Fujio Okamoto

1.4k total citations
28 papers, 1.2k citations indexed

About

Fujio Okamoto is a scholar working on Molecular Biology, Nutrition and Dietetics and Oncology. According to data from OpenAlex, Fujio Okamoto has authored 28 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 8 papers in Nutrition and Dietetics and 7 papers in Oncology. Recurrent topics in Fujio Okamoto's work include Bone Metabolism and Diseases (14 papers), Ion channel regulation and function (10 papers) and Bone health and treatments (6 papers). Fujio Okamoto is often cited by papers focused on Bone Metabolism and Diseases (14 papers), Ion channel regulation and function (10 papers) and Bone health and treatments (6 papers). Fujio Okamoto collaborates with scholars based in Japan, United States and Spain. Fujio Okamoto's co-authors include Koji Okabe, Hiroshi Kajiya, Hidefumi Fukushima, Eijiro Jimi, Kazuhiro Aoki, Hiroaki Saito, Keiichi Ohya, Michael J. May, Fulvio D’Acquisto and Ichiro Nakamura and has published in prestigious journals such as Nature Medicine, Molecular and Cellular Biology and Scientific Reports.

In The Last Decade

Fujio Okamoto

27 papers receiving 1.2k citations

Peers

Fujio Okamoto

ONCOL

RHEUM

IMMUN

Takashi Iezaki Japan

Mi Yang China

Weiwen Zhu China

Karina G. Zecchin Brazil

L V Avioli United States

A.V. Neutzsky-Wulff Denmark

Qiang Jie China

T. Sunyer United States

Yukiko Kitase United States

T.J. Martin United States

Takashi Iezaki Japan

Fujio Okamoto

728 ×0.8

219 ×0.7

ONCOL

221 ×0.8

136 ×0.9

RHEUM

64 ×0.5

IMMUN

Citations per year, relative to Fujio Okamoto Fujio Okamoto (= 1×) peers Takashi Iezaki

Countries citing papers authored by Fujio Okamoto

Since Specialization

Citations

This map shows the geographic impact of Fujio Okamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fujio Okamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fujio Okamoto more than expected).

Fields of papers citing papers by Fujio Okamoto

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fujio Okamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fujio Okamoto. The network helps show where Fujio Okamoto may publish in the future.

Co-authorship network of co-authors of Fujio Okamoto

This figure shows the co-authorship network connecting the top 25 collaborators of Fujio Okamoto. A scholar is included among the top collaborators of Fujio Okamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fujio Okamoto. Fujio Okamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Shin, Masashi, Toshihide Mizoguchi, Atsushi Arai, et al.. (2022). Mesenchymal cell TRPM7 expression is required for bone formation via the regulation of chondrogenesis. Bone. 166. 116579–116579. 9 indexed citations

Kajiya, Hiroshi, et al.. (2018). Toll-like receptor 2 activation primes and upregulates osteoclastogenesis via lox-1. Lipids in Health and Disease. 17(1). 132–132. 25 indexed citations

Oka, Kyoko, Masashi Shin, Fujio Okamoto, et al.. (2017). The crucial role of the TRPM7 kinase domain in the early stage of amelogenesis. Scientific Reports. 7(1). 18099–18099. 20 indexed citations

Kajiya, Hiroshi, et al.. (2013). A novel inhibitory mechanism of nitrogen-containing bisphosphonate on the activity of Cl− extrusion in osteoclasts. Naunyn-Schmiedeberg s Archives of Pharmacology. 386(7). 589–598. 7 indexed citations

Okabe, Koji, Fujio Okamoto, & Hiroshi Kajiya. (2012). [Odontoclasts and calcitonin].. PubMed. 22(1). 19–26. 3 indexed citations

Okamoto, Fujio, et al.. (2010). Antibodies against ClC7 inhibit extracellular acidification-induced Cl− currents and bone resorption activity in mouse osteoclasts. Naunyn-Schmiedeberg s Archives of Pharmacology. 383(1). 79–90. 14 indexed citations

Kajiya, Hiroshi, et al.. (2010). RANKL-induced TRPV2 expression regulates osteoclastogenesis via calcium oscillations. Cell Calcium. 48(5). 260–269. 61 indexed citations

Kajiya, Hiroshi, et al.. (2009). Characteristics of ClC7 Cl− channels and their inhibition in mutant (G215R) associated with autosomal dominant osteopetrosis type II in native osteoclasts and hClcn7 gene-expressing cells. Pflügers Archiv - European Journal of Physiology. 458(6). 1049–1059. 24 indexed citations

Okamoto, Fujio, Hiroshi Kajiya, Kazuko Toh, et al.. (2008). Intracellular ClC-3 chloride channels promote bone resorption in vitro through organelle acidification in mouse osteoclasts. American Journal of Physiology-Cell Physiology. 294(3). C693–C701. 42 indexed citations

10.

Li, Jingping, Hiroshi Kajiya, Fujio Okamoto, et al.. (2007). Three Na⁺/Ca²⁺Exchanger (NCX) Variants Are Expressed in Mouse Osteoclasts and Mediate Calcium Transport during Bone Resorption. Endocrinology. 148(5). 2116–2125. 34 indexed citations

11.

Fukushima, Hidefumi, et al.. (2005). IL-1-induced receptor activator of NF-κB ligand in human periodontal ligament cells involves ERK-dependent PGE2 production. Bone. 36(2). 267–275. 68 indexed citations

12.

Fukushima, Hidefumi, et al.. (2004). Calcitonin in human odontoclasts regulates root resorption activity via protein kinase A. Journal of Bone and Mineral Metabolism. 22(1). 12–18. 14 indexed citations

13.

Okamoto, Fujio, Hiroshi Kajiya, Hidefumi Fukushima, Eijiro Jimi, & Koji Okabe. (2004). Prostaglandin E₂ activates outwardly rectifying Cl⁻ channels via a cAMP-dependent pathway and reduces cell motility in rat osteoclasts. American Journal of Physiology-Cell Physiology. 287(1). C114–C124. 13 indexed citations

14.

Fukushima, Hidefumi, Hiroshi Kajiya, Keisuke Takada, Fujio Okamoto, & Koji Okabe. (2003). Expression and role of RANKL in periodontal ligament cells during physiological root‐resorption in human deciduous teeth. European Journal Of Oral Sciences. 111(4). 346–352. 107 indexed citations

15.

Okamoto, Fujio, et al.. (2003). Amino Acid-Permeable Anion Channels in Early Mouse Embryos and Their Possible Effects on Cleavage1. Biology of Reproduction. 68(3). 947–953. 10 indexed citations

16.

Kajiya, Hiroshi, Fujio Okamoto, Hidefumi Fukushima, & Koji Okabe. (2003). Calcitonin inhibits proton extrusion in resorbing rat osteoclasts via protein kinase A. Pflügers Archiv - European Journal of Physiology. 445(6). 651–658. 17 indexed citations

17.

Okabe, Koji, Fujio Okamoto, Hiroshi Kajiya, Keisuke Takada, & Hiroyuki Soeda. (2000). Estrogen directly acts on osteoclasts via inhibition of inward rectifier K + channels. Naunyn-Schmiedeberg s Archives of Pharmacology. 361(6). 610–620. 16 indexed citations

18.

Kajiya, Hiroshi, et al.. (2000). Protein tyrosine kinase inhibitors increase cytosolic calcium and inhibit actin organization as resorbing activity in rat osteoclasts. Journal of Cellular Physiology. 183(1). 83–90. 40 indexed citations

19.

Okabe, Koji, Yuichi Inoue, Takeshi Kawarabayashi, et al.. (1999). Physiological significance of hyperpolarization-activated inward currents (I h) in smooth muscle cells from the circular layers of pregnant rat myometrium. Pflügers Archiv - European Journal of Physiology. 439(1). 76–85. 17 indexed citations

20.

Okamoto, Fujio. (1985). Inorganic crystal structure database and its application.. Journal of Information Processing and Management. 28(1). 50–58. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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