Gwendolyn E. Kaeser
- Neurology top 5%
- Biological Psychiatry top 10%
- Developmental Neuroscience top 10%
- Molecular Biology top 10%
- Single-cell and spatial transcriptomics 2
- DNA Repair Mechanisms 2
- Gene Regulatory Network Analysis 2
- RNA and protein synthesis mechanisms 2
- Gene expression and cancer classification 2
- Cancer Research top 10%
-
- Coagulation, Bradykinin, Polyphosphates, and Angioedema 2
- Genomic variations and chromosomal abnormalities 1
-
- Mast cells and histamine 1
- Journals
- Nature (1 paper)New England Journal of Medicine (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- United StatesNetherlandsCanada
In The Last Decade
Gwendolyn E. Kaeser
11 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 82
- Neurology 178
- Biological Psychiatry 44
- Developmental Neuroscience 53
- Molecular Biology 863
- Cancer Research 175
Countries citing papers authored by Gwendolyn E. Kaeser
This map shows the geographic impact of Gwendolyn E. Kaeser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gwendolyn E. Kaeser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gwendolyn E. Kaeser more than expected).
Fields of papers citing papers by Gwendolyn E. Kaeser
This network shows the impact of papers produced by Gwendolyn E. Kaeser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gwendolyn E. Kaeser. The network helps show where Gwendolyn E. Kaeser may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Gwendolyn E. Kaeser, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 2 | |
| 2 | 2024 | 5 | |
| 3 | 2020 | 16 | |
| 4 | 2020 | 21 | |
| 5 | 2020 | 28 | |
| 6 | 2019 | 28 | |
| 7 | 2018 | 151 | |
| 8 | Integrative single-cell analysis of transcriptional and epigenetic states in the human adult brainbreakdown → | 2017 | 569 |
| 9 | 2016 | 236 | |
| 10 | 2015 | 102 | |
| 11 | 2011 | 10 |
About Gwendolyn E. Kaeser
Gwendolyn E. Kaeser is a scholar working on Developmental Neuroscience, Virology and Rheumatology, having authored 11 papers that have together received 1.2k indexed citations. Recurring topics across this work include Coagulation, Bradykinin, Polyphosphates, and Angioedema (2 papers), Single-cell and spatial transcriptomics (2 papers), DNA Repair Mechanisms (2 papers), Gene Regulatory Network Analysis (2 papers), RNA and protein synthesis mechanisms (2 papers), Gene expression and cancer classification (2 papers), Genomic variations and chromosomal abnormalities (1 paper) and Mast cells and histamine (1 paper). The work is most often cited by research in Neurology (178 citations), Biological Psychiatry (44 citations) and Developmental Neuroscience (53 citations). Gwendolyn E. Kaeser has collaborated with scholars based in United States, Netherlands and Canada. Frequent co-authors include Jerold Chun, Yun C. Yung, Peter V. Kharchenko, Jean Fan, Kun Zhang, Blue B. Lake, Derek Gao, Song Chen, Thu Elizabeth Duong and Brandon C. Sos. Their work appears in journals such as Nature, New England Journal of Medicine and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.