Fausto Fazzini

858 total citations
7 papers, 718 citations indexed

About

Fausto Fazzini is a scholar working on Immunology, Molecular Biology and Infectious Diseases. According to data from OpenAlex, Fausto Fazzini has authored 7 papers receiving a total of 718 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Molecular Biology and 0 papers in Infectious Diseases. Recurrent topics in Fausto Fazzini's work include Biomarkers in Disease Mechanisms (5 papers), Inflammasome and immune disorders (3 papers) and Phagocytosis and Immune Regulation (2 papers). Fausto Fazzini is often cited by papers focused on Biomarkers in Disease Mechanisms (5 papers), Inflammasome and immune disorders (3 papers) and Phagocytosis and Immune Regulation (2 papers). Fausto Fazzini collaborates with scholars based in Italy and Netherlands. Fausto Fazzini's co-authors include Angelo A. Manfredi, Alberto Mantovani, Maria Grazia Sabbadini, Giuseppe Peri, Andrea Doni, Patrizia Rovere, Attilio Bondanza, Valérie S. Zimmermann, C Rugarli and Patrizia Rovere‐Querini and has published in prestigious journals such as Blood, European Journal of Histochemistry and Arthritis & Rheumatism.

In The Last Decade

Fausto Fazzini

7 papers receiving 710 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Fausto Fazzini Italy 6 651 174 71 50 47 7 718
Benjamin Madden United States 9 138 0.2× 194 1.1× 96 1.4× 131 2.6× 92 2.0× 17 565
Norzawani B Buang United Kingdom 5 223 0.3× 116 0.7× 77 1.1× 17 0.3× 32 0.7× 5 360
Alexander Ulges Germany 6 400 0.6× 77 0.4× 19 0.3× 22 0.4× 116 2.5× 9 531
Renata Žunec Croatia 11 201 0.3× 108 0.6× 53 0.7× 26 0.5× 31 0.7× 68 423
Emma Shelley United Kingdom 7 373 0.6× 61 0.4× 73 1.0× 41 0.8× 9 0.2× 9 517
Hiroshi Tomizuka Japan 12 221 0.3× 100 0.6× 24 0.3× 41 0.8× 38 0.8× 25 399
Christine Deppong United States 11 453 0.7× 46 0.3× 39 0.5× 23 0.5× 58 1.2× 14 529
Nancy Wood United States 9 283 0.4× 39 0.2× 54 0.8× 44 0.9× 168 3.6× 9 455
Young-Hun Cho United States 4 284 0.4× 361 2.1× 31 0.4× 20 0.4× 16 0.3× 5 461
Seiji Segawa Japan 12 179 0.3× 76 0.4× 78 1.1× 42 0.8× 30 0.6× 17 320

Countries citing papers authored by Fausto Fazzini

Since Specialization
Citations

This map shows the geographic impact of Fausto Fazzini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fausto Fazzini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fausto Fazzini more than expected).

Fields of papers citing papers by Fausto Fazzini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fausto Fazzini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fausto Fazzini. The network helps show where Fausto Fazzini may publish in the future.

Co-authorship network of co-authors of Fausto Fazzini

This figure shows the co-authorship network connecting the top 25 collaborators of Fausto Fazzini. A scholar is included among the top collaborators of Fausto Fazzini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fausto Fazzini. Fausto Fazzini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Rovere, Patrizia, Fausto Fazzini, M G Sabbadini, & Angelo A. Manfredi. (2009). Apoptosis and systemic autoimmunity: the dendritic cell connection. European Journal of Histochemistry. 44(3). 229–36. 2 indexed citations
2.
Rossum, André P. van, Hendri H. Pas, Fausto Fazzini, et al.. (2006). Abundance of the long pentraxin PTX3 at sites of leukocytoclastic lesions in patients with small‐vessel vasculitis. Arthritis & Rheumatism. 54(3). 986–991. 53 indexed citations
3.
Rossum, André P. van, Fausto Fazzini, Pieter C. Limburg, et al.. (2004). The prototypic tissue pentraxin PTX3, in contrast to the short pentraxin serum amyloid P, inhibits phagocytosis of late apoptotic neutrophils by macrophages. Arthritis & Rheumatism. 50(8). 2667–2674. 79 indexed citations
4.
Fazzini, Fausto, Giuseppe Peri, Andrea Doni, et al.. (2001). PTX3 in small-vessel vasculitides: An independent indicator of disease activity produced at sites of inflammation. Arthritis & Rheumatism. 44(12). 2841–2850. 229 indexed citations
5.
Rovere, Patrizia, Giuseppe Peri, Fausto Fazzini, et al.. (2000). The long pentraxin PTX3 binds to apoptotic cells and regulates their clearance by antigen-presenting dendritic cells. Blood. 96(13). 4300–4306. 13 indexed citations
6.
Rovere, Patrizia, Maria Grazia Sabbadini, Fausto Fazzini, et al.. (2000). Remnants of suicidal cells fostering systemic autoaggression: Apoptosis in the origin and maintenance of autoimmunity. Arthritis & Rheumatism. 43(8). 1663–1672. 73 indexed citations
7.
Rovere, Patrizia, Giuseppe Peri, Fausto Fazzini, et al.. (2000). The long pentraxin PTX3 binds to apoptotic cells and regulates their clearance by antigen-presenting dendritic cells. Blood. 96(13). 4300–4306. 269 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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