G. Webb McKnight
- Surgery top 10%
- Pharmacology top 5%
- Physiology top 10%
- Biochemistry top 5%
- Molecular Biology
- Co-authors
- John L. WallaceK.‐E. ArforsPaul L. BeckCatherine M. KeenanWallace K. MacNaughtonGary R. MartinJosé G. FerrazD. Euan MacIntyre
- Topics
- Helicobacter pylori-related gastroenterology studies (5 papers)Inflammatory mediators and NSAID effects (4 papers)Clostridium difficile and Clostridium perfringens research (3 papers)
- Partner nations
- CanadaItalyUnited States
In The Last Decade
G. Webb McKnight
18 papers receiving 922 citations
Peers
Comparison fields: 5 of 94
- Surgery 323
- Pharmacology 248
- Physiology 234
- Biochemistry 165
- Molecular Biology 146
Countries citing papers authored by G. Webb McKnight
This map shows the geographic impact of G. Webb McKnight's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Webb McKnight with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Webb McKnight more than expected).
Fields of papers citing papers by G. Webb McKnight
This network shows the impact of papers produced by G. Webb McKnight. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Webb McKnight. The network helps show where G. Webb McKnight may publish in the future.
Co-authorship network of co-authors of G. Webb McKnight
This figure shows the co-authorship network connecting the top 25 collaborators of G. Webb McKnight. A scholar is included among the top collaborators of G. Webb McKnight based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Webb McKnight. G. Webb McKnight is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 115 | |
| 2 | 98 | |
| 3 | 33 | |
| 4 | 24 | |
| 5 | 29 | |
| 6 | 23 | |
| 7 | Protective effects of PF-10040 in experimental NSAID-gastritis: role of leukocytes, leukotrienes and PAF. | 2 |
| 8 | 40 | |
| 9 | 36 | |
| 10 | 63 | |
| 11 | 31 | |
| 12 | 233 | |
| 13 | 9 | |
| 14 | 68 | |
| 15 | 24 | |
| 16 | 85 | |
| 17 | 29 | |
| 18 | 15 |
About G. Webb McKnight
G. Webb McKnight is a scholar working on Pharmacology, Gastroenterology and Biochemistry, having authored 18 papers that have together received 957 indexed citations. Recurring topics across this work include Helicobacter pylori-related gastroenterology studies (5 papers), Inflammatory mediators and NSAID effects (4 papers) and Clostridium difficile and Clostridium perfringens research (3 papers). The work is most often cited by research in Biochemistry (165 citations), Gastroenterology (97 citations) and Pharmacology (248 citations). G. Webb McKnight has collaborated with scholars based in Canada, Italy and United States. Frequent co-authors include John L. Wallace, K.‐E. Arfors, Paul L. Beck, Catherine M. Keenan, Wallace K. MacNaughton, Gary R. Martin, José G. Ferraz, D. Euan MacIntyre, Michael Dicay and Carla S. Coffin. Their work appears in journals such as The Journal of Experimental Medicine, Gastroenterology and European Journal of Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.