G. Leitl

840 total citations
9 papers, 653 citations indexed

About

G. Leitl is a scholar working on Pediatrics, Perinatology and Child Health, Nutrition and Dietetics and Physiology. According to data from OpenAlex, G. Leitl has authored 9 papers receiving a total of 653 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Pediatrics, Perinatology and Child Health, 3 papers in Nutrition and Dietetics and 2 papers in Physiology. Recurrent topics in G. Leitl's work include Sodium Intake and Health (3 papers), Birth, Development, and Health (3 papers) and Nutrition, Genetics, and Disease (2 papers). G. Leitl is often cited by papers focused on Sodium Intake and Health (3 papers), Birth, Development, and Health (3 papers) and Nutrition, Genetics, and Disease (2 papers). G. Leitl collaborates with scholars based in United States. G. Leitl's co-authors include Lewis K. Dahl, M. Heine, Knud D. Knudsen, Junichi Iwai, J. Iwai, Keith H. Thompson, Robert W. Tuthill, Edward V. Ohanian and Lorraine Tassinari and has published in prestigious journals such as Nature, The Journal of Experimental Medicine and Circulation Research.

In The Last Decade

G. Leitl

9 papers receiving 557 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G. Leitl United States	9	237	186	149	121	99	9	653
Knud D. Knudsen United States	13	185 0.8×	261 1.4×	245 1.6×	153 1.3×	128 1.3×	16	742
T A Kotchen United States	13	122 0.5×	203 1.1×	161 1.1×	234 1.9×	88 0.9×	20	758
Lorraine Tassinari United States	10	357 1.5×	359 1.9×	363 2.4×	263 2.2×	206 2.1×	13	1.2k
R. Di Nicolantonio Australia	18	201 0.8×	321 1.7×	181 1.2×	168 1.4×	168 1.7×	65	884
Michiya Ohtaka Japan	10	95 0.4×	139 0.7×	54 0.4×	125 1.0×	84 0.8×	51	444
E. G. Schneider United States	18	68 0.3×	171 0.9×	205 1.4×	134 1.1×	336 3.4×	52	849
Saleh Zahedi Asl Iran	13	104 0.4×	60 0.3×	149 1.0×	166 1.4×	83 0.8×	23	570
K Kraft Germany	11	145 0.6×	147 0.8×	78 0.5×	71 0.6×	137 1.4×	30	529
R. A. Gelfand United States	9	105 0.4×	44 0.2×	233 1.6×	383 3.2×	183 1.8×	9	764
Alfred Kershbaum United States	9	67 0.3×	98 0.5×	45 0.3×	135 1.1×	67 0.7×	23	569

Countries citing papers authored by G. Leitl

Since Specialization

Citations

This map shows the geographic impact of G. Leitl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Leitl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Leitl more than expected).

Fields of papers citing papers by G. Leitl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Leitl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Leitl. The network helps show where G. Leitl may publish in the future.

Co-authorship network of co-authors of G. Leitl

This figure shows the co-authorship network connecting the top 25 collaborators of G. Leitl. A scholar is included among the top collaborators of G. Leitl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Leitl. G. Leitl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Ohanian, Edward V., J. Iwai, G. Leitl, & Robert W. Tuthill. (1978). Genetic influence on cadmium-induced hypertension. American Journal of Physiology-Heart and Circulatory Physiology. 235(4). H385–H391. 22 indexed citations

2.

Dahl, Lewis K., G. Leitl, & M. Heine. (1972). INFLUENCE OF DIETARY POTASSIUM AND SODIUM/POTASSIUM MOLAR RATIOS ON THE DEVELOPMENT OF SALT HYPERTENSION. The Journal of Experimental Medicine. 136(2). 318–330. 173 indexed citations

3.

Knudsen, Knud D., Lewis K. Dahl, Keith H. Thompson, et al.. (1970). EFFECTS OF CHRONIC EXCESS SALT INGESTION. The Journal of Experimental Medicine. 132(5). 976–1000. 68 indexed citations

4.

Dahl, Lewis K., M. Heine, G. Leitl, & Lorraine Tassinari. (1970). Hypertension and Death from Consumption of Processed Baby Foods by Rats. Experimental Biology and Medicine. 133(4). 1405–1408. 17 indexed citations

5.

Knudsen, Knud D., Junichi Iwai, M. Heine, G. Leitl, & Lewis K. Dahl. (1969). GENETIC INFLUENCE ON THE DEVELOPMENT OF RENOPRIVAL HYPERTENSION IN PARABIOTIC RATS. The Journal of Experimental Medicine. 130(6). 1353–1365. 34 indexed citations

6.

Iwai, J., Knud D. Knudsen, Lewis K. Dahl, M. Heine, & G. Leitl. (1969). GENETIC INFLUENCE ON THE DEVELOPMENT OF RENAL HYPERTENSION IN PARABIOTIC RATS. The Journal of Experimental Medicine. 129(3). 507–522. 43 indexed citations

7.

Dahl, Lewis K., Knud D. Knudsen, M. Heine, & G. Leitl. (1968). Hypertension and Stress. Nature. 219(5155). 735–736. 35 indexed citations

8.

Dahl, Lewis K., Knud D. Knudsen, M. Heine, & G. Leitl. (1968). Effects of Chronic Excess Salt Ingestion. Circulation Research. 22(1). 11–18. 175 indexed citations

9.

Dahl, Lewis K., Knud D. Knudsen, M. Heine, & G. Leitl. (1967). EFFECTS OF CHRONIC EXCESS SALT INGESTION. The Journal of Experimental Medicine. 126(4). 687–699. 86 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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