G. Szegedi
- Rheumatology top 1%
- Immunology top 5%
- Pathology and Forensic Medicine top 2%
- Molecular Biology
- Epidemiology top 10%
- Co-authors
- Zoltán SzekaneczPéter SzodorayGabriella LakosMargit ZeherE. BodolayEmese KissPál SoltészTünde Tarr
- Topics
- Systemic Lupus Erythematosus Research (29 papers)Systemic Sclerosis and Related Diseases (13 papers)T-cell and B-cell Immunology (11 papers)
In The Last Decade
G. Szegedi
77 papers receiving 1.8k citations
Peers
Comparison fields: 5 of 97
- Rheumatology 923
- Immunology 711
- Pathology and Forensic Medicine 611
- Molecular Biology 262
- Epidemiology 259
Countries citing papers authored by G. Szegedi
This map shows the geographic impact of G. Szegedi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Szegedi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Szegedi more than expected).
Fields of papers citing papers by G. Szegedi
This network shows the impact of papers produced by G. Szegedi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Szegedi. The network helps show where G. Szegedi may publish in the future.
Co-authorship network of co-authors of G. Szegedi
This figure shows the co-authorship network connecting the top 25 collaborators of G. Szegedi. A scholar is included among the top collaborators of G. Szegedi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Szegedi. G. Szegedi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 67 | |
| 3 | 35 | |
| 4 | 54 | |
| 5 | 22 | |
| 6 | 40 | |
| 7 | 99 | |
| 8 | 22 | |
| 9 | [Significance of anti-nucleosome (anti-chromatin) auto-antibodies in systemic lupus erythematosus]. | 3 |
| 10 | 22 | |
| 11 | 6 | |
| 12 | 2 | |
| 13 | 18 | |
| 14 | 22 | |
| 15 | 16 | |
| 16 | 6 | |
| 17 | Lymphocyte markers in patients with progressive systemic sclerosis. | 2 |
| 18 | Antibody-dependent cellular cytotoxicity in systemic lupus erythematosus. | 0 |
| 19 | 6 | |
| 20 | Comparative study on the interferon production by the leukocytes of healthy and leukaemic subjects. | 13 |
About G. Szegedi
G. Szegedi is a scholar working on Rheumatology, Immunology and Pathology and Forensic Medicine, having authored 79 papers that have together received 1.9k indexed citations. Recurring topics across this work include Systemic Lupus Erythematosus Research (29 papers), Systemic Sclerosis and Related Diseases (13 papers) and T-cell and B-cell Immunology (11 papers). The work is most often cited by research in Rheumatology (923 citations), Pathology and Forensic Medicine (611 citations) and Immunology (711 citations). G. Szegedi has collaborated with scholars based in Hungary, Norway and Israel. Frequent co-authors include Zoltán Szekanecz, Péter Szodoray, Gabriella Lakos, Margit Zeher, E. Bodolay, Emese Kiss, Pál Soltész, Tünde Tarr, Sándor Sipka and István Csípő. Their work appears in journals such as Annals of the Rheumatic Diseases, Bone and British Journal of Dermatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.