with scholars based in Switzerland, United States and Germany. Frequent co-authors include E. R. Baumgartner, Terttu Suormala, C. Bachmann, R. Baumgartner, Joachim Seelig, Katrin Schweizer, Nicolau Beckmann, D Clerc, Jochen Bausch and B Steinmann. Their work appears in journals such as Pediatric Research, European Journal of Pediatrics, Clinica Chimica Acta, Journal of Inherited Metabolic Disease and The Lancet.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.