Frédéric Marteau

566 total citations
8 papers, 458 citations indexed

About

Frédéric Marteau is a scholar working on Physiology, Immunology and Molecular Biology. According to data from OpenAlex, Frédéric Marteau has authored 8 papers receiving a total of 458 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Physiology, 5 papers in Immunology and 3 papers in Molecular Biology. Recurrent topics in Frédéric Marteau's work include Adenosine and Purinergic Signaling (8 papers), Immune Cell Function and Interaction (4 papers) and Immunotherapy and Immune Responses (2 papers). Frédéric Marteau is often cited by papers focused on Adenosine and Purinergic Signaling (8 papers), Immune Cell Function and Interaction (4 papers) and Immunotherapy and Immune Responses (2 papers). Frédéric Marteau collaborates with scholars based in Belgium, South Korea and France. Frédéric Marteau's co-authors include Jean‐Marie Boeynaems, Didier Communi, Nathalie Suarez Gonzalez, David Communi, Pierre Savi, Emmanuel Le Poul, Liaman Mamedova, Michel Goldman, Katrin Sak and Jung‐Sun Lee and has published in prestigious journals such as Blood, The Journal of Immunology and FEBS Letters.

In The Last Decade

Frédéric Marteau

8 papers receiving 449 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Frédéric Marteau Belgium	7	356	184	105	64	63	8	458
Heiko Ullmann Germany	8	331 0.9×	186 1.0×	42 0.4×	67 1.0×	68 1.1×	8	470
Jean Philippe Breittmayer France	15	174 0.5×	228 1.2×	54 0.5×	80 1.3×	84 1.3×	26	518
Liaman K. Mamedova Israel	9	231 0.6×	194 1.1×	22 0.2×	49 0.8×	53 0.8×	12	388
Maria Giulia Callegari Italy	7	608 1.7×	159 0.9×	123 1.2×	23 0.4×	41 0.7×	7	723
Petra Heine Germany	7	519 1.5×	179 1.0×	35 0.3×	76 1.2×	10 0.2×	8	604
H H Oei Switzerland	5	262 0.7×	206 1.1×	54 0.5×	81 1.3×	50 0.8×	7	444
Nicholas White United Kingdom	5	293 0.8×	122 0.7×	49 0.5×	17 0.3×	9 0.1×	6	382
Valeria Iannotta Italy	9	295 0.8×	202 1.1×	30 0.3×	46 0.7×	12 0.2×	9	451
Bettina Barthel Germany	9	127 0.4×	173 0.9×	21 0.2×	63 1.0×	12 0.2×	16	429
José Bernardo Noronha‐Matos Portugal	12	152 0.4×	142 0.8×	17 0.2×	63 1.0×	17 0.3×	20	356

Countries citing papers authored by Frédéric Marteau

Since Specialization

Citations

This map shows the geographic impact of Frédéric Marteau's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frédéric Marteau with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frédéric Marteau more than expected).

Fields of papers citing papers by Frédéric Marteau

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frédéric Marteau. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frédéric Marteau. The network helps show where Frédéric Marteau may publish in the future.

Co-authorship network of co-authors of Frédéric Marteau

This figure shows the co-authorship network connecting the top 25 collaborators of Frédéric Marteau. A scholar is included among the top collaborators of Frédéric Marteau based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frédéric Marteau. Frédéric Marteau is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Horckmans, Michael, Anne Lefort, F. Libert, et al.. (2007). Gene Expression Profiling Defines ATP as a Key Regulator of Human Dendritic Cell Functions. The Journal of Immunology. 179(6). 3550–3558. 16 indexed citations

2.

Horckmans, Michael, Brice Marcet, Frédéric Marteau, et al.. (2006). Extracellular adenine nucleotides inhibit the release of major monocyte recruiters by human monocyte‐derived dendritic cells. FEBS Letters. 580(3). 747–754. 21 indexed citations

3.

Kim, Yong‐Chul, Jung‐Sun Lee, Katrin Sak, et al.. (2005). Synthesis of pyridoxal phosphate derivatives with antagonist activity at the P2Y13 receptor. Biochemical Pharmacology. 70(2). 266–274. 81 indexed citations

4.

Communi, Didier, et al.. (2005). Nucleotide receptor P2y13. 4 indexed citations

5.

Marteau, Frédéric, Nathalie Suarez Gonzalez, David Communi, et al.. (2005). Thrombospondin-1 and indoleamine 2,3-dioxygenase are major targets of extracellular ATP in human dendritic cells. Blood. 106(12). 3860–3866. 74 indexed citations

6.

Marteau, Frédéric, Didier Communi, Jean‐Marie Boeynaems, & Nathalie Suarez Gonzalez. (2004). Involvement of multiple P2Y receptors and signaling pathways in the action of adenine nucleotides diphosphates on human monocyte-derived dendritic cells. Journal of Leukocyte Biology. 76(4). 796–803. 66 indexed citations

7.

Marteau, Frédéric, Emmanuel Le Poul, David Communi, et al.. (2003). Pharmacological Characterization of the Human P2Y13 Receptor. Molecular Pharmacology. 64(1). 104–112. 184 indexed citations

8.

Boeynaems, Jean‐Marie, Françoise Wilkin, Frédéric Marteau, et al.. (2003). P2Y receptors: New subtypes, new functions. Drug Development Research. 59(1). 30–35. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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