G D Ross

3.3k total citations · 1 hit paper
35 papers, 2.8k citations indexed

About

G D Ross is a scholar working on Immunology, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, G D Ross has authored 35 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Immunology, 8 papers in Molecular Biology and 6 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in G D Ross's work include Complement system in diseases (8 papers), Immune Cell Function and Interaction (6 papers) and Monoclonal and Polyclonal Antibodies Research (5 papers). G D Ross is often cited by papers focused on Complement system in diseases (8 papers), Immune Cell Function and Interaction (6 papers) and Monoclonal and Polyclonal Antibodies Research (5 papers). G D Ross collaborates with scholars based in United States, United Kingdom and Italy. G D Ross's co-authors include Václav Větvička, Benjamin P. Thornton, Michael C. Pitman, Rachel Goldman, Margaret J. Polley, R. J. Winchester, John D. Lambris, Enrique Rabellino, Robert Winchester and James Halper and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and The Journal of Experimental Medicine.

In The Last Decade

G D Ross

35 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Analysis of the sugar specificity and molecular location of the β-glucan-binding lectin site of complement receptor type 3 (CD11b/CD18)

1996 500 citations Benjamin P. Thornton, Václav Větvička et al. The Journal of Immunology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G D Ross United States	24	1.4k	595	540	413	376	35	2.8k
Gordon D. Ross United States	29	2.2k 1.6×	798 1.3×	659 1.2×	612 1.5×	648 1.7×	48	4.0k
Simon L. Newman United States	38	1.4k 1.0×	889 1.5×	328 0.6×	162 0.4×	447 1.2×	65	4.0k
Walid Mourad Canada	33	1.9k 1.4×	775 1.3×	270 0.5×	420 1.0×	49 0.1×	120	3.3k
Edina Schweighoffer United Kingdom	30	4.1k 3.0×	1.9k 3.2×	468 0.9×	309 0.7×	361 1.0×	43	6.4k
Jørgen Rygaard Denmark	38	1.2k 0.8×	2.1k 3.6×	228 0.4×	213 0.5×	117 0.3×	108	5.0k
Gersende Caron France	28	2.2k 1.6×	1.1k 1.8×	144 0.3×	119 0.3×	98 0.3×	70	4.3k
G L Asherson United Kingdom	38	2.8k 2.0×	508 0.9×	180 0.3×	420 1.0×	40 0.1×	128	4.3k
P. G. H. Gell United Kingdom	27	1.4k 1.0×	823 1.4×	252 0.5×	675 1.6×	87 0.2×	70	2.9k
H. U. Schorlemmer Germany	27	751 0.5×	608 1.0×	217 0.4×	165 0.4×	42 0.1×	110	2.1k
David G. Klapper United States	35	600 0.4×	1.5k 2.6×	108 0.2×	382 0.9×	119 0.3×	82	3.6k

Countries citing papers authored by G D Ross

Since Specialization

Citations

This map shows the geographic impact of G D Ross's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G D Ross with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G D Ross more than expected).

Fields of papers citing papers by G D Ross

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G D Ross. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G D Ross. The network helps show where G D Ross may publish in the future.

Co-authorship network of co-authors of G D Ross

This figure shows the co-authorship network connecting the top 25 collaborators of G D Ross. A scholar is included among the top collaborators of G D Ross based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G D Ross. G D Ross is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Haywood, S., Deborah M. Simpson, G D Ross, & Robert J. Beynon. (2005). The Greater Susceptibility of North Ronaldsay Sheep Compared with Cambridge Sheep to Copper-induced Oxidative Stress, Mitochondrial Damage and Hepatic Stellate Cell Activation. Journal of Comparative Pathology. 133(2-3). 114–127. 34 indexed citations

Ratajczak, Mariusz Z., Ryan Reca, Marcin Wysoczynski, et al.. (2004). Transplantation studies in C3-deficient animals reveal a novel role of the third complement component (C3) in engraftment of bone marrow cells. Leukemia. 18(9). 1482–1490. 84 indexed citations

Woldehiwet, Z., G D Ross, Ulrike G. Munderloh, et al.. (2002). Cultivation of an Ovine Strain of Ehrlichia phagocytophila in Tick Cell Cultures. Journal of Comparative Pathology. 127(2-3). 142–149. 38 indexed citations

Haywood, S., Thomas Müller, Wendt Müller, et al.. (2001). Copper-associated liver disease in North Ronaldsay sheep: a possible animal model for non-Wilsonian hepatic copper toxicosis of infancy and childhood. The Journal of Pathology. 195(2). 264–269. 37 indexed citations

GÖKÇE, Halil İ̇brahim, G D Ross, & Z. Woldehiwet. (1999). Inhibition of Phagosome-lysosome Fusion in Ovine Polymorphonuclear Leucocytes byEhrlichia (Cytoecetes) phagocytophila. Journal of Comparative Pathology. 120(4). 369–381. 26 indexed citations

Větvička, Václav, Benjamin P. Thornton, T. Jeffery Wieman, & G D Ross. (1997). Targeting of natural killer cells to mammary carcinoma via naturally occurring tumor cell-bound iC3b and beta-glucan-primed CR3 (CD11b/CD18). The Journal of Immunology. 159(2). 599–605. 67 indexed citations

Větvička, Václav, Benjamin P. Thornton, & G D Ross. (1996). Soluble beta-glucan polysaccharide binding to the lectin site of neutrophil or natural killer cell complement receptor type 3 (CD11b/CD18) generates a primed state of the receptor capable of mediating cytotoxicity of iC3b-opsonized target cells.. Journal of Clinical Investigation. 98(1). 50–61. 342 indexed citations

Carter, Stuart, et al.. (1996). Increased metabolism of collagen VI in canine osteoarthritis. Journal of Comparative Pathology. 114(3). 249–256. 8 indexed citations

Thornton, Benjamin P., Václav Větvička, Michael C. Pitman, Rachel Goldman, & G D Ross. (1996). Analysis of the sugar specificity and molecular location of the β-glucan-binding lectin site of complement receptor type 3 (CD11b/CD18). The Journal of Immunology. 156(3). 1235–1246. 500 indexed citations breakdown →

10.

Větvička, Václav, William Reed, Marie L. Hoover, & G D Ross. (1993). Complement factors H and I synthesized by B cell lines function to generate a growth factor activity from C3. The Journal of Immunology. 150(9). 4052–4060. 29 indexed citations

11.

Ross, G D. (1989). Complement and complement receptors. Current Opinion in Immunology. 2(1). 50–62. 31 indexed citations

12.

Buyon, Jill P., Steven B. Abramson, Mark R. Philips, et al.. (1988). Dissociation between increased surface expression of gp165/95 and homotypic neutrophil aggregation.. The Journal of Immunology. 140(9). 3156–3160. 126 indexed citations

13.

Stern, Fritz, Jane Jenson, & G D Ross. (1985). The View from inside: A French Communist Cell in Crisis. Foreign Affairs. 64(2). 372–372. 4 indexed citations

14.

Cox, David, John B. Dixon, P. Jenkins, et al.. (1984). Lymphocyte transformation as an aspect of immune recognition in echinococcosis. A review of some current experiments. Annals of Tropical Medicine and Parasitology. 78(3). 206–209. 5 indexed citations

15.

Lambris, John D., et al.. (1981). Isolation of lymphocyte membrane complement receptor type two (the C3d receptor) and preparation of receptor-specific antibody.. Proceedings of the National Academy of Sciences. 78(3). 1828–1832. 57 indexed citations

16.

Lambris, John D., et al.. (1980). Release of endogenous C3b inactivator from lymphocytes in response to triggering membrane receptors for beta 1H globulin.. The Journal of Experimental Medicine. 152(6). 1625–1644. 111 indexed citations

17.

Rabellino, Enrique, Ralph L. Nachman, N Williams, R. J. Winchester, & G D Ross. (1979). Human megakaryocytes. I. Characterization of the membrane and cytoplasmic components of isolated marrow megakaryocytes.. The Journal of Experimental Medicine. 149(6). 1273–1287. 137 indexed citations

18.

Ross, G D, et al.. (1978). The sequential appearance of Ia-like antigens and two different complement receptors during the maturation of human neutrophils.. The Journal of Experimental Medicine. 147(3). 730–744. 91 indexed citations

19.

Ross, G D & Margaret J. Polley. (1976). Assay for the Two Different Types of Lymphocyte Complement Receptors. Scandinavian Journal of Immunology. 5(s5). 99–111. 35 indexed citations

20.

Ross, G D & Margaret J. Polley. (1975). Specificity of human lymphocyte complement receptors.. The Journal of Experimental Medicine. 141(5). 1163–1180. 139 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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