Feng‐Quan Zhou

5.5k total citations · 1 hit paper
51 papers, 3.8k citations indexed

About

Feng‐Quan Zhou is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Feng‐Quan Zhou has authored 51 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 17 papers in Developmental Neuroscience. Recurrent topics in Feng‐Quan Zhou's work include Nerve injury and regeneration (27 papers), Neurogenesis and neuroplasticity mechanisms (17 papers) and Axon Guidance and Neuronal Signaling (15 papers). Feng‐Quan Zhou is often cited by papers focused on Nerve injury and regeneration (27 papers), Neurogenesis and neuroplasticity mechanisms (17 papers) and Axon Guidance and Neuronal Signaling (15 papers). Feng‐Quan Zhou collaborates with scholars based in United States, China and Israel. Feng‐Quan Zhou's co-authors include Eun‐Mi Hur, William D. Snider, Christopher S. Cohan, Shoukat Dedhar, Jiang Zhou, Saijilafu, Shuxin Li, Clare M. Waterman, John C. Dill and Hongyu Wang and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Feng‐Quan Zhou

48 papers receiving 3.7k citations

Hit Papers

GSK3 signalling in neural development 2010 2026 2015 2020 2010 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Feng‐Quan Zhou United States 30 1.9k 1.9k 838 807 451 51 3.8k
Markus Plomann Germany 31 1.3k 0.6× 2.2k 1.1× 1.1k 1.3× 427 0.5× 335 0.7× 49 3.7k
Keling Zang United States 23 1.7k 0.9× 1.6k 0.8× 464 0.6× 856 1.1× 436 1.0× 27 3.8k
Nariko Arimura Japan 24 2.2k 1.1× 2.2k 1.2× 1.5k 1.8× 834 1.0× 352 0.8× 32 4.1k
Shernaz X. Bamji Canada 32 1.9k 1.0× 2.0k 1.1× 609 0.7× 604 0.7× 579 1.3× 47 3.8k
Josef P. Kapfhammer Switzerland 31 2.9k 1.5× 1.9k 1.0× 648 0.8× 1.4k 1.7× 309 0.7× 80 4.4k
Kelly R. Monk United States 34 1.6k 0.8× 2.0k 1.0× 565 0.7× 692 0.9× 252 0.6× 69 3.9k
Patrice D. Smith Canada 20 2.1k 1.1× 1.8k 1.0× 477 0.6× 821 1.0× 427 0.9× 33 3.9k
Maria K. Lehtinen United States 30 1.2k 0.6× 2.5k 1.3× 642 0.8× 782 1.0× 387 0.9× 63 4.5k
Philip Barker Canada 40 3.0k 1.5× 2.3k 1.2× 423 0.5× 1.2k 1.5× 450 1.0× 69 4.5k
Rejji Kuruvilla United States 29 1.6k 0.8× 1.6k 0.9× 706 0.8× 523 0.6× 424 0.9× 45 3.3k

Countries citing papers authored by Feng‐Quan Zhou

Since Specialization
Citations

This map shows the geographic impact of Feng‐Quan Zhou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Feng‐Quan Zhou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Feng‐Quan Zhou more than expected).

Fields of papers citing papers by Feng‐Quan Zhou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Feng‐Quan Zhou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Feng‐Quan Zhou. The network helps show where Feng‐Quan Zhou may publish in the future.

Co-authorship network of co-authors of Feng‐Quan Zhou

This figure shows the co-authorship network connecting the top 25 collaborators of Feng‐Quan Zhou. A scholar is included among the top collaborators of Feng‐Quan Zhou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Feng‐Quan Zhou. Feng‐Quan Zhou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Wang, Yuchen, Qiong Sun, Menachem Hanani, et al.. (2025). Demyelination-related pain: role of lysophosphatidic acid in satellite glial cell–neuron crosstalk. Journal of Translational Medicine. 23(1). 1425–1425.
3.
Zack, Donald J., et al.. (2024). Intercellular communication atlas reveals Oprm1 as a neuroprotective factor for retinal ganglion cells. Nature Communications. 15(1). 2206–2206. 3 indexed citations
4.
Yang, Shuguang, et al.. (2023). Glycogen synthase kinase 3 signaling in neural regeneration in vivo. Journal of Molecular Cell Biology. 15(12). 9 indexed citations
5.
Xia, Yu, Lu Ding, Changlin Zhang, et al.. (2023). Inflammatory Factor IL1α Induces Aberrant Astrocyte Proliferation in Spinal Cord Injury Through the Grin2c/Ca2+/CaMK2b Pathway. Neuroscience Bulletin. 40(4). 421–438. 5 indexed citations
6.
Chu, Weiwei, et al.. (2023). UCHL1 facilitates protein aggregates clearance to enhance neural stem cell activation in spinal cord injury. Cell Death and Disease. 14(7). 479–479. 14 indexed citations
7.
Zhang, Mei, Yong Zhang, Qian Xu, et al.. (2023). Neuronal Histone Methyltransferase EZH2 Regulates Neuronal Morphogenesis, Synaptic Plasticity, and Cognitive Behavior in Mice. Neuroscience Bulletin. 39(10). 1512–1532. 14 indexed citations
8.
Tower, Robert J., Yu‐Hao Cheng, Xuewei Wang, et al.. (2021). Spatial transcriptomics reveals a role for sensory nerves in preserving cranial suture patency through modulation of BMP/TGF-β signaling. Proceedings of the National Academy of Sciences. 118(42). 41 indexed citations
9.
Zhou, Feng‐Quan, et al.. (2020). In vivo glial trans‐differentiation for neuronal replacement and functional recovery in central nervous system. FEBS Journal. 288(16). 4773–4785. 20 indexed citations
10.
Li, Changjun, Yu Chai, Lei Wang, et al.. (2017). Programmed cell senescence in skeleton during late puberty. Nature Communications. 8(1). 1312–1312. 70 indexed citations
11.
Kim, Yu Shin, Michael Anderson, Kyoungsook Park, et al.. (2016). Coupled Activation of Primary Sensory Neurons Contributes to Chronic Pain. Neuron. 91(5). 1085–1096. 220 indexed citations
12.
Kim, Yun Tai, et al.. (2012). Slit2 Inactivates GSK3β to Signal Neurite Outgrowth Inhibition. PLoS ONE. 7(12). e51895–e51895. 13 indexed citations
13.
Saijilafu, Eun‐Mi Hur, & Feng‐Quan Zhou. (2011). Genetic dissection of axon regeneration via in vivo electroporation of adult mouse sensory neurons. Nature Communications. 2(1). 543–543. 56 indexed citations
14.
Kim, Yun Tai, Eun‐Mi Hur, William D. Snider, & Feng‐Quan Zhou. (2011). Role of GSK3 Signaling in Neuronal Morphogenesis. Frontiers in Molecular Neuroscience. 4. 48–48. 86 indexed citations
15.
Dill, John C., Hongyu Wang, Feng‐Quan Zhou, & Shuxin Li. (2008). Inactivation of Glycogen Synthase Kinase 3 Promotes Axonal Growth and Recovery in the CNS. Journal of Neuroscience. 28(36). 8914–8928. 188 indexed citations
17.
Zhou, Feng‐Quan, Jian Zhong, & William D. Snider. (2003). Extracellular Crosstalk. Cell. 113(7). 814–815. 17 indexed citations
18.
Cohan, Christopher S., et al.. (2003). Culturing Neurons from the Snail Helisoma. Methods in cell biology. 71. 157–170. 7 indexed citations
19.
Zhou, Feng‐Quan & Christopher S. Cohan. (2003). How actin filaments and microtubules steer growth cones to their targets. Journal of Neurobiology. 58(1). 84–91. 97 indexed citations
20.
Snider, William D., Feng‐Quan Zhou, Jian Zhong, & Annette Markus. (2002). Signaling the Pathway to Regeneration. Neuron. 35(1). 13–16. 117 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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