H. J. Cornell

888 total citations
32 papers, 644 citations indexed

About

H. J. Cornell is a scholar working on Gastroenterology, Nutrition and Dietetics and Genetics. According to data from OpenAlex, H. J. Cornell has authored 32 papers receiving a total of 644 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Gastroenterology, 12 papers in Nutrition and Dietetics and 6 papers in Genetics. Recurrent topics in H. J. Cornell's work include Celiac Disease Research and Management (15 papers), Microbial Metabolites in Food Biotechnology (10 papers) and Food composition and properties (7 papers). H. J. Cornell is often cited by papers focused on Celiac Disease Research and Management (15 papers), Microbial Metabolites in Food Biotechnology (10 papers) and Food composition and properties (7 papers). H. J. Cornell collaborates with scholars based in Australia, United States and United Kingdom. H. J. Cornell's co-authors include R. R. W. Townley, Adrian C. Herington, C J Rolles, Andrew P. Negri, Prithi S. Bhathal, Donald E. Rivett, Francisco Barro, A. Real, Carolina Sousa and Ángel Cebolla and has published in prestigious journals such as The Lancet, Gut and Biochemical Journal.

In The Last Decade

H. J. Cornell

28 papers receiving 589 citations

Peers

H. J. Cornell

GASTR

EPIDE

SURGE

GENET

Ilaria Russo Italy

Myung‐Chul Kim South Korea

Robert D. Streilein United States

Ulderico Ventura Italy

Cong Liang China

Beatriz Martínez‐Abad Sweden

Chunli Zhou China

Petra Voland Germany

Laura Zanobbio Italy

Eric B. Nonnecke United States

Ilaria Russo Italy

H. J. Cornell

234 ×0.6

GASTR

47 ×0.3

181 ×1.2

EPIDE

146 ×1.5

SURGE

79 ×0.8

GENET

Citations per year, relative to H. J. Cornell H. J. Cornell (= 1×) peers Ilaria Russo

Countries citing papers authored by H. J. Cornell

Since Specialization

Citations

This map shows the geographic impact of H. J. Cornell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. J. Cornell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. J. Cornell more than expected).

Fields of papers citing papers by H. J. Cornell

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. J. Cornell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. J. Cornell. The network helps show where H. J. Cornell may publish in the future.

Co-authorship network of co-authors of H. J. Cornell

This figure shows the co-authorship network connecting the top 25 collaborators of H. J. Cornell. A scholar is included among the top collaborators of H. J. Cornell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. J. Cornell. H. J. Cornell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Comino, Isabel, A. Real, H. J. Cornell, et al.. (2011). Diversity in oat potential immunogenicity: basis for the selection of oat varieties with no toxicity in coeliac disease. Gut. 60(7). 915–922. 116 indexed citations

Cornell, H. J., William O.S. Doherty, & T Stelmasiak. (2009). Papaya latex enzymes capable of detoxification of gliadin. Amino Acids. 38(1). 155–165. 12 indexed citations

Cornell, H. J. & T Stelmasiak. (2006). A unified hypothesis of coeliac disease with implications for management of patients. Amino Acids. 33(1). 43–49. 11 indexed citations

Cornell, H. J., et al.. (2002). Extraction of Cereal Prolamins and their Toxicity in Coeliac Disease. PubMed. 6(2). 151–158. 5 indexed citations

Cornell, H. J., et al.. (2001). Structure-activity relationships in coeliac-toxic gliadin peptides. Amino Acids. 21(3). 243–253. 29 indexed citations

Cornell, H. J.. (1996). Coeliac disease: A review of the causative agents and their possible mechanisms of action. Amino Acids. 10(1). 1–19. 11 indexed citations

Cornell, H. J., John H. Skerritt, Robert Puy, & Maryam M. Javadpour. (1994). Studies of in vitro γ-interferon production in coeliac disease as a response to gliadin peptides. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1226(2). 126–130. 9 indexed citations

Cornell, H. J., Renata Auricchio, G De Ritis, et al.. (1988). Intestinal Mucosa of Celiacs in Remission Is Unable To Abolish Toxicity of Gliadin Peptides on in Vitro Developing Fetal Rat Intestine And Cultured Atrophic Celiac Mucosa. Pediatric Research. 24(2). 233–237. 17 indexed citations

Cornell, H. J.. (1988). Amino acid composition of peptides remaining after in vitro digestion of a gliadin sub-fraction with duodenal mucosa from patients with coeliac disease. Clinica Chimica Acta. 176(3). 279–289. 26 indexed citations

10.

Cornell, H. J., et al.. (1988). Solvent pretreatments of wool and their effect on shrinkproofing and the rate of dye uptake. Journal of the Society of Dyers and Colourists. 104(1). 22–27.

11.

Cornell, H. J., Gösta Enberg, & Adrian C. Herington. (1987). Preferential association of the insulin-like growth factors I and II with metabolically inactive and active carrier-bound complexes in serum. Biochemical Journal. 241(3). 745–750. 23 indexed citations

12.

Herington, Adrian C., et al.. (1983). Recent advances in the biochemistry and physiology of the insulin-like growth factor/somatomedin family. International Journal of Biochemistry. 15(10). 1201–1210. 47 indexed citations

13.

Cornell, H. J.. (1982). The Effect of Different Isolation Procedures on the Yields of Insulin-Like Growth Factors from Human Plasma. Preparative Biochemistry. 12(1). 57–76. 7 indexed citations

14.

Cockerill, Peter N., H. J. Cornell, & Adrian C. Herington. (1980). PARTIAL PURIFICATION OF LOW MOLECULAR WEIGHT NON-SUPPRESSIBLE INSULIN-LIKE ACTIVITY FROM HUMAN PLASMA: DEMONSTRATION OF THE PRESENCE OF MULTIPLE FORMS. Journal of Endocrinology. 85(2). 267–277. 17 indexed citations

15.

Cornell, H. J. & C J Rolles. (1978). Further evidence of a primary mucosal defect in coeliac disease. Gut. 19(4). 253–259. 26 indexed citations

16.

Cornell, H. J., et al.. (1976). NON-SPECIFIC CYTOTOXICITY OF WHEAT GLIADIN COMPONENTS TOWARDS CULTURED HUMAN CELLS. The Lancet. 307(7955). 339–341. 27 indexed citations

17.

Cornell, H. J. & R. R. W. Townley. (1973). Investigation of possible intestinal peptidase deficiency in coeliac disease. Clinica Chimica Acta. 43(1). 113–125. 61 indexed citations

18.

Bhathal, Prithi S., et al.. (1972). Gluten enteropathy in vitro: a culture system for the study of coeliac disease.. PubMed. 13(10). 848–848. 5 indexed citations

19.

Cornell, H. J.. (1970). Some of the Minor Constituents of Wheat Starch and its Hydrolysates ‐ Part 1.‐. Starch - Stärke. 22(2). 55–64. 5 indexed citations

20.

Cornell, H. J., et al.. (1965). The Application of Conductivity Measurement to the Study of Starch Hydrolysis Kinetics. Starch - Stärke. 17(1). 7–13. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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