Gísli Másson
Impact in
- Genetics top 0.1%
- Genetic Associations and Epidemiology
- Genomics and Rare Diseases
- Genomic variations and chromosomal abnormalities
- Genetic Mapping and Diversity in Plants and Animals
- BRCA gene mutations in cancer
- Evolution and Genetic Dynamics
- Cancer Research top 1%
- Cancer Genomics and Diagnostics
Papers in
- Genetics 63
- Genetic Associations and Epidemiology 33
- Genomics and Rare Diseases 17
- Genomic variations and chromosomal abnormalities 14
- Genetics and Neurodevelopmental Disorders 8
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- Epigenetics and DNA Methylation 11
- Genomics and Chromatin Dynamics 9
- RNA modifications and cancer 8
- DNA Repair Mechanisms 7
- Co-authors
- Kāri Stefánsson (24 shared papers)Augustine Kong (19 shared papers)Daniel Fannar Gudbjartsson (19 shared papers)Michael L. Frigge (8 shared papers)Unnur Arna Thorsteinsdottir (18 shared papers)Sigurjon Axel Gudjonsson (8 shared papers)Guðmar Þorleifsson (10 shared papers)Agnar Helgason (14 shared papers)
- Journals
- Nature Genetics (46 papers)Nature Communications (24 papers)Nature (12 papers)Human Molecular Genetics (6 papers)Science (5 papers)
- Partner nations
- IcelandNetherlandsUnited States
In The Last Decade
Gísli Másson
132 papers receiving 25.3k citations
Gísli Másson's Hit Papers
Peers
Comparison fields: 5 of 214
- Genetics 9.8k
- Cancer Research 2.0k
- Biological Psychiatry 323
- Molecular Biology 8.4k
- Aging 185
Countries citing papers authored by Gísli Másson
This map shows the geographic impact of Gísli Másson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gísli Másson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gísli Másson more than expected).
Fields of papers citing papers by Gísli Másson
This network shows the impact of papers produced by Gísli Másson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gísli Másson. The network helps show where Gísli Másson may publish in the future.
Co-authors
The 25 scholars most cited alongside Gísli Másson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 133 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Rate of de novo mutations and the importance of father’s age to disease risk Hit paper breakdown → | 2012 | 1534 |
| 2 | Common variants conferring risk of schizophrenia Hit paper breakdown → | 2009 | 1402 |
| 3 | A high-resolution recombination map of the human genome Hit paper breakdown → | 2002 | 1397 |
| 4 | A Common Variant on Chromosome 9p21 Affects the Risk of Myocardial Infarction Hit paper breakdown → | 2007 | 1332 |
| 5 | Variants in ELL2 influencing immunoglobulin levels associate with multiple myeloma Hit paper breakdown → | 2015 | 1024 |
| 6 | Large-scale integration of the plasma proteome with genetics and disease Hit paper breakdown → | 2021 | 925 |
| 7 | Spread of SARS-CoV-2 in the Icelandic Population Hit paper breakdown → | 2020 | 907 |
| 8 | Humoral Immune Response to SARS-CoV-2 in Iceland Hit paper breakdown → | 2020 | 709 |
| 9 | 2007 | 663 | |
| 10 | 2005 | 634 | |
| 11 | Clonal hematopoiesis, with and without candidate driver mutations, is common in the elderly Hit paper breakdown → | 2017 | 622 |
| 12 | The nature of nurture: Effects of parental genotypes Hit paper breakdown → | 2018 | 621 |
| 13 | Large-scale whole-genome sequencing of the Icelandic population Hit paper breakdown → | 2015 | 583 |
| 14 | Common Sequence Variants in the LOXL1 Gene Confer Susceptibility to Exfoliation Glaucoma Hit paper breakdown → | 2007 | 542 |
| 15 | 2009 | 521 | |
| 16 | Parent-of-origin-specific allelic associations among 106 genomic loci for age at menarche Hit paper breakdown → | 2014 | 458 |
| 17 | 2009 | 449 | |
| 18 | 2010 | 431 | |
| 19 | Loss-of-function mutations in SLC30A8 protect against type 2 diabetes Hit paper breakdown → | 2014 | 402 |
| 20 | 2008 | 392 |
About Gísli Másson
Gísli Másson is a scholar working on Genetics, Molecular Biology, Rheumatology, Pathology and Forensic Medicine and Genetics, having authored 133 papers that have together received 26.0k indexed citations. Recurring topics across this work include Genetic Associations and Epidemiology (33 papers), Genomics and Rare Diseases (17 papers), Genomic variations and chromosomal abnormalities (14 papers), Epigenetics and DNA Methylation (11 papers), Genomics and Chromatin Dynamics (9 papers), RNA modifications and cancer (8 papers), Genetics and Neurodevelopmental Disorders (8 papers) and DNA Repair Mechanisms (7 papers). The work is most often cited by research in Genetics (9.8k citations), Cancer Research (2.0k citations), Biological Psychiatry (323 citations), Molecular Biology (8.4k citations) and Aging (185 citations). Gísli Másson has collaborated with scholars based in Iceland, Netherlands and United States. Frequent co-authors include Kāri Stefánsson, Augustine Kong, Daniel Fannar Gudbjartsson, Michael L. Frigge, Unnur Arna Thorsteinsdottir, Sigurjon Axel Gudjonsson, Guðmar Þorleifsson, Agnar Helgason, Thorgeir Elís Thorgeirsson and Patrick Sulem. Their work appears in journals such as Nature Genetics, Nature Communications, Nature, Human Molecular Genetics and Science.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.