Giuseppina Nicolini
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- Thyroid Disorders and Treatments 6
- Hematology top 10%
- Hematopoietic Stem Cell Transplantation 5
- Genetics top 10%
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- Cardiac Fibrosis and Remodeling 5
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- Ion channel regulation and function 10
- Mitochondrial Function and Pathology 9
- Ion Transport and Channel Regulation 6
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- Cardiac Ischemia and Reperfusion 7
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- Erythrocyte Function and Pathophysiology 4
- Co-authors
- Francesca ForiniGiorgio IervasiClaudia KusmicSilvana BalzanLetizia PittoValter LubranoMarco MatteucciLaura Mariani
- Partner nations
- ItalyCzechiaUnited States
In The Last Decade
Giuseppina Nicolini
45 papers receiving 935 citations
Peers
Comparison fields: 5 of 87
- Endocrinology, Diabetes and Metabolism 272
- Hematology 102
- Genetics 91
- Cancer Research 110
- Cardiology and Cardiovascular Medicine 166
Countries citing papers authored by Giuseppina Nicolini
This map shows the geographic impact of Giuseppina Nicolini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giuseppina Nicolini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giuseppina Nicolini more than expected).
Fields of papers citing papers by Giuseppina Nicolini
This network shows the impact of papers produced by Giuseppina Nicolini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giuseppina Nicolini. The network helps show where Giuseppina Nicolini may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Giuseppina Nicolini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 1 | |
| 3 | 2024 | 1 | |
| 4 | 2023 | 4 | |
| 5 | 2023 | 5 | |
| 6 | 2023 | 10 | |
| 7 | 2020 | 13 | |
| 8 | 2019 | 44 | |
| 9 | 2019 | 6 | |
| 10 | 2018 | 21 | |
| 11 | 2016 | 32 | |
| 12 | 2015 | 28 | |
| 13 | 2013 | 29 | |
| 14 | 2012 | 45 | |
| 15 | 2011 | 1 | |
| 16 | 2010 | 77 | |
| 17 | Early long‐term L‐T3 replacement rescues mitochondria and prevents ischemic cardiac remodeling in rats | 2009 | 2 |
| 18 | 2009 | 11 | |
| 19 | 2002 | 12 | |
| 20 | 2000 | 17 |
About Giuseppina Nicolini
Giuseppina Nicolini is a scholar working on Endocrinology, Diabetes and Metabolism, Cardiology and Cardiovascular Medicine and Hematology, having authored 47 papers that have together received 957 indexed citations. Recurring topics across this work include Ion channel regulation and function (10 papers), Mitochondrial Function and Pathology (9 papers), Cardiac Ischemia and Reperfusion (7 papers), Ion Transport and Channel Regulation (6 papers), Thyroid Disorders and Treatments (6 papers), Hematopoietic Stem Cell Transplantation (5 papers), Cardiac Fibrosis and Remodeling (5 papers) and Erythrocyte Function and Pathophysiology (4 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (272 citations), Hematology (102 citations) and Genetics (91 citations). Giuseppina Nicolini has collaborated with scholars based in Italy, Czechia and United States. Frequent co-authors include Francesca Forini, Giorgio Iervasi, Claudia Kusmic, Silvana Balzan, Letizia Pitto, Valter Lubrano, Marco Matteucci, Laura Mariani, Riccardo Zucchi and R. Del Carratore.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.