Franco Fais

6.8k total citations · 2 hit papers
71 papers, 4.8k citations indexed

About

Franco Fais is a scholar working on Genetics, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, Franco Fais has authored 71 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Genetics, 40 papers in Immunology and 19 papers in Pathology and Forensic Medicine. Recurrent topics in Franco Fais's work include Chronic Lymphocytic Leukemia Research (38 papers), Immunodeficiency and Autoimmune Disorders (22 papers) and Lymphoma Diagnosis and Treatment (18 papers). Franco Fais is often cited by papers focused on Chronic Lymphocytic Leukemia Research (38 papers), Immunodeficiency and Autoimmune Disorders (22 papers) and Lymphoma Diagnosis and Treatment (18 papers). Franco Fais collaborates with scholars based in Italy, United States and United Kingdom. Franco Fais's co-authors include Manlio Ferrarini, Fabio Ghiotto, Nicholas Chiorazzi, Steven L. Allen, R. Kanti, Angelo Valetto, Philip Schulman, Jonathan E. Kolitz, Rajendra N. Damle and Stuart M. Lichtman and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and Blood.

In The Last Decade

Franco Fais

69 papers receiving 4.7k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Ig V Gene Mutation Status and CD38 Expression As Novel Prognostic Indicators in Chronic Lymphocytic Leukemia

1999 1.9k citations Franco Fais, Fabio Ghiotto et al. profile →
Chronic lymphocytic leukemia B cells express restricted sets of mutated and unmutated antigen receptors.

1998 690 citations Franco Fais, Fabio Ghiotto et al. Journal of Clinical Investigation profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Franco Fais	3.5k	2.5k	2.4k	977	768	71	4.8k
Valter Gattei	2.5k 0.7×	2.4k 0.9×	2.1k 0.9×	1.7k 1.7×	1.4k 1.9×	222	5.7k
Ricardo Morilla	2.5k 0.7×	2.2k 0.9×	1.8k 0.8×	764 0.8×	940 1.2×	93	4.5k
Zachary R. Hunter	5.1k 1.4×	4.3k 1.7×	2.5k 1.0×	901 0.9×	902 1.2×	231	6.2k
Cristina Scielzo	1.7k 0.5×	1.1k 0.4×	1.5k 0.6×	783 0.8×	558 0.7×	56	2.8k
Frédéric Davi	1.7k 0.5×	2.2k 0.9×	1.2k 0.5×	749 0.8×	1.6k 2.1×	128	4.2k
Anne J. Novak	1.1k 0.3×	2.1k 0.8×	3.2k 1.4×	1.2k 1.2×	2.4k 3.2×	142	5.7k
Eva Giné	1.4k 0.4×	2.0k 0.8×	662 0.3×	444 0.5×	1.4k 1.8×	107	3.0k
Donatella Raspadori	1.5k 0.4×	984 0.4×	1.2k 0.5×	712 0.7×	848 1.1×	147	3.0k
José A. García‐Marco	1.5k 0.4×	758 0.3×	798 0.3×	816 0.8×	589 0.8×	88	2.6k
Karen Pulford	700 0.2×	2.5k 1.0×	825 0.3×	1.2k 1.3×	1.5k 2.0×	49	4.2k

Countries citing papers authored by Franco Fais

Since Specialization

Citations

This map shows the geographic impact of Franco Fais's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Franco Fais with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Franco Fais more than expected).

Fields of papers citing papers by Franco Fais

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Franco Fais. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Franco Fais. The network helps show where Franco Fais may publish in the future.

Co-authorship network of co-authors of Franco Fais

This figure shows the co-authorship network connecting the top 25 collaborators of Franco Fais. A scholar is included among the top collaborators of Franco Fais based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Franco Fais. Franco Fais is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bagnara, Davide, Andrea Nicola Mazzarello, Stefano Vergani, et al.. (2025). Chronic lymphocytic leukemia often arises by a multiclonal selection process. Haematologica. 110(8). 1747–1757.

Giannoni, Paolo, et al.. (2023). Unraveling the Bone Tissue Microenvironment in Chronic Lymphocytic Leukemia. Cancers. 15(20). 5058–5058. 4 indexed citations

Giannoni, Paolo, Cecilia Marini, Giovanna Cutrona, et al.. (2022). A High Percentage of CD16+ Monocytes Correlates with the Extent of Bone Erosion in Chronic Lymphocytic Leukemia Patients: The Impact of Leukemic B Cells in Monocyte Differentiation and Osteoclast Maturation. Cancers. 14(23). 5979–5979. 3 indexed citations

Lastraioli, Sonia, Romana Conte, Adalberto Ibatici, et al.. (2022). The spectrum of subclonal TP53 mutations in chronic lymphocytic leukemia: A next generation sequencing retrospective study. Hematological Oncology. 40(5). 962–975. 5 indexed citations

Vitale, Candida, Valentina Griggio, Daniela Drandi, et al.. (2022). Case Report: Sequential Development of Three Mature Lymphoid Neoplasms in a Single Patient: Clonal Relationship and Molecular Insights. Frontiers in Oncology. 12. 917115–917115. 3 indexed citations

Bagnara, Davide, Monica Colombo, Daniele Reverberi, et al.. (2022). Characterizing Features of Human Circulating B Cells Carrying CLL-Like Stereotyped Immunoglobulin Rearrangements. Frontiers in Oncology. 12. 894419–894419. 5 indexed citations

Menichini, Paola, Paola Monti, Giovanna Cutrona, et al.. (2021). Antitumor Effects of PRIMA-1 and PRIMA-1Met (APR246) in Hematological Malignancies: Still a Mutant P53-Dependent Affair?. Cells. 10(1). 98–98. 32 indexed citations

Giannoni, Paolo, Marco Grosso, Giuseppina Fugazza, et al.. (2021). Establishment and Characterization of a Novel Fibroblastic Cell Line (SCI13D) Derived from the Broncho-Alveolar Lavage of a Patient with Fibrotic Hypersensitivity Pneumonitis. Biomedicines. 9(9). 1193–1193. 3 indexed citations

Matis, Serena, Martina Rossi, Daniele Reverberi, et al.. (2021). LINC00152 expression in normal and Chronic Lymphocytic Leukemia B cells. Hematological Oncology. 40(1). 41–48. 5 indexed citations

10.

Pesenti, Elisa, Marı́a Rosa Bono, Andrea Nicola Mazzarello, et al.. (2021). SH3BGRL3 binds to myosin 1c in a calcium dependent manner and modulates migration in the MDA-MB-231 cell line. BMC Molecular and Cell Biology. 22(1). 41–41. 9 indexed citations

11.

Giannoni, Paolo, Franco Fais, Giovanna Cutrona, & Daniela de Totero. (2019). Hepatocyte Growth Factor: A Microenvironmental Resource for Leukemic Cell Growth. International Journal of Molecular Sciences. 20(2). 292–292. 10 indexed citations

12.

Sangaletti, Sabina, Claudio Tripodo, Caterina Vitali, et al.. (2013). Defective Stromal Remodeling and Neutrophil Extracellular Traps in Lymphoid Tissues Favor the Transition from Autoimmunity to Lymphoma. Cancer Discovery. 4(1). 110–129. 90 indexed citations

13.

Bruno, Silvia, Fabio Ghiotto, Claudya Tenca, et al.. (2012). N-(4-hydroxyphenyl)retinamide promotes apoptosis of resting and proliferating B-cell chronic lymphocytic leukemia cells and potentiates fludarabine and ABT-737 cytotoxicity. Leukemia. 26(10). 2260–2268. 13 indexed citations

14.

Ognibene, Marzia, Ottavia Barbieri, Cristina Vanni, et al.. (2011). High frequency of development of B cell lymphoproliferation and diffuse large B cell lymphoma in Dbl knock-in mice. Journal of Molecular Medicine. 89(5). 493–504. 6 indexed citations

15.

Tasso, Roberta, Andrea Augello, Simona Boccardo, et al.. (2009). Recruitment of a Host's Osteoprogenitor Cells Using Exogenous Mesenchymal Stem Cells Seeded on Porous Ceramic. Tissue Engineering Part A. 15(8). 2203–2212. 75 indexed citations

16.

Tasso, Roberta, Andrea Augello, Maria Grazia Tibiletti, et al.. (2008). Development of sarcomas in mice implanted with mesenchymal stem cells seeded onto bioscaffolds. Carcinogenesis. 30(1). 150–157. 90 indexed citations

17.

Bagnara, Davide, Vincenzo Callea, Caterina Stelitano, et al.. (2006). IgV gene intraclonal diversification and clonal evolution in B‐cell chronic lymphocytic leukaemia. British Journal of Haematology. 133(1). 50–58. 18 indexed citations

18.

Ghiotto, Fabio, Franco Fais, Angelo Valetto, et al.. (2004). Remarkably similar antigen receptors among a subset of patients with chronic lymphocytic leukemia. Journal of Clinical Investigation. 113(7). 1008–1016. 175 indexed citations

19.

Fais, Franco, Fabio Ghiotto, Shiori Hashimoto, et al.. (1998). Chronic lymphocytic leukemia B cells express restricted sets of mutated and unmutated antigen receptors.. Journal of Clinical Investigation. 102(8). 1515–1525. 690 indexed citations breakdown →

20.

Cutrona, Giovanna, Massimo Ulivi, Franco Fais, Silvio Roncella, & Manlio Ferrarini. (1995). Transfection of the c-myc oncogene into normal Epstein-Barr virus-harboring B cells results in new phenotypic and functional features resembling those of Burkitt lymphoma cells and normal centroblasts.. The Journal of Experimental Medicine. 181(2). 699–711. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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