H. Bittiger
- Cellular and Molecular Neuroscience top 0.5%
- Molecular Biology top 5%
- Physiology top 5%
- Cognitive Neuroscience top 5%
- Biomaterials top 5%
- Co-authors
- Wolfgang FroestlSerge BischoffJakob HeidStuart J. MickelBernhard BettlerKlemens KaupmannW. D. NiegischR. H. Marchessault
- Topics
- Neuroscience and Neuropharmacology Research (32 papers)Receptor Mechanisms and Signaling (18 papers)Ion channel regulation and function (10 papers)
- Partner nations
- SwitzerlandGermanyFrance
In The Last Decade
H. Bittiger
65 papers receiving 3.6k citations
Hit Papers
Peers
Comparison fields: 5 of 132
- Cellular and Molecular Neuroscience 2.4k
- Molecular Biology 1.9k
- Physiology 406
- Cognitive Neuroscience 340
- Biomaterials 262
Countries citing papers authored by H. Bittiger
This map shows the geographic impact of H. Bittiger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Bittiger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Bittiger more than expected).
Fields of papers citing papers by H. Bittiger
This network shows the impact of papers produced by H. Bittiger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Bittiger. The network helps show where H. Bittiger may publish in the future.
Co-authorship network of co-authors of H. Bittiger
This figure shows the co-authorship network connecting the top 25 collaborators of H. Bittiger. A scholar is included among the top collaborators of H. Bittiger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Bittiger. H. Bittiger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 14 | |
| 2 | 1 | |
| 3 | 168 | |
| 4 | 70 | |
| 5 | 2 | |
| 6 | Expression cloning of GABAB receptors uncovers similarity to metabotropic glutamate receptorsbreakdown → | 831 |
| 7 | 121 | |
| 8 | 3 | |
| 9 | 4 | |
| 10 | 8 | |
| 11 | 3 | |
| 12 | 82 | |
| 13 | 79 | |
| 14 | 72 | |
| 15 | 174 | |
| 16 | 272 | |
| 17 | 34 | |
| 18 | 22 | |
| 19 | 9 | |
| 20 | 26 |
About H. Bittiger
H. Bittiger is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Spectroscopy, having authored 65 papers that have together received 3.7k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (32 papers), Receptor Mechanisms and Signaling (18 papers) and Ion channel regulation and function (10 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (2.4k citations), Toxicology (130 citations) and Biological Psychiatry (90 citations). H. Bittiger has collaborated with scholars based in Switzerland, Germany and France. Frequent co-authors include Wolfgang Froestl, Serge Bischoff, Jakob Heid, Stuart J. Mickel, Bernhard Bettler, Klemens Kaupmann, W. D. Niegisch, R. H. Marchessault, David Dooley and Christof Angst. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.