Fabeha Fazal
- Immunology and Allergy top 5%
- Cell Adhesion Molecules Research 11
- Immunology top 10%
- Immune Response and Inflammation 7
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 3
- Cancer Research top 10%
- NF-κB Signaling Pathways 13
- Biochemistry top 5%
- Molecular Biology top 10%
- Protein Kinase Regulation and GTPase Signaling 4
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- Blood Coagulation and Thrombosis Mechanisms 5
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- Endoplasmic Reticulum Stress and Disease 3
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- Autophagy in Disease and Therapy 3
- Co-authors
- Arshad RahmanKaiser M. BijliMohd MinhajuddinAntony LeonardS.M. HadiJ.H. ParishKhandaker N. AnwarAsrar B. Malik
- Partner nations
- United StatesIndiaUnited Kingdom
In The Last Decade
Fabeha Fazal
38 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 105
- Immunology and Allergy 127
- Immunology 425
- Cancer Research 269
- Biochemistry 95
- Molecular Biology 780
Countries citing papers authored by Fabeha Fazal
This map shows the geographic impact of Fabeha Fazal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fabeha Fazal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fabeha Fazal more than expected).
Fields of papers citing papers by Fabeha Fazal
This network shows the impact of papers produced by Fabeha Fazal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fabeha Fazal. The network helps show where Fabeha Fazal may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Fabeha Fazal, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 0 | |
| 2 | 2020 | 21 | |
| 3 | 2019 | 27 | |
| 4 | 2017 | 37 | |
| 5 | 2014 | 34 | |
| 6 | 2012 | 26 | |
| 7 | 2011 | 3 | |
| 8 | 2011 | 173 | |
| 9 | 2009 | 56 | |
| 10 | 2008 | 130 | |
| 11 | 2008 | 49 | |
| 12 | 2008 | 63 | |
| 13 | Activation of mTOR by PKC-delta: Role in modulating thrombin-induced NF-kappaB activation and ICAM-1 expression in endothelial cells | 2007 | 1 |
| 14 | 2006 | 29 | |
| 15 | 2006 | 57 | |
| 16 | 2005 | 61 | |
| 17 | 2004 | 125 | |
| 18 | 1995 | 75 | |
| 19 | 1995 | 10 | |
| 20 | 1992 | 55 |
About Fabeha Fazal
Fabeha Fazal is a scholar working on Immunology and Allergy, Cancer Research and Immunology, having authored 39 papers that have together received 1.6k indexed citations. Recurring topics across this work include NF-κB Signaling Pathways (13 papers), Cell Adhesion Molecules Research (11 papers), Immune Response and Inflammation (7 papers), Blood Coagulation and Thrombosis Mechanisms (5 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers), Endoplasmic Reticulum Stress and Disease (3 papers) and Autophagy in Disease and Therapy (3 papers). The work is most often cited by research in Immunology and Allergy (127 citations), Immunology (425 citations) and Cancer Research (269 citations). Fabeha Fazal has collaborated with scholars based in United States, India and United Kingdom. Frequent co-authors include Arshad Rahman, Kaiser M. Bijli, Mohd Minhajuddin, Antony Leonard, S.M. Hadi, J.H. Parish, Khandaker N. Anwar, Asrar B. Malik, Spencer Slavin and Valerie Grose.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.