Gabriel Persi

26 papers receiving 284 citations

Peers

Gabriel Persi
Comparison fields: 5 of 60
  • Behavioral Neuroscience 24
  • Cellular and Molecular Neuroscience 112
  • Endocrinology, Diabetes and Metabolism 77
  • Neurology 63
  • Neurology 17
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Laura Bellingacci Italy
Stephanie L. Ciarlone United States
Weonjin Yu South Korea
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Citations per year

Countries citing papers authored by Gabriel Persi

Since Specialization
Citations

This map shows the geographic impact of Gabriel Persi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gabriel Persi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gabriel Persi more than expected).

Fields of papers citing papers by Gabriel Persi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gabriel Persi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gabriel Persi. The network helps show where Gabriel Persi may publish in the future.

Co-authors

The 25 scholars most cited alongside Gabriel Persi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gabriel Persi Line = papers co-authored together Gabriel Persi links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 31 papers — load more, or switch the sort, to bring in the rest.

#Work
1 200453
2 202045
3 200735
4 201825
5 201119
6 200918
7 201514
8 202012
9 200612
10 201410
11 200210
12 20175
13 20175
14 20175
15 20065
16
[Apathy in Parkinson's disease. Impairment in quality of life].
20095
17
Consenso de Diagnóstico y Tratamiento Agudo del Accidente Cerebrovascular Isquémico Consejo de Stroke - Sociedad Argentina de Cardiología
20123
18 20173
19 20241
20 20241

About Gabriel Persi

Gabriel Persi is a scholar working on Neurology, Cellular and Molecular Neuroscience, Endocrinology, Diabetes and Metabolism, Epidemiology and Molecular Biology, having authored 31 papers that have together received 293 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (8 papers), Adrenal Hormones and Disorders (4 papers), Parkinson's Disease Mechanisms and Treatments (4 papers), Acute Ischemic Stroke Management (3 papers), Neurological disorders and treatments (3 papers), Mitochondrial Function and Pathology (3 papers), Restless Legs Syndrome Research (2 papers) and Neurological and metabolic disorders (2 papers). The work is most often cited by research in Behavioral Neuroscience (24 citations), Cellular and Molecular Neuroscience (112 citations), Endocrinology, Diabetes and Metabolism (77 citations), Neurology (63 citations) and Neurology (17 citations). Gabriel Persi has collaborated with scholars based in Argentina, United States and Canada. Frequent co-authors include Emilia Gatto, José Luis Etcheverry, Estela Cardoso, Alejandro L. Arregger, Liliana N. Contreras, Claudia Perandones, Ferdinando Squitieri, Juan José Poderoso, Rui Gao and Tetsuo Ashizawa. Their work appears in journals such as Parkinsonism & Related Disorders, Neurology, Steroids, Journal of Parkinson s Disease and Alzheimer s & Dementia.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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