G. A. Schuiling

1.6k total citations
96 papers, 1.2k citations indexed

About

G. A. Schuiling is a scholar working on Endocrinology, Diabetes and Metabolism, Reproductive Medicine and Immunology. According to data from OpenAlex, G. A. Schuiling has authored 96 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Endocrinology, Diabetes and Metabolism, 39 papers in Reproductive Medicine and 22 papers in Immunology. Recurrent topics in G. A. Schuiling's work include Growth Hormone and Insulin-like Growth Factors (35 papers), Ovarian function and disorders (25 papers) and Hypothalamic control of reproductive hormones (23 papers). G. A. Schuiling is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (35 papers), Ovarian function and disorders (25 papers) and Hypothalamic control of reproductive hormones (23 papers). G. A. Schuiling collaborates with scholars based in Netherlands, United Kingdom and Belgium. G. A. Schuiling's co-authors include Maria Faas, Julius F.W. Baller, Marijke M. Faas, Winston W. Bakker, C. Visscher, Annechien Bouman, Maas Jan Heineman, Loe de Leij, H. Moes and Ahmet Yanık and has published in prestigious journals such as Brain Research, American Journal of Obstetrics and Gynecology and Cellular and Molecular Life Sciences.

In The Last Decade

G. A. Schuiling

94 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
G. A. Schuiling Netherlands 17 444 437 334 329 226 96 1.2k
Elizabeth A. Lenton United Kingdom 22 412 0.9× 181 0.4× 274 0.8× 950 2.9× 637 2.8× 43 1.4k
Christopher R. Harlow United Kingdom 22 252 0.6× 221 0.5× 123 0.4× 802 2.4× 497 2.2× 39 1.4k
Shannon Whirledge United States 16 279 0.6× 120 0.3× 166 0.5× 324 1.0× 230 1.0× 34 1.2k
C.P. Puri India 20 407 0.9× 151 0.3× 65 0.2× 582 1.8× 297 1.3× 75 1.1k
Eng-Cheng Chan Australia 7 193 0.4× 124 0.3× 171 0.5× 81 0.2× 248 1.1× 9 959
J.E. Buster United States 15 135 0.3× 121 0.3× 387 1.2× 414 1.3× 585 2.6× 40 1.5k
B. J. Waddell Australia 16 130 0.3× 310 0.7× 596 1.8× 55 0.2× 93 0.4× 25 1.0k
S Phaneuf United Kingdom 20 262 0.6× 159 0.4× 171 0.5× 87 0.3× 233 1.0× 34 1.3k
Peter Burton Australia 13 78 0.2× 146 0.3× 400 1.2× 312 0.9× 222 1.0× 31 840
H. P. G. Schneider Germany 28 168 0.4× 167 0.4× 302 0.9× 854 2.6× 480 2.1× 95 2.2k

Countries citing papers authored by G. A. Schuiling

Since Specialization
Citations

This map shows the geographic impact of G. A. Schuiling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. A. Schuiling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. A. Schuiling more than expected).

Fields of papers citing papers by G. A. Schuiling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. A. Schuiling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. A. Schuiling. The network helps show where G. A. Schuiling may publish in the future.

Co-authorship network of co-authors of G. A. Schuiling

This figure shows the co-authorship network connecting the top 25 collaborators of G. A. Schuiling. A scholar is included among the top collaborators of G. A. Schuiling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. A. Schuiling. G. A. Schuiling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schuiling, G. A.. (2005). Reproduce and die! Why aging? Part I. Journal of Psychosomatic Obstetrics & Gynecology. 26(1). 73–77.
2.
Schuiling, G. A.. (2002). Sex, gender and reproduction. Journal of Psychosomatic Obstetrics & Gynecology. 23(2). 83–88. 1 indexed citations
3.
Faas, Marijke M. & G. A. Schuiling. (2001). Pre-eclampsia and the inflammatory response. European Journal of Obstetrics & Gynecology and Reproductive Biology. 95(2). 213–217. 28 indexed citations
4.
Schuiling, G. A.. (2001). Honor your father and your mother. Journal of Psychosomatic Obstetrics & Gynecology. 22(4). 215–219. 2 indexed citations
5.
Schuiling, G. A.. (2001). It's the gene, stupid!. Journal of Psychosomatic Obstetrics & Gynecology. 22(3). 177–180. 3 indexed citations
6.
Schuiling, G. A.. (2001). Early menopause?. Journal of Psychosomatic Obstetrics & Gynecology. 22(2). 123–126. 1 indexed citations
7.
Faas, Marijke M., et al.. (2000). The immune response during the luteal phase of the ovarian cycle: a Th2-type response?. Fertility and Sterility. 74(5). 1008–1013. 149 indexed citations
8.
Faas, Maria, et al.. (2000). Activation of peripheral leukocytes in rat pregnancy and experimental preeclampsia. American Journal of Obstetrics and Gynecology. 182(2). 351–357. 53 indexed citations
9.
Schuiling, G. A., Marijke M. Faas, G.A. Dekker, & B. M. Sibai. (1999). Etiology and pathogenesis of preeclampsia: Current concepts [2] (multiple letters). American Journal of Obstetrics and Gynecology. 181(4). 1036–1037. 2 indexed citations
10.
Schuiling, G. A., et al.. (1999). Effect of Estradiol and Progesterone on the Low‐Dose Endotoxin‐Induced Glomerular Inflammatory Response of the Female Rat. American Journal of Reproductive Immunology. 41(3). 224–231. 19 indexed citations
11.
Faas, Maria, Winston W. Bakker, Julius F.W. Baller, & G. A. Schuiling. (1999). Pregnancy enhances the sensitivity of glomerular ecto–adenosine triphosphate–diphosphohydrolase to products of activated polymorphonuclear leukocytes. American Journal of Obstetrics and Gynecology. 180(1). 112–113. 9 indexed citations
12.
Faas, Maria, et al.. (1999). Pregnancy aggravates proteinuria in subclinical glomerulonephritis in the rat. Journal of Laboratory and Clinical Medicine. 134(3). 267–274. 4 indexed citations
13.
Nieuwenhuizen, Arie G., et al.. (1998). Effects of Food Restriction on Glucose Tolerance, Insulin Secretion, and Islet-Cell Proliferation in Pregnant Rats. Physiology & Behavior. 65(4-5). 671–677. 7 indexed citations
14.
15.
Faas, Maria, G. A. Schuiling, Julius F.W. Baller, C. Visscher, & Winston W. Bakker. (1994). A new animal model for human preeclampsia: Ultra-lowdose endotoxin infusion in pregnant rats. American Journal of Obstetrics and Gynecology. 171(1). 158–164. 243 indexed citations
16.
17.
Visscher, C., et al.. (1993). Reproductive Condition, Glomerular Adenosine Diphosphatase Activity, and Platelet Aggregation in the Rat: Effect of Endotoxin1. Biology of Reproduction. 49(6). 1303–1309. 8 indexed citations
18.
Moes, H., et al.. (1988). Commercially available analogues of GnRH and LH secretion. European Journal of Endocrinology. 119(3). 345–352. 5 indexed citations
19.
Schuiling, G. A., et al.. (1985). REGULATION OF THE RELEASE OF LH AND FSH - PHYSIOLOGICAL AND PHARMACOLOGICAL ASPECTS. Frontiers of hormone research. 14. 34–52. 2 indexed citations
20.
Schuiling, G. A., et al.. (1985). Progesterone and the Control of Functional Luteolysis, of Secretion of Prolactin and of Pituitary LHRH Responsiveness. Neuroendocrinology. 41(1). 52–59. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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