George P. Studzinski

6.6k citations
180 papers · 5.4k indexed · h-index 43

Impact in

Papers in

George P. Studzinski

180 papers receiving 5.2k citations

Peers

George P. Studzinski
Comparison fields: 5 of 122
  • Pathology and Forensic Medicine 1.3k
  • Molecular Biology 3.5k
  • Hematology 552
  • Toxicology 155
  • Oncology 1.2k
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Citations per field
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Citations per year

Countries citing papers authored by George P. Studzinski

Since Specialization
Citations

This map shows the geographic impact of George P. Studzinski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by George P. Studzinski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites George P. Studzinski more than expected).

Fields of papers citing papers by George P. Studzinski

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by George P. Studzinski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by George P. Studzinski. The network helps show where George P. Studzinski may publish in the future.

Co-authors

The 25 scholars most cited alongside George P. Studzinski, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with George P. Studzinski Line = papers co-authored together George P. Studzinski links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20189
2 20166
3 201418
4 201212
5 201022
6 201017
7 201038
8 200561
9 200525
10 200212
11 200211
12 2001119
13 200183
14
Apoptosis : a practical approach
199945
15 199816
16 199730
17
Transition to tetraploidy in 1,25-dihydroxyvitamin D3-resistant HL60 cells is preceded by reduced growth factor dependence and constitutive up-regulation of Sp1 and AP-1 transcription factors.
199624
18 199361
19 19914
20 196333

About George P. Studzinski

George P. Studzinski is a scholar working on Pathology and Forensic Medicine, Molecular Biology, Hematology, Oncology and Toxicology, having authored 180 papers that have together received 5.4k indexed citations. Recurring topics across this work include Retinoids in leukemia and cellular processes (47 papers), Vitamin D Research Studies (39 papers), Cancer-related Molecular Pathways (26 papers), DNA and Nucleic Acid Chemistry (24 papers), Acute Myeloid Leukemia Research (17 papers), DNA Repair Mechanisms (17 papers), Cytokine Signaling Pathways and Interactions (13 papers) and Estrogen and related hormone effects (12 papers). The work is most often cited by research in Pathology and Forensic Medicine (1.3k citations), Molecular Biology (3.5k citations), Hematology (552 citations), Toxicology (155 citations) and Oncology (1.2k citations). George P. Studzinski has collaborated with scholars based in United States, Israel and Poland. Frequent co-authors include Xuening Wang, Michael Danilenko, Elżbieta Gocek, Zamir Brelvi, Xuening Wang, Qing Mei Wang, Jennifer B. Jones, Milan R. Uskoković, Jonathan S. Harrison and Susan C. Feldman. Their work appears in journals such as Journal of Cellular Physiology, Experimental Cell Research, Journal of Cellular Biochemistry, The Journal of Steroid Biochemistry and Molecular Biology and JNCI Journal of the National Cancer Institute.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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