Federico Sesti

4.8k total citations · 1 hit paper
58 papers, 3.4k citations indexed

About

Federico Sesti is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Federico Sesti has authored 58 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 46 papers in Molecular Biology, 32 papers in Cellular and Molecular Neuroscience and 15 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Federico Sesti's work include Ion channel regulation and function (37 papers), Neuroscience and Neuropharmacology Research (25 papers) and Cardiac electrophysiology and arrhythmias (15 papers). Federico Sesti is often cited by papers focused on Ion channel regulation and function (37 papers), Neuroscience and Neuropharmacology Research (25 papers) and Cardiac electrophysiology and arrhythmias (15 papers). Federico Sesti collaborates with scholars based in United States, Italy and Germany. Federico Sesti's co-authors include Steven A. Goldstein, Geoffrey W. Abbott, Mark T. Keating, Marianne E. Buck, Katherine W. Timothy, Michael H. Lehmann, Igor Splawski, Ki Ho Park, Shi‐Qing Cai and Manish Chhowalla and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Federico Sesti

58 papers receiving 3.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

MiRP1 Forms IKr Potassium Channels with HERG and Is Associated with Cardiac Arrhythmia

1999 1.0k citations Federico Sesti, Steven A. Goldstein et al. Cell profile →

Peers

Countries citing papers authored by Federico Sesti

Since Specialization

Citations

This map shows the geographic impact of Federico Sesti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Federico Sesti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Federico Sesti more than expected).

Fields of papers citing papers by Federico Sesti

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Federico Sesti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Federico Sesti. The network helps show where Federico Sesti may publish in the future.

Co-authorship network of co-authors of Federico Sesti

This figure shows the co-authorship network connecting the top 25 collaborators of Federico Sesti. A scholar is included among the top collaborators of Federico Sesti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Federico Sesti. Federico Sesti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Abnormal cytoskeletal remodeling but normal neuronal excitability in a mouse model of the recurrent developmental and epileptic encephalopathy-susceptibility KCNB1-p.R312H variant 2024 ·Communications Biology ·(unknown), (unknown), (unknown), Megha Dubey, (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Ping‐Yue Pan, David P. Crockett, Federico Sesti	1
2	Isolation of Targeted Hypothalamic Neurons for Studies of Hormonal, Metabolic, and Electrical Regulation 2023 ·Journal of Visualized Experiments ·(unknown), (unknown), (unknown), Federico Sesti	3
3	Ion channels in neurodevelopment: lessons from the Integrin-KCNB1 channel complex 2023 ·Neural Regeneration Research ·(unknown), Federico Sesti	6
4	Non-conducting functions of potassium channels in cancer and neurological disease 2023 ·Current topics in membranes ·Federico Sesti, (unknown), (unknown)	1
5	Integrin-KCNB1 potassium channel complexes regulate neocortical neuronal development and are implicated in epilepsy 2022 ·Cell Death and Differentiation ·(unknown), Wei Yu, (unknown), (unknown), (unknown), Madhuvika Murugan, Denise Fedele, (unknown), Detlev Boison, Mladen‐Roko Rašin, Federico Sesti	12
6	A novel assay for drug screening that utilizes the heat shock response of Caenorhabditis elegans nematodes 2020 ·PLoS ONE ·Chih‐Hsiung Chen, (unknown), (unknown), Federico Sesti	3
7	Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer’s disease 2018 ·Cell Death and Disease ·Wei Yu, (unknown), Federico Sesti	22
8	Cytoskeletal remodeling via Rho GTPases during oxidative and thermal stress in Caenorhabditis elegans 2017 ·Biochemical and Biophysical Research Communications ·(unknown), (unknown), (unknown), (unknown), (unknown), Federico Sesti	7
9	Oxidation of KCNB1 Potassium Channels Causes Neurotoxicity and Cognitive Impairment in a Mouse Model of Traumatic Brain Injury 2016 ·Journal of Neuroscience ·Wei Yu, (unknown), Shavonne Teng, (unknown), (unknown), Smita Thakker‐Varia, Janet Alder, Federico Sesti	31
10	Guanine Nucleotide Exchange Factor OSG-1 Confers Functional Aging via Dysregulated Rho Signaling in Caenorhabditis elegans Neurons 2014 ·Genetics ·Zhibing Duan, Federico Sesti	2
11	Delayed pharyngeal repolarization promotes abnormal calcium buildup in aging muscle 2013 ·Biochemical and Biophysical Research Communications ·Przemyslaw Swiatkowski, Federico Sesti	2
12	Molecular Mechanisms Underlying the Apoptotic Effect of KCNB1 K+ Channel Oxidation 2012 ·Journal of Biological Chemistry ·Xilong Wu, (unknown), (unknown), (unknown), Federico Sesti	32
13	Toxicity induced enhanced extracellular matrix production in osteoblastic cells cultured on single-walled carbon nanotube networks 2009 ·Nanotechnology ·Wojtek Tutak, Ki Ho Park, (unknown), Valentin Starovoytov, Giovanni Fanchini, (unknown), Nicola C. Partridge, Federico Sesti, Manish Chhowalla	47
14	Auto‐phosphorylation of a voltage‐gated K+ channel controls non‐associative learning 2009 ·The EMBO Journal ·(unknown), Yi Wang, Ki Ho Park, Xin Tong, Zui Pan, Federico Sesti	9
15	Multiple Modes of A-Type Potassium Current Regulation 2007 ·Current Pharmaceutical Design ·(unknown), Wenchao Li, Federico Sesti	18
16	MPS-1 is a K+ channel β-subunit and a serine/threonine kinase 2005 ·Nature Neuroscience ·(unknown), Leonardo Hernández, Yi Wang, Ki Ho Park, Federico Sesti	27
17	Pore- and State-Dependent Cadmium Block of IKs Channels Formed with MinK-55C and Wild-Type KCNQ1 Subunits 2003 ·Biophysical Journal ·Haijun Chen, Federico Sesti, Steven A. Goldstein	25
18	MinK Endows the IKs Potassium Channel Pore with Sensitivity to Internal Tetraethylammonium 2000 ·Biophysical Journal ·Federico Sesti, Kwok‐Keung Tai, Steven A. Goldstein	22
19	A Molecular Target for Viral Killer Toxin 1999 ·Cell ·Aamir Ahmed, Federico Sesti, Nitza Ilan, (unknown), Stephen L. Sturley, Steven A. Goldstein	83
20	Different channel-gating properties of two classes of cyclic GMP-activated channel in vertebrate photoreceptors 1992 ·Proceedings of the Royal Society B Biological Sciences ·Vincent Torre, (unknown), Federico Sesti, T.D. Lamb	28

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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