Fumiko Sekiguchi

6.1k total citations
174 papers, 5.1k citations indexed

About

Fumiko Sekiguchi is a scholar working on Physiology, Molecular Biology and Biochemistry. According to data from OpenAlex, Fumiko Sekiguchi has authored 174 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Physiology, 51 papers in Molecular Biology and 37 papers in Biochemistry. Recurrent topics in Fumiko Sekiguchi's work include Nitric Oxide and Endothelin Effects (29 papers), Pain Mechanisms and Treatments (25 papers) and Ion channel regulation and function (23 papers). Fumiko Sekiguchi is often cited by papers focused on Nitric Oxide and Endothelin Effects (29 papers), Pain Mechanisms and Treatments (25 papers) and Ion channel regulation and function (23 papers). Fumiko Sekiguchi collaborates with scholars based in Japan, United States and China. Fumiko Sekiguchi's co-authors include Atsufumi Kawabata, Hideo Ishitsuka, Noriaki Sawada, Maho Tsubota, Yutaka Tanaka, Hiroyuki Nishikawa, Tohru Ishikawa, Yu Fukase, Satoru Sunano and S. Yoshida and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Gastroenterology and Gut.

In The Last Decade

Fumiko Sekiguchi

173 papers receiving 5.0k citations

Peers

Fumiko Sekiguchi
Richard M. Mortensen United States
Erding Hu United States
Joseph Satriano United States
S Bunting United States
Fumiko Sekiguchi
Citations per year, relative to Fumiko Sekiguchi Fumiko Sekiguchi (= 1×) peers Jean Sébastien Saulnier‐Blache

Countries citing papers authored by Fumiko Sekiguchi

Since Specialization
Citations

This map shows the geographic impact of Fumiko Sekiguchi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumiko Sekiguchi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumiko Sekiguchi more than expected).

Fields of papers citing papers by Fumiko Sekiguchi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fumiko Sekiguchi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumiko Sekiguchi. The network helps show where Fumiko Sekiguchi may publish in the future.

Co-authorship network of co-authors of Fumiko Sekiguchi

This figure shows the co-authorship network connecting the top 25 collaborators of Fumiko Sekiguchi. A scholar is included among the top collaborators of Fumiko Sekiguchi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fumiko Sekiguchi. Fumiko Sekiguchi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tsubota, Maho, Kana Sasaki, Eun-Kyung Shin, et al.. (2025). HMGB1 derived from macrophages and enteric glial cells contributes to the butyrate-induced colonic hypersensitivity in mice. European Journal of Pharmacology. 999. 177660–177660. 1 indexed citations
2.
Sekiguchi, Fumiko, Maho Tsubota, & Atsufumi Kawabata. (2024). Sulfide and polysulfide as pronociceptive mediators: Focus on Cav3.2 function enhancement and TRPA1 activation. Journal of Pharmacological Sciences. 155(3). 113–120. 2 indexed citations
3.
Sekiguchi, Fumiko, et al.. (2023). Cav3.2-dependent hyperalgesia/allodynia following intrathecal and intraplantar zinc chelator administration in rodents. Journal of Pharmacological Sciences. 152(2). 86–89. 3 indexed citations
4.
Sekiguchi, Fumiko, et al.. (2022). Development of hepatic impairment aggravates chemotherapy-induced peripheral neuropathy following oxaliplatin treatment: Evidence from clinical and preclinical studies. Journal of Pharmacological Sciences. 148(3). 315–325. 3 indexed citations
5.
Sekiguchi, Fumiko, Yasuhiro Shimada, Hiroshi Masuda, et al.. (2022). A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Cav3.2-dependent pain by sequestering exogenous and endogenous sulfide. Redox Biology. 59. 102579–102579. 10 indexed citations
6.
Tsubota, Maho, Yusuke Hayashi, Fumiko Sekiguchi, et al.. (2021). Caspase-Dependent HMGB1 Release from Macrophages Participates in Peripheral Neuropathy Caused by Bortezomib, a Proteasome-Inhibiting Chemotherapeutic Agent, in Mice. Cells. 10(10). 2550–2550. 9 indexed citations
7.
Sekiguchi, Fumiko, et al.. (2021). Macrophage as a Peripheral Pain Regulator. Cells. 10(8). 1881–1881. 96 indexed citations
8.
Tsujita, Ryuichi, Maho Tsubota, Fumiko Sekiguchi, & Atsufumi Kawabata. (2020). Role of high‐mobility group box 1 and its modulation by thrombomodulin/thrombin axis in neuropathic and inflammatory pain. British Journal of Pharmacology. 178(4). 798–812. 28 indexed citations
9.
Sekiguchi, Fumiko & Atsufumi Kawabata. (2020). Role of HMGB1 in Chemotherapy-Induced Peripheral Neuropathy. International Journal of Molecular Sciences. 22(1). 367–367. 21 indexed citations
10.
Tsubota, Maho, et al.. (2019). Dietary ascorbic acid restriction in GNL/SMP30-knockout mice unveils the role of ascorbic acid in regulation of somatic and visceral pain sensitivity. Biochemical and Biophysical Research Communications. 511(3). 705–710. 3 indexed citations
11.
Tsubota, Maho, Yusuke Hayashi, Fumiko Sekiguchi, et al.. (2019). Role of non-macrophage cell-derived HMGB1 in oxaliplatin-induced peripheral neuropathy and its prevention by the thrombin/thrombomodulin system in rodents: negative impact of anticoagulants. Journal of Neuroinflammation. 16(1). 199–199. 39 indexed citations
12.
Sekiguchi, Takeshi, Nobuaki Furuno, Takashi Ishii, et al.. (2018). RagA, an mTORC1 activator, interacts with a hedgehog signaling protein, WDR35/IFT121. Genes to Cells. 24(2). 151–161. 6 indexed citations
13.
Okada, Takuya, Fumiko Sekiguchi, Maho Tsubota, et al.. (2018). Design and synthesis of novel anti-hyperalgesic agents based on 6-prenylnaringenin as the T-type calcium channel blockers. Bioorganic & Medicinal Chemistry. 26(15). 4410–4427. 16 indexed citations
14.
Sekiguchi, Fumiko, Maho Tsubota, S. Yoshida, et al.. (2018). Critical role of Cav3.2 T-type calcium channels in the peripheral neuropathy induced by bortezomib, a proteasome-inhibiting chemotherapeutic agent, in mice. Toxicology. 413. 33–39. 43 indexed citations
15.
16.
Tsujita, Ryuichi, et al.. (2017). Role of Thrombin in Soluble Thrombomodulin-Induced Suppression of Peripheral HMGB1-Mediated Allodynia in Mice. Journal of Neuroimmune Pharmacology. 13(2). 179–188. 16 indexed citations
17.
Tsubota, Maho, et al.. (2017). Involvement of NF‐κB in the upregulation of cystathionine‐γ‐lyase, a hydrogen sulfide‐forming enzyme, and bladder pain accompanying cystitis in mice. Clinical and Experimental Pharmacology and Physiology. 45(4). 355–361. 12 indexed citations
18.
Hayashi, Yusuke, Ryuichi Tsujita, Maho Tsubota, et al.. (2017). Human soluble thrombomodulin-induced blockade of peripheral HMGB1-dependent allodynia in mice requires both the lectin-like and EGF-like domains. Biochemical and Biophysical Research Communications. 495(1). 634–638. 14 indexed citations
19.
Nishida, Takeshi, Maho Tsubota, Fumiko Sekiguchi, et al.. (2016). Involvement of high mobility group box 1 in the development and maintenance of chemotherapy-induced peripheral neuropathy in rats. Toxicology. 365. 48–58. 43 indexed citations
20.
Yoneda, Satoshi, Makoto Kadowaki, Fumiko Sekiguchi, et al.. (2001). Rhythmic Spontaneous Contractions in the Rat Proximal Colon.. The Japanese Journal of Physiology. 51(6). 717–723. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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