Fei Teng

2.4k total citations · 1 hit paper
15 papers, 1.3k citations indexed

About

Fei Teng is a scholar working on Immunology, Surgery and Molecular Biology. According to data from OpenAlex, Fei Teng has authored 15 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Immunology, 6 papers in Surgery and 6 papers in Molecular Biology. Recurrent topics in Fei Teng's work include Immune Cell Function and Interaction (8 papers), Eosinophilic Esophagitis (6 papers) and IL-33, ST2, and ILC Pathways (6 papers). Fei Teng is often cited by papers focused on Immune Cell Function and Interaction (8 papers), Eosinophilic Esophagitis (6 papers) and IL-33, ST2, and ILC Pathways (6 papers). Fei Teng collaborates with scholars based in United States, France and United Kingdom. Fei Teng's co-authors include Krysta M. Felix, Caroline Bradley, Debdut Naskar, Hsin-Jung Wu, Gregory F. Sonnenberg, Gérard Eberl, Jérémy Goc, Manish A. Shah, Lei Zhou and Coco Chu and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Fei Teng

15 papers receiving 1.3k citations

Hit Papers

1 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identifying species of symbiont bacteria from the human gut that, alone, can induce intestinal Th17 cells in mice

2016 324 citations Tze Guan Tan, Esen Sefik et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fei Teng United States	12	647	595	272	250	175	15	1.3k
Carolina Galan United States	9	605 0.9×	813 1.4×	243 0.9×	207 0.8×	186 1.1×	13	1.4k
George P. Christophi United States	18	525 0.8×	583 1.0×	276 1.0×	185 0.7×	277 1.6×	34	1.4k
Caroline Johner Germany	5	936 1.4×	1.1k 1.9×	314 1.2×	360 1.4×	168 1.0×	7	2.2k
Goo‐Young Seo South Korea	19	537 0.8×	498 0.8×	201 0.7×	125 0.5×	76 0.4×	38	1.4k
Sascha Cording France	11	399 0.6×	787 1.3×	139 0.5×	143 0.6×	111 0.6×	12	1.2k
Kimie Hattori United States	3	756 1.2×	481 0.8×	110 0.4×	319 1.3×	238 1.4×	3	1.3k
Marta Galán-Díez United States	13	656 1.0×	364 0.6×	97 0.4×	281 1.1×	132 0.8×	17	1.4k
Yoshiyuki Mishima Japan	24	508 0.8×	541 0.9×	379 1.4×	153 0.6×	333 1.9×	66	1.5k
Sakhina Begum-Haque United States	12	797 1.2×	616 1.0×	71 0.3×	311 1.2×	125 0.7×	14	1.5k
Angela Schippers Germany	19	400 0.6×	915 1.5×	135 0.5×	78 0.3×	176 1.0×	35	1.5k

Countries citing papers authored by Fei Teng

Since Specialization

Citations

This map shows the geographic impact of Fei Teng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fei Teng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fei Teng more than expected).

Fields of papers citing papers by Fei Teng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fei Teng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fei Teng. The network helps show where Fei Teng may publish in the future.

Co-authorship network of co-authors of Fei Teng

This figure shows the co-authorship network connecting the top 25 collaborators of Fei Teng. A scholar is included among the top collaborators of Fei Teng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fei Teng. Fei Teng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Teng, Fei, et al.. (2024). A synthetic cytotoxic T cell platform for rapidly prototyping TCR function. npj Precision Oncology. 8(1). 182–182. 2 indexed citations

Joseph, Ann Mary, Anees Ahmed, Jérémy Goc, et al.. (2024). RIPK3 and caspase-8 interpret cytokine signals to regulate ILC3 survival in the gut. Mucosal Immunology. 17(6). 1212–1221. 6 indexed citations

Zhou, Lei, Wenqing Zhou, Ann Mary Joseph, et al.. (2022). Group 3 innate lymphoid cells produce the growth factor HB-EGF to protect the intestine from TNF-mediated inflammation. Nature Immunology. 23(2). 251–261. 61 indexed citations

Teng, Fei, Biin Sung, Donna L. Färber, et al.. (2021). ILC3s control airway inflammation by limiting T cell responses to allergens and microbes. Cell Reports. 37(8). 110051–110051. 26 indexed citations

Goc, Jérémy, Mengze Lv, Nicholas J. Bessman, et al.. (2021). Dysregulation of ILC3s unleashes progression and immunotherapy resistance in colon cancer. Cell. 184(19). 5015–5030.e16. 135 indexed citations

Zhou, Lei, Coco Chu, Fei Teng, et al.. (2019). Innate lymphoid cells support regulatory T cells in the intestine through interleukin-2. Nature. 568(7752). 405–409. 212 indexed citations

Felix, Krysta M., et al.. (2019). P2RX7 Deletion in T Cells Promotes Autoimmune Arthritis by Unleashing the Tfh Cell Response. Frontiers in Immunology. 10. 411–411. 13 indexed citations

Teng, Fei, Jérémy Goc, Lei Zhou, et al.. (2019). A circadian clock is essential for homeostasis of group 3 innate lymphoid cells in the gut. Science Immunology. 4(40). 81 indexed citations

Bradley, Caroline, Fei Teng, Krysta M. Felix, et al.. (2017). Segmented Filamentous Bacteria Provoke Lung Autoimmunity by Inducing Gut-Lung Axis Th17 Cells Expressing Dual TCRs. Cell Host & Microbe. 22(5). 697–704.e4. 147 indexed citations

10.

Teng, Fei, et al.. (2017). The impact of age and gut microbiota on Th17 and Tfh cells in K/BxN autoimmune arthritis. Arthritis Research & Therapy. 19(1). 188–188. 34 indexed citations

11.

Naskar, Debdut, Fei Teng, Krysta M. Felix, Caroline Bradley, & Hsin-Jung Wu. (2017). Synthetic Retinoid AM80 Ameliorates Lung and Arthritic Autoimmune Responses by Inhibiting T Follicular Helper and Th17 Cell Responses. The Journal of Immunology. 198(5). 1855–1864. 26 indexed citations

12.

Tan, Tze Guan, Esen Sefik, Naama Geva‐Zatorsky, et al.. (2016). Identifying species of symbiont bacteria from the human gut that, alone, can induce intestinal Th17 cells in mice. Proceedings of the National Academy of Sciences. 113(50). E8141–E8150. 324 indexed citations breakdown →

13.

Teng, Fei, Christina Klinger, Krysta M. Felix, et al.. (2016). Gut Microbiota Drive Autoimmune Arthritis by Promoting Differentiation and Migration of Peyer’s Patch T Follicular Helper Cells. Immunity. 44(4). 875–888. 216 indexed citations

14.

Wang, Haiyun, Xiaoqi Zheng, Fei Teng, et al.. (2015). Towards pathway‐centric cancer therapies via pharmacogenomic profiling analysis of ERK signalling pathway. SHILAP Revista de lepidopterología. 4(1). 66–66. 3 indexed citations

15.

Zhang, Jianping, Fei Teng, Zhongwei Li, et al.. (2013). BMP Induces Cochlin Expression to Facilitate Self-renewal and Suppress Neural Differentiation of Mouse Embryonic Stem Cells. Journal of Biological Chemistry. 288(12). 8053–8060. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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