Fengjin Guo

2.4k total citations
41 papers, 1.0k citations indexed

About

Fengjin Guo is a scholar working on Molecular Biology, Cell Biology and Epidemiology. According to data from OpenAlex, Fengjin Guo has authored 41 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 19 papers in Cell Biology and 8 papers in Epidemiology. Recurrent topics in Fengjin Guo's work include Endoplasmic Reticulum Stress and Disease (18 papers), Autophagy in Disease and Therapy (8 papers) and RNA regulation and disease (7 papers). Fengjin Guo is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (18 papers), Autophagy in Disease and Therapy (8 papers) and RNA regulation and disease (7 papers). Fengjin Guo collaborates with scholars based in China, United States and United Kingdom. Fengjin Guo's co-authors include Liu C, Rong Jiang, Xiaofeng Han, Rui Luo, Edward Lin, Weiwei Xie, Min Liu, Wei Zheng, Naibo Feng and Qingyun Tian and has published in prestigious journals such as Nature Communications, The FASEB Journal and Journal of Cell Science.

In The Last Decade

Fengjin Guo

41 papers receiving 994 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Fengjin Guo China 18 466 263 227 149 125 41 1.0k
Luca Ulianich Italy 27 642 1.4× 313 1.2× 255 1.1× 41 0.3× 44 0.4× 42 1.5k
Tingting Lü China 20 501 1.1× 62 0.2× 188 0.8× 53 0.4× 126 1.0× 75 1.2k
Serena Grossi Italy 20 730 1.6× 169 0.6× 150 0.7× 63 0.4× 61 0.5× 33 1.2k
Masaru Honma Japan 21 382 0.8× 159 0.6× 142 0.6× 151 1.0× 44 0.4× 78 1.4k
Ching‐Chi Chiu Taiwan 20 538 1.2× 194 0.7× 121 0.5× 27 0.2× 193 1.5× 32 1.1k
Ye Zhao China 17 575 1.2× 87 0.3× 113 0.5× 50 0.3× 104 0.8× 33 1.3k
Yanan Ji China 19 659 1.4× 73 0.3× 136 0.6× 61 0.4× 66 0.5× 56 1.2k
Edoardo Nusco Italy 22 720 1.5× 319 1.2× 519 2.3× 94 0.6× 41 0.3× 37 1.5k
María Gabriela Morales Chile 26 1.1k 2.4× 252 1.0× 114 0.5× 63 0.4× 79 0.6× 42 1.8k

Countries citing papers authored by Fengjin Guo

Since Specialization
Citations

This map shows the geographic impact of Fengjin Guo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fengjin Guo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fengjin Guo more than expected).

Fields of papers citing papers by Fengjin Guo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fengjin Guo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fengjin Guo. The network helps show where Fengjin Guo may publish in the future.

Co-authorship network of co-authors of Fengjin Guo

This figure shows the co-authorship network connecting the top 25 collaborators of Fengjin Guo. A scholar is included among the top collaborators of Fengjin Guo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fengjin Guo. Fengjin Guo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fan, Mengtian, Biao Kuang, Fengmei Zhang, et al.. (2024). 10-hydroxy-2-decenoic acid prevents osteoarthritis by targeting aspartyl β hydroxylase and inhibiting chondrocyte senescence in male mice preclinically. Nature Communications. 15(1). 7712–7712. 19 indexed citations
2.
Feng, Naibo, Biao Kuang, Cheng Chen, et al.. (2024). The circUbqln1, regulated by XBP1s, interplays with 14–3–3ζ to inhibit collagen synthesis and promote osteoarthritis by controlling PRODH activity and proline metabolism. Journal of Advanced Research. 66. 267–284. 6 indexed citations
3.
Jiang, Rong, Shahid Masood Raza, Mengtian Fan, et al.. (2023). Gut-joint axis: Oral Probiotic ameliorates Osteoarthritis. Journal of Traditional and Complementary Medicine. 14(1). 26–39. 12 indexed citations
4.
5.
Fan, Mengtian, Xingyue Li, Danyang Yin, et al.. (2022). IRE1α regulates the PTHrP-IHH feedback loop to orchestrate chondrocyte hypertrophy and cartilage mineralization. Genes & Diseases. 11(1). 464–478. 6 indexed citations
6.
Fan, Mengtian, Cheng Chen, Rong Jiang, et al.. (2022). Progranulin regulation of autophagy contributes to its chondroprotective effect in osteoarthritis. Genes & Diseases. 10(4). 1582–1595. 12 indexed citations
7.
Li, Xiaoli, Mengtian Fan, Xingyue Li, et al.. (2021). Effect Of XBP1 Deficiency In Cartilage On The Regulatory Network Of LncRNA/circRNA-miRNA-mRNA. International Journal of Biological Sciences. 18(1). 315–330. 12 indexed citations
8.
Feng, Naibo & Fengjin Guo. (2020). Nanoparticle-siRNA: A potential strategy for rheumatoid arthritis therapy?. Journal of Controlled Release. 325. 380–393. 38 indexed citations
9.
Guo, Fengjin, Xiaojing Song, Y. Li, et al.. (2020). Longitudinal change in bone mineral density among Chinese individuals with HIV after initiation of antiretroviral therapy. Osteoporosis International. 32(2). 321–332. 12 indexed citations
10.
Zheng, Wei, et al.. (2019). ATG5 and ATG7 induced autophagy interplays with UPR via PERK signaling. Cell Communication and Signaling. 17(1). 42–42. 131 indexed citations
11.
Xie, Weiwei, et al.. (2018). BRF1 ameliorates LPS-induced inflammation through autophagy crosstalking with MAPK/ERK signaling. Genes & Diseases. 5(3). 226–234. 21 indexed citations
12.
Han, Xiaofeng, Jinghua Zhou, Peng Zhang, et al.. (2013). IRE1α dissociates with BiP and inhibits ER stress-mediated apoptosis in cartilage development. Cellular Signalling. 25(11). 2136–2146. 34 indexed citations
13.
Zhou, Jinghua, Xiaofeng Han, Yanna Liu, Peng Zhang, & Fengjin Guo. (2013). The effect on BiP regulation of IRE1a promoter transcription activity and protein expression. Hereditas (Beijing). 35(3). 343–351. 2 indexed citations
14.
Li, Xiangzhu, Huifang Zhu, Huizhe Huang, et al.. (2012). Study on the effect of IRE1α on cell growth and apoptosis via modulation PLK1 in ER stress response. Molecular and Cellular Biochemistry. 365(1-2). 99–108. 17 indexed citations
15.
Zhao, Wenjun, et al.. (2011). Effects of transcription factor XBP1S on cell proliferation and apoptosis of hepatoma HepG2 cells. Tumori. 31(1). 11–16. 1 indexed citations
16.
Guo, Fengjin, et al.. (2010). XBP1U inhibits the XBP1S-mediated upregulation of the iNOS gene expression in mammalian ER stress response. Cellular Signalling. 22(12). 1818–1828. 38 indexed citations
17.
Kong, Li, Qingyun Tian, Fengjin Guo, et al.. (2010). Interaction between cartilage oligomeric matrix protein and extracellular matrix protein 1 mediates endochondral bone growth. Matrix Biology. 29(4). 276–286. 37 indexed citations
18.
19.
Guo, Fengjin, Fangzhou Song, Jing Zhang, Jing Li, & Yong Tang. (2007). Study of Transcription Activity of X-Box Binding Protein 1 Gene in Human Different Cell Lines. Journal of genetics and genomics. 34(9). 790–799. 1 indexed citations
20.
Xu, Tao, et al.. (2006). Inhibition of cell growth and telomerase activity in osteosarcoma cells by DN-hTERT. Journal of Huazhong University of Science and Technology [Medical Sciences]. 26(5). 601–603. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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