Florence A. Arts

564 total citations
9 papers, 369 citations indexed

About

Florence A. Arts is a scholar working on Rheumatology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Florence A. Arts has authored 9 papers receiving a total of 369 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Rheumatology, 3 papers in Molecular Biology and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Florence A. Arts's work include Eosinophilic Disorders and Syndromes (4 papers), Soft tissue tumor case studies (4 papers) and Sarcoma Diagnosis and Treatment (2 papers). Florence A. Arts is often cited by papers focused on Eosinophilic Disorders and Syndromes (4 papers), Soft tissue tumor case studies (4 papers) and Sarcoma Diagnosis and Treatment (2 papers). Florence A. Arts collaborates with scholars based in Belgium, United States and Sweden. Florence A. Arts's co-authors include Jean‐Baptiste Demoulin, Guillaume Dachy, Amélie I. Velghe, Ahmed Essaghir, Monique Stevens, Dinel Pond, Damini Chand, Yoav H. Messinger, Bengt Hallberg and Stefan N. Constantinescu and has published in prestigious journals such as Oncogene, Human Molecular Genetics and Cellular and Molecular Life Sciences.

In The Last Decade

Florence A. Arts

9 papers receiving 369 citations

Peers

Florence A. Arts

RHEUM

PRM

MB

PFM

ONCOL

Amélie I. Velghe Belgium

Atsuko Saito Japan

Maria J. Merino United States

Kristin Andersen Norway

Jakob Hofvander Sweden

Anna Felisiak-Gołąbek Poland

Sonja Chen United States

Pamela A. Althof United States

Esther O’Regan Ireland

Lisa Shane United States

Amélie I. Velghe Belgium

Florence A. Arts

128 ×0.7

RHEUM

117 ×1.0

PRM

140 ×1.3

MB

55 ×0.7

PFM

72 ×1.3

ONCOL

Citations per year, relative to Florence A. Arts Florence A. Arts (= 1×) peers Amélie I. Velghe

Countries citing papers authored by Florence A. Arts

Since Specialization

Citations

This map shows the geographic impact of Florence A. Arts's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Florence A. Arts with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Florence A. Arts more than expected).

Fields of papers citing papers by Florence A. Arts

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Florence A. Arts. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Florence A. Arts. The network helps show where Florence A. Arts may publish in the future.

Co-authorship network of co-authors of Florence A. Arts

This figure shows the co-authorship network connecting the top 25 collaborators of Florence A. Arts. A scholar is included among the top collaborators of Florence A. Arts based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Florence A. Arts. Florence A. Arts is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

1.

Dachy, Guillaume, et al.. (2022). Penttinen syndrome‐associated PDGFRB Val665Ala variant causes aberrant constitutive STAT1 signalling. Journal of Cellular and Molecular Medicine. 26(14). 3902–3912. 7 indexed citations

2.

Arts, Florence A., et al.. (2021). PDGF receptor mutations in human diseases. Cellular and Molecular Life Sciences. 78(8). 3867–3881. 83 indexed citations

3.

Dachy, Guillaume, Ronald R. de Krijger, Sylvie Fraïtag, et al.. (2019). Association of PDGFRB Mutations With Pediatric Myofibroma and Myofibromatosis. JAMA Dermatology. 155(8). 946–946. 35 indexed citations

4.

Arts, Florence A., Paul Smyth, Sharon O’Toole, et al.. (2017). miR-223 potentially targets SWI/SNF complex protein SMARCD1 in atypical proliferative serous tumor and high-grade ovarian serous carcinoma. Human Pathology. 70. 98–104. 15 indexed citations

5.

Pond, Dinel, Florence A. Arts, Nancy J. Mendelsohn, et al.. (2017). A patient with germ-line gain-of-function PDGFRB p.N666H mutation and marked clinical response to imatinib. Genetics in Medicine. 20(1). 142–150. 45 indexed citations

6.

Arts, Florence A., Raf Sciot, Bénédicte Brichard, et al.. (2017). PDGFRB gain-of-function mutations in sporadic infantile myofibromatosis. Human Molecular Genetics. 26(10). 1801–1810. 63 indexed citations

7.

Arts, Florence A., Damini Chand, C. Pecquet, et al.. (2015). PDGFRB mutants found in patients with familial infantile myofibromatosis or overgrowth syndrome are oncogenic and sensitive to imatinib. Oncogene. 35(25). 3239–3248. 58 indexed citations

8.

Arts, Florence A., Amélie I. Velghe, Monique Stevens, et al.. (2014). Idiopathic basal ganglia calcification‐associated PDGFRB mutations impair the receptor signalling. Journal of Cellular and Molecular Medicine. 19(1). 239–248. 48 indexed citations

9.

Arts, Florence A., Carmen P. Montano‐Almendras, Luk Cox, et al.. (2014). The tyrosine phosphatase SHP2 is required for cell transformation by the receptor tyrosine kinase mutants FIP1L1‐PDGFRα and PDGFRα D842V. Molecular Oncology. 8(3). 728–740. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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