H. Veldman

1.4k total citations
40 papers, 1.1k citations indexed

About

H. Veldman is a scholar working on Neurology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, H. Veldman has authored 40 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Neurology, 18 papers in Cellular and Molecular Neuroscience and 7 papers in Cell Biology. Recurrent topics in H. Veldman's work include Nerve injury and regeneration (10 papers), Peripheral Neuropathies and Disorders (8 papers) and Hereditary Neurological Disorders (7 papers). H. Veldman is often cited by papers focused on Nerve injury and regeneration (10 papers), Peripheral Neuropathies and Disorders (8 papers) and Hereditary Neurological Disorders (7 papers). H. Veldman collaborates with scholars based in Netherlands, Germany and United Kingdom. H. Veldman's co-authors include F.G.I. Jennekens, C.J.M. van den Oord, John H. J. Wokke, F.G.I. Jennekens, L. M. E. Smit, Angela Vincent, Dirk de Korte, Willem H. Ouwehand, Dirk Roos and Piet W. Modderman and has published in prestigious journals such as Journal of Neuroscience, Brain and Neurology.

In The Last Decade

H. Veldman

38 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
H. Veldman Netherlands 20 381 344 335 126 121 40 1.1k
J. R. Muddle United Kingdom 25 333 0.9× 521 1.5× 408 1.2× 69 0.5× 42 0.3× 41 1.6k
Friederike Knerlich‐Lukoschus Germany 18 152 0.4× 259 0.8× 299 0.9× 236 1.9× 422 3.5× 37 1.4k
Li‐Ru Zhao United States 18 430 1.1× 193 0.6× 424 1.3× 108 0.9× 46 0.4× 41 1.2k
T. Soukup Czechia 22 106 0.3× 262 0.8× 825 2.5× 94 0.7× 20 0.2× 106 1.6k
Julian Cahill United Kingdom 13 909 2.4× 179 0.5× 365 1.1× 69 0.5× 38 0.3× 32 1.5k
Tomoya Terashima Japan 21 178 0.5× 271 0.8× 432 1.3× 147 1.2× 25 0.2× 53 1.4k
Qing Yin China 18 166 0.4× 141 0.4× 421 1.3× 152 1.2× 30 0.2× 51 1.2k
Keiji Igase Japan 14 239 0.6× 144 0.4× 126 0.4× 62 0.5× 219 1.8× 30 779
Valentine Brussee Canada 13 168 0.4× 327 1.0× 434 1.3× 188 1.5× 217 1.8× 14 1.3k
Shinjiro Kaneko Japan 20 117 0.3× 447 1.3× 334 1.0× 74 0.6× 19 0.2× 60 1.7k

Countries citing papers authored by H. Veldman

Since Specialization
Citations

This map shows the geographic impact of H. Veldman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Veldman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Veldman more than expected).

Fields of papers citing papers by H. Veldman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. Veldman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Veldman. The network helps show where H. Veldman may publish in the future.

Co-authorship network of co-authors of H. Veldman

This figure shows the co-authorship network connecting the top 25 collaborators of H. Veldman. A scholar is included among the top collaborators of H. Veldman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Veldman. H. Veldman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Malsen, J.G. de Mooij‐van, Ka Lou Yu, H. Veldman, et al.. (2009). Variations in ventral root axon morphology and locomotor behavior components across different inbred strains of mice. Neuroscience. 164(4). 1477–1483. 6 indexed citations
2.
Dijkstra, Sipke, Simone Duis, Alex J. Lankhorst, et al.. (2006). Intraspinal administration of an antibody against CD81 enhances functional recovery and tissue sparing after experimental spinal cord injury. Experimental Neurology. 202(1). 57–66. 19 indexed citations
3.
4.
Groeneveld, Geert Jan, F. L. Van Muiswinkel, H. Veldman, et al.. (2003). Zinc amplifies mSOD1-mediated toxicity in a transgenic mouse model of amyotrophic lateral sclerosis. Neuroscience Letters. 352(3). 175–178. 23 indexed citations
5.
Joosten, Elbert A.J., et al.. (2001). Cellular changes in motoneurons in a transgenic mouse model for amyotrophic lateral sclerosis as revealed by monoclonal antibody Py. Developmental Brain Research. 131(1-2). 153–159. 3 indexed citations
6.
Berg, Leonard H. van den, et al.. (2000). Unmyelinated nerve fiber degeneration in chronic inflammatory demyelinating polyneuropathy. Acta Neuropathologica. 99(5). 571–578. 8 indexed citations
7.
Teunissen, Laurien L., et al.. (2000). Thickness of endoneurial vessel basal lamina area in chronic idiopathic axonal polyneuropathy. Acta Neuropathologica. 100(4). 445–450. 19 indexed citations
8.
Houweling, Diane A., Gary A. Brook, Roben G. Gieling, et al.. (1999). Differential distribution of immunoreactivity in the developing rat spinal cord revealed by the monoclonal antibody Py. Developmental Brain Research. 116(1). 87–96. 2 indexed citations
9.
Worp, H. Bart van der, Peter Sodaar, H. Veldman, et al.. (1997). An Advanced In Vitro Model to Study Hypoxia/Low Glucose‐Induced Neuronal Cell Damage and Death. Annals of the New York Academy of Sciences. 825(1). 267–278. 2 indexed citations
10.
Dijk, Gert W. van, et al.. (1995). A new variant of sensory ataxic neuropathy with autosomal dominant inheritance. Brain. 118(6). 1557–1563. 8 indexed citations
11.
Hassan, Sherif Maher, et al.. (1994). GAP-43 and p75NGFR immunoreactivity in presynaptic cells following neuromuscular blockade by botulinum toxin in rat. Journal of Neurocytology. 23(6). 354–363. 28 indexed citations
12.
Hoop, R. Gerritsen van der, Frank P.T. Hamers, J.P. Neijt, et al.. (1994). Protection against cisplatin induced neurotoxicity by ORG 2766: histological and electrophysiological evidence. Journal of the Neurological Sciences. 126(2). 109–115. 28 indexed citations
13.
Jennekens, F.G.I., et al.. (1993). Development of innervation of skeletal muscle fibers in man: Relation to acetylcholine receptors. The Anatomical Record. 236(3). 553–562. 100 indexed citations
14.
Graus, Y., Mieke E.R. Henfling, Jan J.G.M. Verschuuren, et al.. (1991). Age related resistance to experimental autoimmune myasthenia gravis in rats. Journal of Autoimmunity. 4(6). xxviii–xxviii. 23 indexed citations
15.
Fijnheer, Rob, Piet W. Modderman, H. Veldman, et al.. (1990). Detection of platelet activation with monoclonal antibodies and flow cytometry. Changes during platelet storage. Transfusion. 30(1). 20–25. 148 indexed citations
16.
Wokke, John H. J., et al.. (1990). Morphological changes in the human end plate with age. Journal of the Neurological Sciences. 95(3). 291–310. 99 indexed citations
17.
Jennekens, F.G.I., et al.. (1989). A light and electron microscopical study of B-50 (GAP-43) in human intramuscular nerve and neuromuscular junctions during development. Journal of the Neurological Sciences. 89(2-3). 301–311. 12 indexed citations
18.
Smit, L. M. E., et al.. (1988). A myasthenic syndrome with congenital paucity of secondary synaptic clefts: CPSC Syndrome. Muscle & Nerve. 11(4). 337–348. 22 indexed citations
19.
Oosterhuis, Harry, John Newsom–Davis, John H. J. Wokke, et al.. (1987). THE SLOW CHANNEL SYNDROME. Brain. 110(4). 1061–1079. 60 indexed citations
20.
Smit, L. M. E., F.G.I. Jennekens, H. Veldman, & P. G. Barth. (1984). Paucity of secondary synaptic clefts in a case of congenital myasthenia with multiple contractures: ultrastructural morphology of a developmental disorder.. Journal of Neurology Neurosurgery & Psychiatry. 47(10). 1091–1097. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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