Gleb Baida

768 total citations
24 papers, 577 citations indexed

About

Gleb Baida is a scholar working on Dermatology, Genetics and Molecular Biology. According to data from OpenAlex, Gleb Baida has authored 24 papers receiving a total of 577 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Dermatology, 10 papers in Genetics and 8 papers in Molecular Biology. Recurrent topics in Gleb Baida's work include Dermatology and Skin Diseases (8 papers), Estrogen and related hormone effects (6 papers) and Bacteriophages and microbial interactions (5 papers). Gleb Baida is often cited by papers focused on Dermatology and Skin Diseases (8 papers), Estrogen and related hormone effects (6 papers) and Bacteriophages and microbial interactions (5 papers). Gleb Baida collaborates with scholars based in United States, Russia and Philippines. Gleb Baida's co-authors include Irina Budunova, Alexander S. Solonin, Joel T. Dudley, Pankaj Bhalla, Marianna G. Yakubovskaya, К. I. Kirsanov, Ekaterina A. Lesovaya, Ben Readhead, Oscar R. Colamonici and Ed Croze and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Virology and Biochimica et Biophysica Acta (BBA) - Biomembranes.

In The Last Decade

Gleb Baida

24 papers receiving 560 citations

Peers

Gleb Baida
Josef Laimer Austria
Lara J. Kohler United States
Yulin Shou United States
J. Ashby United Kingdom
Bernhard Steiner Switzerland
Gleb Baida
Citations per year, relative to Gleb Baida Gleb Baida (= 1×) peers P. B. Rasmussen

Countries citing papers authored by Gleb Baida

Since Specialization
Citations

This map shows the geographic impact of Gleb Baida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gleb Baida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gleb Baida more than expected).

Fields of papers citing papers by Gleb Baida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gleb Baida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gleb Baida. The network helps show where Gleb Baida may publish in the future.

Co-authorship network of co-authors of Gleb Baida

This figure shows the co-authorship network connecting the top 25 collaborators of Gleb Baida. A scholar is included among the top collaborators of Gleb Baida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gleb Baida. Gleb Baida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chudakova, Daria A., et al.. (2023). REDD1 (regulated in development and DNA damage 1) modulates the glucocorticoid receptor function in keratinocytes. Experimental Dermatology. 32(10). 1725–1733. 2 indexed citations
2.
Lesovaya, Ekaterina A., et al.. (2022). The long winding road to the safer glucocorticoid receptor (GR) targeting therapies. Oncotarget. 13(1). 408–424. 28 indexed citations
3.
Kłopot, Anna, Gleb Baida, Alexander Kel, et al.. (2021). Transcriptome Analysis Reveals Intrinsic Proinflammatory Signaling in Healthy African American Skin. Journal of Investigative Dermatology. 142(5). 1360–1371.e15. 10 indexed citations
4.
Lesovaya, Ekaterina A., О. В. Морозова, К. I. Kirsanov, et al.. (2020). A Novel Approach to Safer Glucocorticoid Receptor–Targeted Anti-lymphoma Therapy via REDD1 (Regulated in Development and DNA Damage 1) Inhibition. Molecular Cancer Therapeutics. 19(9). 1898–1908. 8 indexed citations
5.
Rivera-Gonzalez, Guillermo C., et al.. (2020). Regulated in Development and DNA Damage Responses 1 Prevents Dermal Adipocyte Differentiation and Is Required for Hair Cycle–Dependent Dermal Adipose Expansion. Journal of Investigative Dermatology. 140(9). 1698–1705.e1. 7 indexed citations
6.
Baida, Gleb, Shivani Agarwal, Ben Readhead, Joel T. Dudley, & Irina Budunova. (2020). Sexual dimorphism in atrophic effects of topical glucocorticoids is driven by differential regulation of atrophogene REDD1 in male and female skin. Oncotarget. 11(4). 409–418. 6 indexed citations
7.
Lili, Loukia N., Anna Kłopot, Benjamin Readhead, et al.. (2019). Transcriptomic Network Interactions in Human Skin Treated with Topical Glucocorticoid Clobetasol Propionate. Journal of Investigative Dermatology. 139(11). 2281–2291. 18 indexed citations
8.
Lesovaya, Ekaterina A., Shivani Agarwal, Ben Readhead, et al.. (2018). Rapamycin Modulates Glucocorticoid Receptor Function, Blocks Atrophogene REDD1, and Protects Skin from Steroid Atrophy. Journal of Investigative Dermatology. 138(9). 1935–1944. 29 indexed citations
9.
Sarkar, Mrinal K., Nihal Kaplan, Lam C. Tsoi, et al.. (2017). Endogenous Glucocorticoid Deficiency in Psoriasis Promotes Inflammation and Abnormal Differentiation. Journal of Investigative Dermatology. 137(7). 1474–1483. 40 indexed citations
10.
Kłopot, Anna, Gleb Baida, Pankaj Bhalla, Guy Haegeman, & Irina Budunova. (2015). Selective Activator of the Glucocorticoid Receptor Compound A Dissociates Therapeutic and Atrophogenic Effects of Glucocorticoid Receptor Signaling in Skin. Journal of Cancer Prevention. 20(4). 250–259. 15 indexed citations
11.
Baida, Gleb, Pankaj Bhalla, К. I. Kirsanov, et al.. (2014). REDD 1 functions at the crossroads between the therapeutic and adverse effects of topical glucocorticoids. EMBO Molecular Medicine. 7(1). 42–58. 51 indexed citations
12.
Ghadge, Ghanashyam D., Robert L. Wollmann, Gleb Baida, Μαρία Τράκα, & Raymond P. Roos. (2011). The L-Coding Region of the DA Strain of Theiler's Murine Encephalomyelitis Virus Causes Dysfunction and Death of Myelin-Synthesizing Cells. Journal of Virology. 85(18). 9377–9384. 5 indexed citations
13.
Stavrou, Spyridon, Gleb Baida, Ekaterina G. Viktorova, et al.. (2009). Theiler's Murine Encephalomyelitis Virus L* Amino Acid Position 93 Is Important for Virus Persistence and Virus-Induced Demyelination. Journal of Virology. 84(3). 1348–1354. 8 indexed citations
14.
Baida, Gleb, Brian Popko, Robert L. Wollmann, et al.. (2008). A Subgenomic Segment of Theiler's Murine Encephalomyelitis Virus RNA Causes Demyelination. Journal of Virology. 82(12). 5879–5886. 5 indexed citations
15.
Sandoval, Raudel, Jiaping Xue, Xinyong Tian, et al.. (2006). A mutant allele of BARA/LIN-9 rescues the cdk4−/− phenotype by releasing the repression on E2F-regulated genes. Experimental Cell Research. 312(13). 2465–2475. 12 indexed citations
17.
Baida, Gleb. (1999). Complete nucleotide sequence and molecular characterization of hemolysin II gene from Bacillus cereus. FEMS Microbiology Letters. 180(1). 7–14. 1 indexed citations
18.
Baida, Gleb, et al.. (1999). Complete nucleotide sequence and molecular characterization of hemolysin II gene from Bacillus cereus. FEMS Microbiology Letters. 180(1). 7–14. 54 indexed citations
19.
Baida, Gleb, et al.. (1996). Mechanism of action of hemolysin III from Bacillus cereus. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1284(2). 122–124. 91 indexed citations
20.
Baida, Gleb, et al.. (1995). Cloning and primary structure of a new hemolysin gene from Bacillus cereus. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1264(2). 151–154. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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