Gerd Scherer

12.2k total citations · 1 hit paper
150 papers, 9.5k citations indexed

About

Gerd Scherer is a scholar working on Molecular Biology, Genetics and Reproductive Medicine. According to data from OpenAlex, Gerd Scherer has authored 150 papers receiving a total of 9.5k indexed citations (citations by other indexed papers that have themselves been cited), including 92 papers in Molecular Biology, 75 papers in Genetics and 19 papers in Reproductive Medicine. Recurrent topics in Gerd Scherer's work include Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (63 papers), Sexual Differentiation and Disorders (38 papers) and Sperm and Testicular Function (19 papers). Gerd Scherer is often cited by papers focused on Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (63 papers), Sexual Differentiation and Disorders (38 papers) and Sperm and Testicular Function (19 papers). Gerd Scherer collaborates with scholars based in Germany, United States and France. Gerd Scherer's co-authors include Ralf Kist, Francisco J. Barrionuevo, W. Schempp, U. Wolf, Jutta Wirth, Hans‐Heinrich Limbach, Jobst Meyer, Elisabeth Hustert, Stefan Bagheri‐Fam and Thomas Wagner and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Gerd Scherer

147 papers receiving 9.2k citations

Hit Papers

Autosomal sex reversal and campomelic dysplasia are cause... 1994 2026 2004 2015 1994 400 800 1.2k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Autosomal sex reversal and campomelic dysplasia are caused by mutations in and around the SRY-related gene SOX9
    1994 1.2k citations Thomas Wagner, Jutta Wirth et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Gerd Scherer Germany 51 6.3k 4.9k 1.3k 928 770 150 9.5k
Elizabeth M. Wilson United States 62 7.3k 1.2× 5.0k 1.0× 1.2k 0.9× 366 0.4× 737 1.0× 150 12.8k
Gordon H. Dixon Canada 56 6.3k 1.0× 2.1k 0.4× 1.2k 0.9× 362 0.4× 239 0.3× 215 9.2k
Marvin L. Meistrich United States 65 5.7k 0.9× 3.2k 0.7× 6.9k 5.2× 1.1k 1.2× 1.1k 1.4× 257 13.1k
Seiichi Mori Japan 46 3.9k 0.6× 1.8k 0.4× 482 0.4× 430 0.5× 1.5k 1.9× 206 9.1k
Wendy Dean United Kingdom 68 17.8k 2.8× 8.0k 1.6× 773 0.6× 877 0.9× 947 1.2× 150 22.0k
Michael D. Griswold United States 37 4.5k 0.7× 2.4k 0.5× 2.1k 1.6× 313 0.3× 264 0.3× 74 6.8k
Nabeel A. Affara United Kingdom 49 4.2k 0.7× 3.2k 0.7× 944 0.7× 301 0.3× 776 1.0× 167 7.1k
Nicholas D. Hastie United Kingdom 54 9.7k 1.5× 3.0k 0.6× 202 0.2× 1.0k 1.1× 652 0.8× 128 12.1k
Wolfgang J. Schneider Austria 64 6.8k 1.1× 2.5k 0.5× 746 0.6× 3.5k 3.7× 2.2k 2.9× 230 14.6k
Michael Stadler Germany 63 11.1k 1.8× 2.1k 0.4× 336 0.3× 535 0.6× 2.0k 2.5× 236 16.5k

Countries citing papers authored by Gerd Scherer

Since Specialization
Citations

This map shows the geographic impact of Gerd Scherer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gerd Scherer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gerd Scherer more than expected).

Fields of papers citing papers by Gerd Scherer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gerd Scherer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gerd Scherer. The network helps show where Gerd Scherer may publish in the future.

Co-authorship network of co-authors of Gerd Scherer

This figure shows the co-authorship network connecting the top 25 collaborators of Gerd Scherer. A scholar is included among the top collaborators of Gerd Scherer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gerd Scherer. Gerd Scherer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Georg, Ina, Francisco J. Barrionuevo, Thorsten Wiech, & Gerd Scherer. (2012). Sox9 and Sox8 Are Required for Basal Lamina Integrity of Testis Cords and for Suppression of FOXL2 During Embryonic Testis Development in Mice1. Biology of Reproduction. 87(4). 99–99. 43 indexed citations
2.
Georg, Ina, Stefan Bagheri‐Fam, Kevin C. Knower, et al.. (2010). Mutations of the SRY-Responsive Enhancer of <i>SOX9</i> Are Uncommon in XY Gonadal Dysgenesis. Sexual Development. 4(6). 321–325. 37 indexed citations
3.
Hargus, Gunnar, Ralf Kist, Jan Kramer, et al.. (2008). Loss of Sox9 function results in defective chondrocyte differentiation of mouse embryonic stem cells in vitro. The International Journal of Developmental Biology. 52(4). 323–332. 29 indexed citations
4.
Lausch, Ekkehart, Pia Hermanns, Henner F. Farin, et al.. (2008). TBX15 Mutations Cause Craniofacial Dysmorphism, Hypoplasia of Scapula and Pelvis, and Short Stature in Cousin Syndrome. The American Journal of Human Genetics. 83(5). 649–655. 46 indexed citations
5.
Rastegar, Sepand, Isabell Hess, Thomas Dickmeis, et al.. (2008). The words of the regulatory code are arranged in a variable manner in highly conserved enhancers. Developmental Biology. 318(2). 366–377. 45 indexed citations
6.
Barrionuevo, Francisco J., Ina Georg, Harry Scherthan, et al.. (2008). Testis cord differentiation after the sex determination stage is independent of Sox9 but fails in the combined absence of Sox9 and Sox8. Developmental Biology. 327(2). 301–312. 133 indexed citations
7.
Barrionuevo, Francisco J., Angela Naumann, Stefan Bagheri‐Fam, et al.. (2008). Sox9 is required for invagination of the otic placode in mice. Developmental Biology. 317(1). 213–224. 56 indexed citations
8.
Lincoln, Joy, Ralf Kist, Gerd Scherer, & Katherine E. Yutzey. (2007). Sox9 is required for precursor cell expansion and extracellular matrix organization during mouse heart valve development. Developmental Biology. 305(1). 120–132. 146 indexed citations
9.
Bagheri‐Fam, Stefan, Helena Sim, Pascal Bernard, et al.. (2007). Loss of Fgfr2 leads to partial XY sex reversal. Developmental Biology. 314(1). 71–83. 95 indexed citations
10.
Leipoldt, M., Martin Erdel, Marta Smyk, et al.. (2006). Two novel translocation breakpoints upstream of SOX9 define borders of the proximal and distal breakpoint cluster region in campomelic dysplasia. Clinical Genetics. 71(1). 67–75. 82 indexed citations
11.
Barrionuevo, Francisco J., Stefan Bagheri‐Fam, Ralf Kist, et al.. (2005). Homozygous Inactivation of Sox9 Causes Complete XY Sex Reversal in Mice1. Biology of Reproduction. 74(1). 195–201. 278 indexed citations
12.
Pfeiffer, R. A., Anita Rauch, Udo Trautmann, et al.. (1999). Defective sexual development in an infant with 46, XY, der(9)t(8;9)(q23.1;p23)mat. European Journal of Pediatrics. 158(3). 213–216. 9 indexed citations
13.
Scherer, Gerd, Martin Erdel, Dieter Meschede, et al.. (1998). Three novel SRY mutations in XY gonadal dysgenesis and the enigma of XY gonadal dysgenesis cases without SRY mutations. Cytogenetic and Genome Research. 80(1-4). 188–192. 55 indexed citations
14.
Wagner, Thomas, Niels Tommerup, Jutta Wirth, et al.. (1997). A somatic cell hybrid panel for distal 17q: GDIA1 maps to 17q25.3. Cytogenetic and Genome Research. 76(3-4). 172–175. 8 indexed citations
15.
Scherer, Gerd, et al.. (1997). Two Independent Nuclear Localization Signals Are Present in the DNA-binding High-mobility Group Domains of SRY and SOX9. Journal of Biological Chemistry. 272(44). 27848–27852. 154 indexed citations
16.
17.
Wagner, Thomas, Jutta Wirth, Jobst Meyer, et al.. (1994). Autosomal sex reversal and campomelic dysplasia are caused by mutations in and around the SRY-related gene SOX9. Cell. 79(6). 1111–1120. 1236 indexed citations breakdown →
18.
Wegner, Rolf‐Dieter, et al.. (1992). Ring Y chromosome: cytogenetic and molecular characterization. Clinical Genetics. 42(2). 71–75. 7 indexed citations
19.
Kiechle‐Schwarz, M., Gerd Scherer, & Gyula Kovács. (1990). No evidence for loss of alleles at 11p in HBV negative hepatocellular carcinomas. Genes Chromosomes and Cancer. 1(4). 312–314. 7 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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