Atan Gross

10.4k total citations · 5 hit papers
32 papers, 8.8k citations indexed

About

Atan Gross is a scholar working on Molecular Biology, Epidemiology and Immunology. According to data from OpenAlex, Atan Gross has authored 32 papers receiving a total of 8.8k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 8 papers in Epidemiology and 6 papers in Immunology. Recurrent topics in Atan Gross's work include Cell death mechanisms and regulation (28 papers), RNA Interference and Gene Delivery (10 papers) and DNA Repair Mechanisms (5 papers). Atan Gross is often cited by papers focused on Cell death mechanisms and regulation (28 papers), RNA Interference and Gene Delivery (10 papers) and DNA Repair Mechanisms (5 papers). Atan Gross collaborates with scholars based in Israel, United States and Canada. Atan Gross's co-authors include S J Korsmeyer, James M. McDonnell, Stanley J. Korsmeyer, Michael C. Wei, Jennifer Jockel, Sandra S. Zinkel, Kevin A. Roth, Elizabeth Yang, Barbara J. Klocke and Yongge Zhao and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Atan Gross

32 papers receiving 8.6k citations

Hit Papers

BCL-2 family members and the mitochondria in apoptosis 1998 2026 2007 2016 1999 1998 1999 1999 1998 1000 2.0k 3.0k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Atan Gross Israel 25 7.0k 1.5k 1.4k 1.2k 999 32 8.8k
Henning R. Stennicke Denmark 29 6.6k 0.9× 1.5k 1.0× 1.5k 1.0× 933 0.8× 938 0.9× 56 8.9k
Xu Luo United States 31 7.0k 1.0× 1.4k 1.0× 1.3k 0.9× 1.2k 1.1× 1.2k 1.2× 67 9.3k
Paul G. Ekert Australia 41 5.9k 0.8× 1.7k 1.2× 1.3k 0.9× 1.3k 1.1× 844 0.8× 128 8.3k
Deepak Nijhawan United States 19 6.8k 1.0× 1.2k 0.8× 1.4k 1.0× 981 0.8× 955 1.0× 28 9.1k
Christoph Borner Germany 50 6.1k 0.9× 1.6k 1.1× 1.3k 0.9× 1.2k 1.0× 715 0.7× 119 8.8k
Ruth M. Kluck Australia 38 9.4k 1.3× 1.7k 1.2× 1.6k 1.1× 1.3k 1.1× 1.1k 1.1× 66 11.9k
Tomomi Kuwana United States 28 7.9k 1.1× 1.2k 0.8× 1.6k 1.1× 1.2k 1.0× 869 0.9× 46 10.0k
Beni B. Wolf United States 26 5.3k 0.8× 1.2k 0.8× 1.3k 0.9× 801 0.7× 893 0.9× 53 7.6k
Masato Enari Japan 32 7.1k 1.0× 1.7k 1.2× 1.3k 0.9× 703 0.6× 850 0.9× 50 8.8k
Shile Huang United States 55 5.3k 0.8× 1.4k 0.9× 1.3k 0.9× 1.2k 1.1× 1.1k 1.1× 177 9.7k

Countries citing papers authored by Atan Gross

Since Specialization
Citations

This map shows the geographic impact of Atan Gross's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Atan Gross with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Atan Gross more than expected).

Fields of papers citing papers by Atan Gross

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Atan Gross. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Atan Gross. The network helps show where Atan Gross may publish in the future.

Co-authorship network of co-authors of Atan Gross

This figure shows the co-authorship network connecting the top 25 collaborators of Atan Gross. A scholar is included among the top collaborators of Atan Gross based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Atan Gross. Atan Gross is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gross, Atan & Samuel G. Katz. (2017). Non-apoptotic functions of BCL-2 family proteins. Cell Death and Differentiation. 24(8). 1348–1358. 146 indexed citations
2.
Katz, Chen, et al.. (2012). Molecular Basis of the Interaction between Proapoptotic Truncated BID (tBID) Protein and Mitochondrial Carrier Homologue 2 (MTCH2) Protein. Journal of Biological Chemistry. 287(18). 15016–15023. 33 indexed citations
3.
Maryanovich, Maria & Atan Gross. (2012). A ROS rheostat for cell fate regulation. Trends in Cell Biology. 23(3). 129–134. 156 indexed citations
4.
Addadi, Yoseph, et al.. (2011). Utilizing mitochondrial events as biomarkers for imaging apoptosis. Cell Death and Disease. 2(6). e166–e166. 12 indexed citations
5.
Ziporen, Lea, et al.. (2009). Programmed Necrotic Cell Death Induced by Complement Involves a Bid-Dependent Pathway. The Journal of Immunology. 182(1). 515–521. 30 indexed citations
6.
Buitrago‐Molina, Laura Elisa, Margitta Lebofsky, Michael P. Manns, et al.. (2009). The strength of the Fas ligand signal determines whether hepatocytes act as type 1 or type 2 cells in murine livers #. Hepatology. 50(5). 1558–1566. 32 indexed citations
7.
Gross, Atan. (2008). A new Aven-ue to DNA-damage checkpoints. Trends in Biochemical Sciences. 33(11). 514–516. 4 indexed citations
8.
Oberkovitz, Galia, Limor Regev, & Atan Gross. (2007). Nucleocytoplasmic shuttling of BID is involved in regulating its activities in the DNA-damage response. Cell Death and Differentiation. 14(9). 1628–1634. 13 indexed citations
9.
Andrade‐Navarro, Miguel A., et al.. (2007). Mitochondrial carriers and pores: Key regulators of the mitochondrial apoptotic program?. APOPTOSIS. 12(5). 869–876. 63 indexed citations
10.
König, Hans‐Georg, Markus Rehm, Stan Krajewski, et al.. (2007). Full Length Bid is sufficient to induce apoptosis of cultured rat hippocampal neurons. BMC Cell Biology. 8(1). 7–7. 38 indexed citations
11.
Zinkel, Sandra S., Atan Gross, & Elizabeth Yang. (2006). BCL2 family in DNA damage and cell cycle control. Cell Death and Differentiation. 13(8). 1351–1359. 395 indexed citations
12.
Gross, Atan. (2006). BID as a Double Agent in Cell Life and Death. Cell Cycle. 5(6). 582–584. 27 indexed citations
13.
Kamer, Iris, Rachel Sarig, Yehudit Zaltsman, et al.. (2005). Proapoptotic BID Is an ATM Effector in the DNA-Damage Response. Cell. 122(4). 593–603. 178 indexed citations
14.
Gross, Atan. (2005). Mitochondrial Carrier Homolog 2: A Clue to Cracking the BCL-2 Family Riddle?. Journal of Bioenergetics and Biomembranes. 37(3). 113–119. 15 indexed citations
15.
Sarig, Rachel, Yehudit Zaltsman, Dan Frumkin, et al.. (2002). tBID Homooligomerizes in the Mitochondrial Membrane to Induce Apoptosis. Journal of Biological Chemistry. 277(14). 12237–12245. 132 indexed citations
16.
Breckenridge, David G., Mai Nguyen, Ing Swie Goping, et al.. (2000). BID-dependent and BID-independent pathways for BAX insertion into mitochondria. Cell Death and Differentiation. 7(11). 1101–1108. 99 indexed citations
17.
Gross, Atan, Xiao Ming Yin, Michael C. Wei, et al.. (1999). Caspase Cleaved BID Targets Mitochondria and Is Required for Cytochrome c Release, while BCL-XL Prevents This Release but Not Tumor Necrosis Factor-R1/Fas Death. Journal of Biological Chemistry. 274(2). 1156–1163. 876 indexed citations breakdown →
18.
Yin, Xiao‐Ming, Kun Wang, Atan Gross, et al.. (1999). Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis. Nature. 400(6747). 886–891. 823 indexed citations breakdown →
19.
Gross, Atan, Jennifer Jockel, Michael C. Wei, & Stanley J. Korsmeyer. (1998). Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis. The EMBO Journal. 17(14). 3878–3885. 937 indexed citations breakdown →
20.
Tytgat, Jan, Ken Nakazawa, Atan Gross, & Philip E. Hess. (1993). Pursuing the voltage sensor of a voltage-gated mammalian potassium channel.. Journal of Biological Chemistry. 268(32). 23777–23779. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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