Fumitoshi Onoda

881 total citations
24 papers, 749 citations indexed

About

Fumitoshi Onoda is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Fumitoshi Onoda has authored 24 papers receiving a total of 749 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 4 papers in Cancer Research and 3 papers in Genetics. Recurrent topics in Fumitoshi Onoda's work include DNA Repair Mechanisms (17 papers), Fungal and yeast genetics research (8 papers) and Genomics and Chromatin Dynamics (7 papers). Fumitoshi Onoda is often cited by papers focused on DNA Repair Mechanisms (17 papers), Fungal and yeast genetics research (8 papers) and Genomics and Chromatin Dynamics (7 papers). Fumitoshi Onoda collaborates with scholars based in Japan and United States. Fumitoshi Onoda's co-authors include Masayuki Seki, Takemi Enomoto, Atsuko Miyajima, Toshiki Enomoto, Ayako Ui, Dana Branzei, Yasufumi Murakami, Minoru Seki, Yoh-ichi Kawabe and Takahiko Utsugi and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Fumitoshi Onoda

24 papers receiving 739 citations

Peers

Countries citing papers authored by Fumitoshi Onoda

Since Specialization

Citations

This map shows the geographic impact of Fumitoshi Onoda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumitoshi Onoda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumitoshi Onoda more than expected).

Fields of papers citing papers by Fumitoshi Onoda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fumitoshi Onoda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumitoshi Onoda. The network helps show where Fumitoshi Onoda may publish in the future.

Co-authorship network of co-authors of Fumitoshi Onoda

This figure shows the co-authorship network connecting the top 25 collaborators of Fumitoshi Onoda. A scholar is included among the top collaborators of Fumitoshi Onoda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fumitoshi Onoda. Fumitoshi Onoda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Work	Indexed citations
1	Interaction of Hand2 and E2a is important for transcription of Phox2b in sympathetic nervous system neuron differentiation 2011 ·Biochemical and Biophysical Research Communications ·Yusuke Hashimoto, (unknown), (unknown), (unknown), Fumitoshi Onoda, Fumio Tashiro, (unknown), Yasufumi Murakami	6
2	Genetic analyses using a mouse cell cycle mutant identifies magoh as a novel gene involved in Cdk regulation 2011 ·Genes to Cells ·(unknown), (unknown), Takahiko Utsugi, Fumitoshi Onoda, Fumio Hanaoka, Yasufumi Murakami	14
3	Nuclear β-catenin and CD44 upregulation characterize invasive cell populations in non-aggressive MCF-7 breast cancer cells 2010 ·BMC Cancer ·(unknown), Hiroko Kojima, Kenta Wada, (unknown), Fumitoshi Onoda, (unknown), Takahiko Utsugi, Yasufumi Murakami	88
4	The mammalian INO80 complex is recruited to DNA damage sites in an ARP8 dependent manner 2010 ·Biochemical and Biophysical Research Communications ·(unknown), (unknown), Takumi Watanabe, Fumitoshi Onoda, (unknown), Yasufumi Murakami	48
5	Development of effective isolation method of ES cells for analysis of differentiation 2009 ·Biochemical and Biophysical Research Communications ·(unknown), (unknown), Fumitoshi Onoda, Yasufumi Murakami	1
6	The bHLH transcription factor Hand2 regulates the expression of nanog in ANS differentiation 2009 ·Biochemical and Biophysical Research Communications ·Yusuke Hashimoto, (unknown), (unknown), (unknown), Fumio Tashiro, Fumitoshi Onoda, Yasufumi Murakami	3
7	Novel DNA Microarray System for Analysis of Nascent mRNAs 2008 ·DNA Research ·Masayasu Ohtsu, (unknown), M. Fukuoka, (unknown), Fumio Hanaoka, Toshihiro Utsugi, Fumitoshi Onoda, Yasufumi Murakami	14
8	Actin-related protein Arp4 functions in kinetochore assembly 2007 ·Nucleic Acids Research ·Hideaki Ogiwara, Ayako Ui, Satoshi Kawashima, Kazuto Kugou, Fumitoshi Onoda, Hitoshi Iwahashi, Masahiko Harata, Kunihiro Ohta, Takemi Enomoto, Masayuki Seki	27
9	Ubc9 is required for damage-tolerance and damage-induced interchromosomal homologous recombination in S. cerevisiae 2004 ·DNA repair ·Daisuke Maeda, Masayuki Seki, Fumitoshi Onoda, Dana Branzei, Yoh-ichi Kawabe, Takemi Enomoto	28
10	SMC6 is required for MMS-induced interchromosomal and sister chromatid recombinations in Saccharomyces cerevisiae 2004 ·DNA repair ·Fumitoshi Onoda, Masahiro Takeda, Masayuki Seki, Daisuke Maeda, (unknown), Ayako Ui, Hideki Yagi, Takemi Enomoto	45
11	The hyper unequal sister chromatid recombination in an sgs1 mutant of budding yeast requires MSH2 2004 ·DNA repair ·Fumitoshi Onoda, Masayuki Seki, Wensheng Wang, Takemi Enomoto	11
12	The ability of Sgs1 to interact with DNA topoisomerase III is essential for damage-induced recombination 2004 ·DNA repair ·Ayako Ui, Masayuki Seki, Hideaki Ogiwara, (unknown), S. Fukushige, Fumitoshi Onoda, Takemi Enomoto	39
13	Budding yeast mms4 is epistatic with rad52 and the function of Mms4 can be replaced by a bacterial Holliday junction resolvase 2003 ·DNA repair ·(unknown), Masayuki Seki, Fumitoshi Onoda, Akari Yoshimura, (unknown), Takemi Enomoto	20
14	Functional and physical interaction between Sgs1 and Top3 and Sgs1-independent function of Top3 in DNA recombination repair. 2002 ·Genes & Genetic Systems ·(unknown), Masayuki Seki, Ayako Ui, Yurie Satoh, Atsuko Miyajima, Fumitoshi Onoda, Takemi Enomoto	24
15	The product of Saccharomyces cerevisiae WHIP/MGS1, a gene related to replication factor C genes, interacts functionally with DNA polymerase δ 2002 ·Molecular Genetics and Genomics ·Dana Branzei, Minoru Seki, Fumitoshi Onoda, Toshiki Enomoto	51
16	A Novel Protein Interacts with the Werner's Syndrome Gene Product Physically and Functionally 2001 ·Journal of Biological Chemistry ·Yoh-ichi Kawabe, Dana Branzei, Tomoko Hayashi, Hirobumi Suzuki, Takashi Masuko, Fumitoshi Onoda, Seok‐Jin Heo, Hideo Ikeda, Akira Shimamoto, Yasuhiro Furuichi, Masayuki Seki, Takemi Enomoto	56
17	Involvement of SGS1 in DNA damage-induced heteroallelic recombination that requires RAD52 in Saccharomyces cerevisiae 2001 ·Molecular and General Genetics MGG ·Fumitoshi Onoda, Masayuki Seki, Atsuko Miyajima, Toshiki Enomoto	53
18	The N-terminal region of Sgs1, which interacts with Top3, is required for complementation of MMS sensitivity and suppression of hyper-recombination in sgs1 disruptants 2001 ·Molecular Genetics and Genomics ·Ayako Ui, Y. Satoh, Fumitoshi Onoda, Atsuko Miyajima, Minoru Seki, Toshiki Enomoto	52
19	Sgs1 Helicase Activity Is Required for Mitotic but Apparently Not for Meiotic Functions 2000 ·Molecular and Cellular Biology ·Atsuko Miyajima, Masayuki Seki, Fumitoshi Onoda, Miwa Shiratori, (unknown), Kunihiro Ohta, Yoshiko Kikuchi, Yasuo Ohno, Takemi Enomoto	41
20	Elevation of sister chromatid exchange in Saccharomyces cerevisiae sgs1 disruptants and the relevance of the disruptants as a system to evaluate mutations in Bloom's syndrome gene 2000 ·Mutation Research/DNA Repair ·Fumitoshi Onoda, Masayuki Seki, Atsuko Miyajima, Takemi Enomoto	74

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact