Giuseppe Paradies

9.1k total citations · 2 hit papers
87 papers, 7.5k citations indexed

About

Giuseppe Paradies is a scholar working on Molecular Biology, Clinical Biochemistry and Physiology. According to data from OpenAlex, Giuseppe Paradies has authored 87 papers receiving a total of 7.5k indexed citations (citations by other indexed papers that have themselves been cited), including 76 papers in Molecular Biology, 32 papers in Clinical Biochemistry and 19 papers in Physiology. Recurrent topics in Giuseppe Paradies's work include Mitochondrial Function and Pathology (73 papers), Metabolism and Genetic Disorders (32 papers) and ATP Synthase and ATPases Research (23 papers). Giuseppe Paradies is often cited by papers focused on Mitochondrial Function and Pathology (73 papers), Metabolism and Genetic Disorders (32 papers) and ATP Synthase and ATPases Research (23 papers). Giuseppe Paradies collaborates with scholars based in Italy, United States and Netherlands. Giuseppe Paradies's co-authors include Francesca Ruggiero, Giuseppe Petrosillo, Valeria Paradies, Marilva Pistolese, Sergio Papa, Ernesto Quagliariello, Nicola Di Venosa, Nicola Moro, Antonio Federici and Maria Nicola Gadaleta and has published in prestigious journals such as Journal of Biological Chemistry, Circulation Research and The FASEB Journal.

In The Last Decade

Giuseppe Paradies

84 papers receiving 7.4k citations

Hit Papers

Oxidative stress, cardiolipin and mitochondrial dysfuncti... 2014 2026 2018 2022 2014 2019 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Giuseppe Paradies Italy 49 5.3k 1.7k 1.3k 827 824 87 7.5k
Giuseppe Petrosillo Italy 41 4.4k 0.8× 1.3k 0.8× 813 0.6× 826 1.0× 564 0.7× 56 6.2k
John M. Land United Kingdom 42 4.2k 0.8× 2.4k 1.4× 1.5k 1.2× 480 0.6× 691 0.8× 111 7.9k
Alberto Boveris Argentina 55 5.4k 1.0× 3.8k 2.3× 566 0.4× 1.0k 1.3× 670 0.8× 136 11.0k
Anatoly A. Starkov United States 51 7.0k 1.3× 2.8k 1.7× 1.1k 0.8× 740 0.9× 803 1.0× 122 11.5k
A Boveris Argentina 22 4.0k 0.8× 1.8k 1.1× 467 0.4× 627 0.8× 473 0.6× 42 7.8k
Narayan G. Avadhani United States 54 6.8k 1.3× 2.2k 1.3× 806 0.6× 600 0.7× 895 1.1× 176 10.3k
Mark A. Yorek United States 45 3.6k 0.7× 4.1k 2.4× 2.2k 1.7× 518 0.6× 727 0.9× 168 11.0k
Louis Hue Belgium 63 9.1k 1.7× 2.8k 1.6× 746 0.6× 619 0.7× 1.4k 1.7× 198 13.4k
Luke I. Szweda United States 60 6.6k 1.3× 2.9k 1.7× 1.2k 0.9× 994 1.2× 1.0k 1.3× 140 11.1k
Kathryn F. LaNoue United States 49 4.3k 0.8× 1.6k 0.9× 1.7k 1.3× 446 0.5× 360 0.4× 102 7.9k

Countries citing papers authored by Giuseppe Paradies

Since Specialization
Citations

This map shows the geographic impact of Giuseppe Paradies's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giuseppe Paradies with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giuseppe Paradies more than expected).

Fields of papers citing papers by Giuseppe Paradies

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Giuseppe Paradies. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giuseppe Paradies. The network helps show where Giuseppe Paradies may publish in the future.

Co-authorship network of co-authors of Giuseppe Paradies

This figure shows the co-authorship network connecting the top 25 collaborators of Giuseppe Paradies. A scholar is included among the top collaborators of Giuseppe Paradies based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giuseppe Paradies. Giuseppe Paradies is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Paradies, Giuseppe, Valeria Paradies, Francesca Ruggiero, & Giuseppe Petrosillo. (2013). Cardiolipin and Mitochondrial Function in Health and Disease. Antioxidants and Redox Signaling. 20(12). 1925–1953. 91 indexed citations
2.
Petrosillo, Giuseppe, et al.. (2013). Decline in cytochrome c oxidase activity in rat-brain mitochondria with aging. Role of peroxidized cardiolipin and beneficial effect of melatonin. Journal of Bioenergetics and Biomembranes. 45(5). 431–440. 53 indexed citations
3.
Paradies, Giuseppe, Valeria Paradies, Francesca Ruggiero, & Giuseppe Petrosillo. (2012). Changes in the mitochondrial permeability transition pore in aging and age-associated diseases. Mechanisms of Ageing and Development. 134(1-2). 1–9. 48 indexed citations
4.
Paradies, Giuseppe, Giuseppe Petrosillo, Valeria Paradies, & Francesca Ruggiero. (2010). Oxidative stress, mitochondrial bioenergetics, and cardiolipin in aging. Free Radical Biology and Medicine. 48(10). 1286–1295. 134 indexed citations
5.
Petrosillo, Giuseppe, Giuseppe Colantuono, Nicola Moro, et al.. (2009). Melatonin protects against heart ischemia-reperfusion injury by inhibiting mitochondrial permeability transition pore opening. American Journal of Physiology-Heart and Circulatory Physiology. 297(4). H1487–H1493. 137 indexed citations
6.
Petrosillo, Giuseppe, Nicola Moro, Francesca Ruggiero, & Giuseppe Paradies. (2009). Melatonin inhibits cardiolipin peroxidation in mitochondria and prevents the mitochondrial permeability transition and cytochrome c release. Free Radical Biology and Medicine. 47(7). 969–974. 100 indexed citations
7.
Petrosillo, Giuseppe, Nicola Di Venosa, Francesca Ruggiero, et al.. (2005). Mitochondrial dysfunction associated with cardiac ischemia/reperfusion can be attenuated by oxygen tension control. Role of oxygen-free radicals and cardiolipin. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1710(2-3). 78–86. 75 indexed citations
8.
Montinaro, L., Giuseppe Paradies, & Samuele Leggio. (2002). The Lortat-Jacob operation by laparoscopic access to treat gastroesophageal reflux in pediatric patients. Surgical Endoscopy. 16(10). 1438–1440. 2 indexed citations
9.
Paradies, Giuseppe, Giuseppe Petrosillo, Marilva Pistolese, & Francesca Ruggiero. (2001). Reactive oxygen species generated by the mitochondrial respiratory chain affect the complex III activity via cardiolipin peroxidation in beef-heart submitochondrial particles. Mitochondrion. 1(2). 151–159. 106 indexed citations
10.
Paradies, Giuseppe, Giuseppe Petrosillo, Marilva Pistolese, & Francesca Ruggiero. (2000). The effect of reactive oxygen species generated from the mitochondrial electron transport chain on the cytochrome c oxidase activity and on the cardiolipin content in bovine heart submitochondrial particles. FEBS Letters. 466(2-3). 323–326. 217 indexed citations
11.
Paradies, Giuseppe, Giuseppe Petrosillo, Maria Nicola Gadaleta, & Francesca Ruggiero. (1999). The effect of aging and acetyl‐L‐carnitine on the pyruvate transport andoxidation in rat heart mitochondria. FEBS Letters. 454(3). 207–209. 71 indexed citations
12.
Paradies, Giuseppe, Giuseppe Petrosillo, & Francesca Ruggiero. (1997). Cardiolipin-dependent decrease of cytochrome c oxidase activity in heart mitochondria from hypothyroid rats. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1319(1). 5–8. 80 indexed citations
13.
Paradies, Giuseppe, Francesca Ruggiero, Giuseppe Petrosillo, M. N. Gadaleta, & E Quagliariello. (1994). The Effect of Aging and Acetyl‐L‐carnitine on the Function and on the Lipid Composition of Rat Heart Mitochondriaa. Annals of the New York Academy of Sciences. 717(1). 233–243. 30 indexed citations
14.
Paradies, Giuseppe, Francesca Ruggiero, Giuseppe Petrosillo, Maria Nicola Gadaleta, & Ernesto Quagliariello. (1994). Effect of aging and acetyl‐l‐carnitine on the activity of cytochrome oxidase and adenine nucleotide translocase in rat heart mitochondria. FEBS Letters. 350(2-3). 213–215. 90 indexed citations
15.
Paradies, Giuseppe, et al.. (1993). Decreased Cytochrome Oxidase Activity and Changes in Phospholipids in Heart Mitochondria from Hypothyroid Rats. Archives of Biochemistry and Biophysics. 307(1). 91–95. 60 indexed citations
16.
Paradies, Giuseppe, Francesca Ruggiero, Maria Nicola Gadaleta, & Ernesto Quagliariello. (1992). The effect of aging and acetyl-l-carnitine on the activity of the phosphate carrier and on the phospholipid composition in rat heart mitochondria. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1103(2). 324–326. 100 indexed citations
17.
Paradies, Giuseppe & Francesca Ruggiero. (1991). Effect of aging on the activity of the phosphate carrier and on the lipid composition in rat liver mitochondria. Archives of Biochemistry and Biophysics. 284(2). 332–337. 72 indexed citations
18.
Paradies, Giuseppe & Francesca Ruggiero. (1989). Decreased activity of the pyruvate translocator and changes in the lipid composition in heart mitochondria from hypothyroid rats. Archives of Biochemistry and Biophysics. 269(2). 595–602. 40 indexed citations
19.
Paradies, Giuseppe. (1988). The effect of phenylglyoxal on the translocation of pyruvate in rat-heart mitochondria. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 932(1). 1–7. 6 indexed citations
20.
Papa, Sergio, et al.. (1970). Studies on the mechanism of action ofncouplers on the transport of inorganic phosphate in rat liver mitochondria. FEBS Letters. 6(1). 1–4. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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