Garrett A. Kaas

763 total citations
11 papers, 546 citations indexed

About

Garrett A. Kaas is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Garrett A. Kaas has authored 11 papers receiving a total of 546 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 3 papers in Genetics and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Garrett A. Kaas's work include Epigenetics and DNA Methylation (7 papers), Genetics and Neurodevelopmental Disorders (3 papers) and RNA modifications and cancer (2 papers). Garrett A. Kaas is often cited by papers focused on Epigenetics and DNA Methylation (7 papers), Genetics and Neurodevelopmental Disorders (3 papers) and RNA modifications and cancer (2 papers). Garrett A. Kaas collaborates with scholars based in United States. Garrett A. Kaas's co-authors include J. David Sweatt, Hongjun Song, Chun Zhong, Guo‐li Ming, Jennifer R. King, Junko Kasuya, Toshihiro Kitamoto, Iva B. Zovkic, Jing Wang and John W. Lewis and has published in prestigious journals such as Neuron, Journal of Neuroscience and Neuroscience.

In The Last Decade

Garrett A. Kaas

11 papers receiving 540 citations

Peers

Countries citing papers authored by Garrett A. Kaas

Since Specialization

Citations

This map shows the geographic impact of Garrett A. Kaas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Garrett A. Kaas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Garrett A. Kaas more than expected).

Fields of papers citing papers by Garrett A. Kaas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Garrett A. Kaas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Garrett A. Kaas. The network helps show where Garrett A. Kaas may publish in the future.

Co-authorship network of co-authors of Garrett A. Kaas

This figure shows the co-authorship network connecting the top 25 collaborators of Garrett A. Kaas. A scholar is included among the top collaborators of Garrett A. Kaas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Garrett A. Kaas. Garrett A. Kaas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	BMP Antagonist Gremlin 2 Regulates Hippocampal Neurogenesis and Is Associated with Seizure Susceptibility and Anxiety 2024 ·eNeuro ·(unknown), Garrett A. Kaas, (unknown), Eric R. Gamazon, Antonis K. Hatzopoulos	2
2	Reduced Function of the Glutathione S-Transferase S1 Suppresses Behavioral Hyperexcitability in Drosophila Expressing Mutant Voltage-Gated Sodium Channels 2020 ·G3 Genes Genomes Genetics ·Hung-Lin Chen, Junko Kasuya, (unknown), Garrett A. Kaas, (unknown), (unknown), Toshihiro Kitamoto	1
3	Tet1 Isoforms Differentially Regulate Gene Expression, Synaptic Transmission, and Memory in the Mammalian Brain 2020 ·Journal of Neuroscience ·Celeste B. Greer, (unknown), Joscha Weiss, Roman M. Lazarenko, Sean P. Moran, (unknown), (unknown), (unknown), Andrew Kennedy, J. David Sweatt, Garrett A. Kaas	27
4	Locus-Specific DNA Methylation Assays to Study Glutamate Receptor Regulation 2019 ·Methods in molecular biology ·Jordan A. Brown, J. David Sweatt, Garrett A. Kaas	2
5	Lithium-Responsive Seizure-Like Hyperexcitability Is Caused by a Mutation in theDrosophilaVoltage-Gated Sodium Channel Geneparalytic 2016 ·eNeuro ·Garrett A. Kaas, Junko Kasuya, (unknown), Atsushi Ueda, (unknown), Chun‐Fang Wu, Toshihiro Kitamoto	21
6	Obesity Weighs down Memory through a Mechanism Involving the Neuroepigenetic Dysregulation of Sirt1 2016 ·Journal of Neuroscience ·Frankie D. Heyward, (unknown), Mark A. Coleman, Cristin F. Gavin, Jing Wang, Garrett A. Kaas, (unknown), John W. Lewis, (unknown), J. David Sweatt	63
7	Tet1 oxidase regulates neuronal gene transcription, active DNA hydroxymethylation, object location memory, and threat recognition memory 2015 ·PubMed ·Dinesh Babu Uthaya Kumar, (unknown), Garrett A. Kaas, John W. Lewis, Jing Wang, (unknown), Chun Zhong, Andrew Kennedy, Hongjun Song, J. David Sweatt	37
8	TET1 Controls CNS 5-Methylcytosine Hydroxylation, Active DNA Demethylation, Gene Transcription, and Memory Formation 2013 ·Neuron ·Garrett A. Kaas, Chun Zhong, (unknown), (unknown), (unknown), Guo‐li Ming, Jennifer R. King, Hongjun Song, J. David Sweatt	313
9	Interindividual Variability in Stress Susceptibility: A Role for Epigenetic Mechanisms in PTSD 2013 ·Frontiers in Psychiatry ·Iva B. Zovkic, (unknown), Garrett A. Kaas, J. David Sweatt	46
10	A putative amino acid transporter of the solute carrier 6 family is upregulated by lithium and is required for resistance to lithium toxicity in Drosophila 2009 ·Neuroscience ·Junko Kasuya, Garrett A. Kaas, Toshihiro Kitamoto	12
11	Effects of lithium chloride on the gene expression profiles in Drosophila heads 2009 ·Neuroscience Research ·Junko Kasuya, Garrett A. Kaas, Toshihiro Kitamoto	22

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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