H. U. Tietze
- Molecular Biology
- Immunology
- Genetics
- Endocrinology, Diabetes and Metabolism
- Pathology and Forensic Medicine
- Co-authors
- Anita RauchH. SingerRainer KoenigR. RauchHelmuth-Guenther DoerrMartin ZenkerG. BuheitelAndré Reis
- Topics
- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (3 papers)Electrolyte and hormonal disorders (3 papers)Chromosomal and Genetic Variations (3 papers)
- Journals
- The Journal of Clinical Endocrinology & MetabolismScientific ReportsThe Journal of Pediatrics
- Partner nations
- GermanyUnited StatesCzechia
In The Last Decade
H. U. Tietze
21 papers receiving 458 citations
Peers
Comparison fields: 5 of 61
- Molecular Biology 318
- Immunology 156
- Genetics 144
- Endocrinology, Diabetes and Metabolism 88
- Pathology and Forensic Medicine 56
Countries citing papers authored by H. U. Tietze
This map shows the geographic impact of H. U. Tietze's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. U. Tietze with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. U. Tietze more than expected).
Fields of papers citing papers by H. U. Tietze
This network shows the impact of papers produced by H. U. Tietze. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. U. Tietze. The network helps show where H. U. Tietze may publish in the future.
Co-authorship network of co-authors of H. U. Tietze
This figure shows the co-authorship network connecting the top 25 collaborators of H. U. Tietze. A scholar is included among the top collaborators of H. U. Tietze based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. U. Tietze. H. U. Tietze is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 3 | |
| 3 | 20 | |
| 4 | 177 | |
| 5 | 2 | |
| 6 | 82 | |
| 7 | 5 | |
| 8 | 14 | |
| 9 | 18 | |
| 10 | 46 | |
| 11 | 57 | |
| 12 | 1 | |
| 13 | 9 | |
| 14 | 5 | |
| 15 | 4 | |
| 16 | 9 | |
| 17 | 2 | |
| 18 | 1 | |
| 19 | 11 | |
| 20 | 1 |
About H. U. Tietze
H. U. Tietze is a scholar working on Developmental Biology, Urology and Endocrinology, Diabetes and Metabolism, having authored 21 papers that have together received 475 indexed citations. Recurring topics across this work include Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (3 papers), Electrolyte and hormonal disorders (3 papers) and Chromosomal and Genetic Variations (3 papers). The work is most often cited by research in Immunology (156 citations), Endocrinology, Diabetes and Metabolism (88 citations) and Genetics (144 citations). H. U. Tietze has collaborated with scholars based in Germany, United States and Czechia. Frequent co-authors include Anita Rauch, H. Singer, Rainer Koenig, R. Rauch, Helmuth-Guenther Doerr, Martin Zenker, G. Buheitel, André Reis, Wolfram Kreß and Michael Hofbeck. Their work appears in journals such as The Journal of Clinical Endocrinology & Metabolism, Scientific Reports and The Journal of Pediatrics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.