G Isenberg
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- Cardiac electrophysiology and arrhythmias 90
- Cardiomyopathy and Myosin Studies 8
- Cellular and Molecular Neuroscience top 0.2%
- Neuroscience and Neural Engineering 53
- Neuroscience and Neuropharmacology Research 29
- Sensory Systems top 0.5%
- Ion Channels and Receptors 5
- Molecular Biology top 0.5%
- Ion channel regulation and function 92
- Nicotinic Acetylcholine Receptors Study 7
- Physiology top 1%
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- Cardiac Ischemia and Reperfusion 9
G Isenberg
134 papers receiving 7.7k citations
Hit Papers
Peers
Comparison fields: 5 of 137
- Cardiology and Cardiovascular Medicine 4.9k
- Cellular and Molecular Neuroscience 3.8k
- Sensory Systems 698
- Molecular Biology 6.4k
- Physiology 279
Countries citing papers authored by G Isenberg
This map shows the geographic impact of G Isenberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G Isenberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G Isenberg more than expected).
Fields of papers citing papers by G Isenberg
This network shows the impact of papers produced by G Isenberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G Isenberg. The network helps show where G Isenberg may publish in the future.
Co-authorship network
The 25 scholars most cited alongside G Isenberg, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 92 | |
| 2 | 2008 | 28 | |
| 3 | 2005 | 6 | |
| 4 | 2003 | 78 | |
| 5 | 2003 | 65 | |
| 6 | Stretch-induced increments in [Na + ] i (mouse ventricular myocytes) | 2002 | 1 |
| 7 | 2002 | 10 | |
| 8 | 2001 | 14 | |
| 9 | 2001 | 106 | |
| 10 | 1998 | 29 | |
| 11 | 1994 | 101 | |
| 12 | 1993 | 96 | |
| 13 | 1992 | 51 | |
| 14 | 1991 | 21 | |
| 15 | 1990 | 22 | |
| 16 | Electrophysiology and contractile function | 1989 | 7 |
| 17 | 1989 | 40 | |
| 18 | 1988 | 3 | |
| 19 | 1977 | 33 | |
| 20 | [Voltage-clamp experiments on isolated skeletal muscle fibers using a technic combining microelectrodes with double sucrose gap]. | 1971 | 2 |
About G Isenberg
G Isenberg is a scholar working on Cellular and Molecular Neuroscience, Cardiology and Cardiovascular Medicine and Molecular Biology, having authored 135 papers that have together received 8.2k indexed citations. Recurring topics across this work include Ion channel regulation and function (92 papers), Cardiac electrophysiology and arrhythmias (90 papers), Neuroscience and Neural Engineering (53 papers), Neuroscience and Neuropharmacology Research (29 papers), Cardiac Ischemia and Reperfusion (9 papers), Cardiomyopathy and Myosin Studies (8 papers), Nicotinic Acetylcholine Receptors Study (7 papers) and Ion Channels and Receptors (5 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (4.9k citations), Cellular and Molecular Neuroscience (3.8k citations) and Sensory Systems (698 citations). G Isenberg has collaborated with scholars based in Germany, United States and Canada. Frequent co-authors include Udo Klöckner, Ryuji Inoue, Luiz Belardinelli, Vladimir Ganitkevich, L Belardinelli, M. F. Wendt-Gallitelli, Uwe Rueckschloss, Gerhard Dahl, W. Trautwein and Clive M. Baumgarten.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.