Gianluigi Castoldi
- Hematology top 2%
- Immunology top 10%
- Genetics top 5%
- Oncology top 10%
- Molecular Biology
- Co-authors
- Stig RödjerFranco DammaccoGian Matteo RigolinFrancesco LanzaAntonio CuneoAntonella BardiMatteo Giovanni Della PortaSabrina Moretti
- Topics
- Acute Myeloid Leukemia Research (14 papers)Chronic Lymphocytic Leukemia Research (9 papers)Lymphoma Diagnosis and Treatment (9 papers)
- Cited by
- HematologyGeneticsImmunology
- Partner nations
- ItalyBelgiumUnited Kingdom
In The Last Decade
Gianluigi Castoldi
36 papers receiving 934 citations
Peers
Comparison fields: 5 of 69
- Hematology 483
- Immunology 330
- Genetics 278
- Oncology 252
- Molecular Biology 237
Countries citing papers authored by Gianluigi Castoldi
This map shows the geographic impact of Gianluigi Castoldi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gianluigi Castoldi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gianluigi Castoldi more than expected).
Fields of papers citing papers by Gianluigi Castoldi
This network shows the impact of papers produced by Gianluigi Castoldi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gianluigi Castoldi. The network helps show where Gianluigi Castoldi may publish in the future.
Co-authorship network of co-authors of Gianluigi Castoldi
This figure shows the co-authorship network connecting the top 25 collaborators of Gianluigi Castoldi. A scholar is included among the top collaborators of Gianluigi Castoldi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gianluigi Castoldi. Gianluigi Castoldi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Validation of an interphase fluorescence in situ hybridization approach for the detection of MLL gene rearrangements and of the MLL/AF9 fusion in acute myeloid leukemia. | 3 |
| 2 | 3 | |
| 3 | 75 | |
| 4 | 10 | |
| 5 | 35 | |
| 6 | 24 | |
| 7 | 16 | |
| 8 | 56 | |
| 9 | 27 | |
| 10 | 14 | |
| 11 | 139 | |
| 12 | 23 | |
| 13 | 12 | |
| 14 | 40 | |
| 15 | 4 | |
| 16 | 35 | |
| 17 | 2 | |
| 18 | 9 | |
| 19 | 12 | |
| 20 | 5 |
About Gianluigi Castoldi
Gianluigi Castoldi is a scholar working on Hematology, Genetics and Immunology, having authored 36 papers that have together received 957 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (14 papers), Chronic Lymphocytic Leukemia Research (9 papers) and Lymphoma Diagnosis and Treatment (9 papers). The work is most often cited by research in Hematology (483 citations), Genetics (278 citations) and Immunology (330 citations). Gianluigi Castoldi has collaborated with scholars based in Italy, Belgium and United Kingdom. Frequent co-authors include Stig Rödjer, Franco Dammacco, Gian Matteo Rigolin, Francesco Lanza, Antonio Cuneo, Antonella Bardi, Matteo Giovanni Della Porta, Sabrina Moretti, Renato Bigoni and Barbara Castagnari. Their work appears in journals such as Blood, Cancer and British Journal of Haematology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.