Fauzia Aslam
- Co-authors
- Leonard H. AugenlichtJanet L. SteinJane B. LianMichael BordonaroJohn M. MariadasonAnna VelcichAndré J. van WijnenBaruch Frenkel
- Topics
- Vitamin D Research Studies (5 papers)Estrogen and related hormone effects (4 papers)Cancer-related Molecular Pathways (3 papers)
- Partner nations
- United States
In The Last Decade
Fauzia Aslam
11 papers receiving 623 citations
Peers
Comparison fields: 5 of 71
- Molecular Biology 455
- Oncology 191
- Pathology and Forensic Medicine 143
- Genetics 129
- Cancer Research 49
Countries citing papers authored by Fauzia Aslam
This map shows the geographic impact of Fauzia Aslam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fauzia Aslam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fauzia Aslam more than expected).
Fields of papers citing papers by Fauzia Aslam
This network shows the impact of papers produced by Fauzia Aslam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fauzia Aslam. The network helps show where Fauzia Aslam may publish in the future.
Co-authorship network of co-authors of Fauzia Aslam
This figure shows the co-authorship network connecting the top 25 collaborators of Fauzia Aslam. A scholar is included among the top collaborators of Fauzia Aslam based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fauzia Aslam. Fauzia Aslam is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | The Sp family of transcription factors in the regulation of the human and mouse MUC2 gene promoters. | 46 |
| 2 | Down-regulation of beta-catenin TCF signaling is linked to colonic epithelial cell differentiation. | 150 |
| 3 | 37 | |
| 4 | The cell cycle regulator p27kip1 contributes to growth and differentiation of osteoblasts. | 88 |
| 5 | Butyrate-induced apoptotic cascade in colonic carcinoma cells: modulation of the beta-catenin-Tcf pathway and concordance with effects of sulindac and trichostatin A but not curcumin. | 83 |
| 6 | 26 | |
| 7 | 20 | |
| 8 | 3 | |
| 9 | 1 | |
| 10 | 70 | |
| 11 | 109 |
About Fauzia Aslam
Fauzia Aslam is a scholar working on Pathology and Forensic Medicine, Genetics and Molecular Biology, having authored 11 papers that have together received 633 indexed citations. Recurring topics across this work include Vitamin D Research Studies (5 papers), Estrogen and related hormone effects (4 papers) and Cancer-related Molecular Pathways (3 papers). The work is most often cited by research in Pathology and Forensic Medicine (143 citations), Oncology (191 citations) and Molecular Biology (455 citations). Fauzia Aslam has collaborated with scholars based in United States. Frequent co-authors include Leonard H. Augenlicht, Janet L. Stein, Jane B. Lian, Michael Bordonaro, John M. Mariadason, Anna Velcich, André J. van Wijnen, Baruch Frenkel, Mari Kuraguchi and Li Shi. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Cell Science and Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.